Describehow p16 andp21 caninhibit thecell cycle.Describe the roleof CETP andPCAT in reversetransport ofcholesterol byHDL. Use the criticaldifference todetermine if twoserial values aresignificantlydifferentDescribe thedifferentlocations ofsynthesis forcatecholaminesand serotoninDescribe the majorcauses, pathology,and clinical signs ofBeriberi, Wernicke-Korsakoff syndrome,Pellagra, and MultipleCarboxylaseDeficiency.List the mainfeatures ofthe DASHdietCompare andcontrast theintrinsic,extrinsic, andcommonpathwaysDescribe theprincipal roleof alpha-tocopherol inour cellsExplain thesignificance ofcodons AUG,UAG, UGA,and UAA.Explain howbase analogscan be used incancerchemotherapy. Define the Pasteureffect in the heartand list thebiochemical changesoccurring duringvessel occlusion thatlead to the Pasteureffect.Describe thesynthesisand use ofSAMDiscuss fetalalcoholsyndromeand ARNDsComparemarasmus,cachexiaandkwashiorkorExplain how the useof Bactrim in patientswith G6PD deficiencyresults in hemolyticanemia and theformation of Heinzbodies.Describe the basisof symptoms in thefollowingporphyrias: AIP,CEP, PCT, andEPP. Explain thecharacteristicsand function ofvon Willebrandfactor.Identify theuniquefeatures ofhumancolostrumExplain howa cell in G0differs from acell in G1.Name the9 essentialaminoacids.List thepopulations at riskfor vitamin D3deficiency andexplain why theyare at riskDescribe howprebioticsfavor calciumabsorptionfrom the colonDescribe theconversion ofcholesterolinto bile acidsand salts.Comparecatabolicand anabolicpathways.Explain the role oftetrahydrobiopterinin neurotransmittersynthesisDescribehow p16 andp21 caninhibit thecell cycle.Describe the roleof CETP andPCAT in reversetransport ofcholesterol byHDL. Use the criticaldifference todetermine if twoserial values aresignificantlydifferentDescribe thedifferentlocations ofsynthesis forcatecholaminesand serotoninDescribe the majorcauses, pathology,and clinical signs ofBeriberi, Wernicke-Korsakoff syndrome,Pellagra, and MultipleCarboxylaseDeficiency.List the mainfeatures ofthe DASHdietCompare andcontrast theintrinsic,extrinsic, andcommonpathwaysDescribe theprincipal roleof alpha-tocopherol inour cellsExplain thesignificance ofcodons AUG,UAG, UGA,and UAA.Explain howbase analogscan be used incancerchemotherapy. Define the Pasteureffect in the heartand list thebiochemical changesoccurring duringvessel occlusion thatlead to the Pasteureffect.Describe thesynthesisand use ofSAMDiscuss fetalalcoholsyndromeand ARNDsComparemarasmus,cachexiaandkwashiorkorExplain how the useof Bactrim in patientswith G6PD deficiencyresults in hemolyticanemia and theformation of Heinzbodies.Describe the basisof symptoms in thefollowingporphyrias: AIP,CEP, PCT, andEPP. Explain thecharacteristicsand function ofvon Willebrandfactor.Identify theuniquefeatures ofhumancolostrumExplain howa cell in G0differs from acell in G1.Name the9 essentialaminoacids.List thepopulations at riskfor vitamin D3deficiency andexplain why theyare at riskDescribe howprebioticsfavor calciumabsorptionfrom the colonDescribe theconversion ofcholesterolinto bile acidsand salts.Comparecatabolicand anabolicpathways.Explain the role oftetrahydrobiopterinin neurotransmittersynthesis

Untitled Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Describe how p16 and p21 can inhibit the cell cycle.
  2. Describe the role of CETP and PCAT in reverse transport of cholesterol by HDL.
  3. Use the critical difference to determine if two serial values are significantly different
  4. Describe the different locations of synthesis for catecholamines and serotonin
  5. Describe the major causes, pathology, and clinical signs of Beriberi, Wernicke-Korsakoff syndrome, Pellagra, and Multiple Carboxylase Deficiency.
  6. List the main features of the DASH diet
  7. Compare and contrast the intrinsic, extrinsic, and common pathways
  8. Describe the principal role of alpha-tocopherol in our cells
  9. Explain the significance of codons AUG, UAG, UGA, and UAA.
  10. Explain how base analogs can be used in cancer chemotherapy.
  11. Define the Pasteur effect in the heart and list the biochemical changes occurring during vessel occlusion that lead to the Pasteur effect.
  12. Describe the synthesis and use of SAM
  13. Discuss fetal alcohol syndrome and ARNDs
  14. Compare marasmus, cachexia and kwashiorkor
  15. Explain how the use of Bactrim in patients with G6PD deficiency results in hemolytic anemia and the formation of Heinz bodies.
  16. Describe the basis of symptoms in the following porphyrias: AIP, CEP, PCT, and EPP.
  17. Explain the characteristics and function of von Willebrand factor.
  18. Identify the unique features of human colostrum
  19. Explain how a cell in G0 differs from a cell in G1.
  20. Name the 9 essential amino acids.
  21. List the populations at risk for vitamin D3 deficiency and explain why they are at risk
  22. Describe how prebiotics favor calcium absorption from the colon
  23. Describe the conversion of cholesterol into bile acids and salts.
  24. Compare catabolic and anabolic pathways.
  25. Explain the role of tetrahydrobiopterin in neurotransmitter synthesis