patients whosedementia is soadvanced theycannon verballycommunicatecommonlyfearedadverseeffect ofopioid usedescribestransmission ofpainful stimuli &recognizes aconnection betweenpain & projection ofpain information tothe brainpain caused bylesion or diseaseof the peripheralor centralsomatosensorynervous systemperipheral receptorsthat respondselectively tomechanical,thermal, chemicalstimuli that arenoxiouspain thatisresistantto therapyinitiated bynociceptors thatare activated byactual orthreateneddamage tononneural tissuescattered painthat originates intendons,ligaments, bones,blood vessels,and nervessuperficialpain thatinvolves theskin or subqtissuecontrolledsubstancesyoung childrenwho can pointto a face toindicate theirlevel of painpainprocess:activation ofpainreceptorsdisease ispresent but theperson doesnot experiencepaintemporary flareup of moderateto severe painthat occurs withATC medicationchronic, relapsingbrain disease withcompulsive drugseeking and usedespite adverseconsequencesactivationof painreceptorspain scaleused forneonates0-6 monthsimportanthormone-likesubstance thatsend additionalpain stimuli toCNSperipheralsomatosensorynerve fibersthat transduceand encodenoxious stimulia neuropeptidethat sensitizesreceptors onnerves to feel pain& increase the rateat which nervesfirereduce painsensation byinhibiting therelease ofsubstance P fromthe terminals of theafferent neuronsbody becomesaccustomed tothe opioid andneeds a largerdosesubstancesthat eitherexcite orinhibit targetnerve cellshuman body'sdefensemechanism thatindicate theperson isexperiencing aprobleminvolves thesensoryprocess thatoccurs when astimulus forpain is presentusing 2 ormore differentmethods ormedications tomanage painpain originates inone part of bodybut is perceivedin an areadistance from itsorigin pointpowerfulvasodilator thatincreases capillarypermeability &constricts smoothmuscleincludesanticonvulsants,antidepressants,multipurposedrugspowerful pain-blocking chemicalsthat haveprolongedanalgesic effectsand produceeuphoriaphenomenonwhen body isphysiologicallyaccustomed toopioid therapy &suffers withdrawalsensationof pain isinhibited ormodifiedpoorly localizedpain; originatesin body organsin thorax,cranium, andabdomenendogenousopioid compoundsthat are morphine-like chemicalregulators in thespinal cord andbrainsymptomsof diseasere-appearPain sensations fromthe site of an injuryare conducted alongpathways to thespinal cord and thento higher centerspatients whosedementia is soadvanced theycannon verballycommunicatecommonlyfearedadverseeffect ofopioid usedescribestransmission ofpainful stimuli &recognizes aconnection betweenpain & projection ofpain information tothe brainpain caused bylesion or diseaseof the peripheralor centralsomatosensorynervous systemperipheral receptorsthat respondselectively tomechanical,thermal, chemicalstimuli that arenoxiouspain thatisresistantto therapyinitiated bynociceptors thatare activated byactual orthreateneddamage tononneural tissuescattered painthat originates intendons,ligaments, bones,blood vessels,and nervessuperficialpain thatinvolves theskin or subqtissuecontrolledsubstancesyoung childrenwho can pointto a face toindicate theirlevel of painpainprocess:activation ofpainreceptorsdisease ispresent but theperson doesnot experiencepaintemporary flareup of moderateto severe painthat occurs withATC medicationchronic, relapsingbrain disease withcompulsive drugseeking and usedespite adverseconsequencesactivationof painreceptorspain scaleused forneonates0-6 monthsimportanthormone-likesubstance thatsend additionalpain stimuli toCNSperipheralsomatosensorynerve fibersthat transduceand encodenoxious stimulia neuropeptidethat sensitizesreceptors onnerves to feel pain& increase the rateat which nervesfirereduce painsensation byinhibiting therelease ofsubstance P fromthe terminals of theafferent neuronsbody becomesaccustomed tothe opioid andneeds a largerdosesubstancesthat eitherexcite orinhibit targetnerve cellshuman body'sdefensemechanism thatindicate theperson isexperiencing aprobleminvolves thesensoryprocess thatoccurs when astimulus forpain is presentusing 2 ormore differentmethods ormedications tomanage painpain originates inone part of bodybut is perceivedin an areadistance from itsorigin pointpowerfulvasodilator thatincreases capillarypermeability &constricts smoothmuscleincludesanticonvulsants,antidepressants,multipurposedrugspowerful pain-blocking chemicalsthat haveprolongedanalgesic effectsand produceeuphoriaphenomenonwhen body isphysiologicallyaccustomed toopioid therapy &suffers withdrawalsensationof pain isinhibited ormodifiedpoorly localizedpain; originatesin body organsin thorax,cranium, andabdomenendogenousopioid compoundsthat are morphine-like chemicalregulators in thespinal cord andbrainsymptomsof diseasere-appearPain sensations fromthe site of an injuryare conducted alongpathways to thespinal cord and thento higher centers

Untitled Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. patients whose dementia is so advanced they cannon verbally communicate
  2. commonly feared adverse effect of opioid use
  3. describes transmission of painful stimuli & recognizes a connection between pain & projection of pain information to the brain
  4. pain caused by lesion or disease of the peripheral or central somatosensory nervous system
  5. peripheral receptors that respond selectively to mechanical, thermal, chemical stimuli that are noxious
  6. pain that is resistant to therapy
  7. initiated by nociceptors that are activated by actual or threatened damage to nonneural tissue
  8. scattered pain that originates in tendons, ligaments, bones, blood vessels, and nerves
  9. superficial pain that involves the skin or subq tissue
  10. controlled substances
  11. young children who can point to a face to indicate their level of pain
  12. pain process: activation of pain receptors
  13. disease is present but the person does not experience pain
  14. temporary flare up of moderate to severe pain that occurs with ATC medication
  15. chronic, relapsing brain disease with compulsive drug seeking and use despite adverse consequences
  16. activation of pain receptors
  17. pain scale used for neonates 0-6 months
  18. important hormone-like substance that send additional pain stimuli to CNS
  19. peripheral somatosensory nerve fibers that transduce and encode noxious stimuli
  20. a neuropeptide that sensitizes receptors on nerves to feel pain & increase the rate at which nerves fire
  21. reduce pain sensation by inhibiting the release of substance P from the terminals of the afferent neurons
  22. body becomes accustomed to the opioid and needs a larger dose
  23. substances that either excite or inhibit target nerve cells
  24. human body's defense mechanism that indicate the person is experiencing a problem
  25. involves the sensory process that occurs when a stimulus for pain is present
  26. using 2 or more different methods or medications to manage pain
  27. pain originates in one part of body but is perceived in an area distance from its origin point
  28. powerful vasodilator that increases capillary permeability & constricts smooth muscle
  29. includes anticonvulsants, antidepressants, multipurpose drugs
  30. powerful pain-blocking chemicals that have prolonged analgesic effects and produce euphoria
  31. phenomenon when body is physiologically accustomed to opioid therapy & suffers withdrawal
  32. sensation of pain is inhibited or modified
  33. poorly localized pain; originates in body organs in thorax, cranium, and abdomen
  34. endogenous opioid compounds that are morphine-like chemical regulators in the spinal cord and brain
  35. symptoms of disease re-appear
  36. Pain sensations from the site of an injury are conducted along pathways to the spinal cord and then to higher centers