Complianceability ofventriclesto distendor expandNegativeinotropedecreases the heartpumping/contractionie. BBEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.TorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASPositiveinotropeincreasesheartpumping/contractionie. DigoxinCreatininekinase(CK)found inheart, brainand skeletalmuscleCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterSAnodethe naturalpacemakerof the heartHR 60-100Chemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosinePositivechronotropeincreasesHR ie.AtropineSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinContractilityability ofheart topump,inotropyCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.StrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeAVnodeintrinsicHR 40-60CardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionNegativechronotropedecreasesHR ie. BBComplianceability ofventriclesto distendor expandNegativeinotropedecreases the heartpumping/contractionie. BBEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.TorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASPositiveinotropeincreasesheartpumping/contractionie. DigoxinCreatininekinase(CK)found inheart, brainand skeletalmuscleCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterSAnodethe naturalpacemakerof the heartHR 60-100Chemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosinePositivechronotropeincreasesHR ie.AtropineSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinContractilityability ofheart topump,inotropyCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.StrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeAVnodeintrinsicHR 40-60CardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionNegativechronotropedecreasesHR ie. BB

Cardiac Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. ability of ventricles to distend or expand
    Compliance
  2. decreases the heart pumping/contraction ie. BB
    Negative inotrope
  3. amount of blood in ventricles at end of diastole before contraction
    End Diastolic Volume (EDV)
  4. the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
    Starling’s Law
  5. Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
    Torsades de Pointes
  6. only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
    Troponin
  7. cause vasodilation, increase natriuresis and inhibit SNS and RAAS
    Natriuretic peptides
  8. increases heart pumping/ contraction ie. Digoxin
    Positive inotrope
  9. found in heart, brain and skeletal muscle
    Creatinine kinase (CK)
  10. specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
    CK-MB
  11. the natural pacemaker of the heart HR 60-100
    SA node
  12. sense changes in PaO2 or PaCO2, triggers SNS
    Chemoreceptors
  13. causes include stimulants, pain, fever, shock, hemorrhage
    Sinus Tachycardia
  14. percent of blood ejected by left ventricle. SV/EDV > 50% is normal
    Ejection Fraction (EF)
  15. dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
    Supraventricular Tachycardia (SVT)
  16. increases HR ie. Atropine
    Positive chronotrope
  17. causes include vagal responses, ICP, MI, BB, digoxin
    Sinus Bradycardia
  18. ability of heart to pump, inotropy
    Contractility
  19. atria beat independently of ventricles. 20-40 BPM, Low CO, requires pacemaker
    Complete Heart Block
  20. resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
    Afterload
  21. volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
    Preload
  22. volume of blood ejected by the heart with each beat
    Stroke Volume (SV)
  23. wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
    Ventricular Tachycardia (VT)
  24. intrinsic HR 40-60
    AV node
  25. the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
    Cardiac Output (CO)
  26. increase or decrease HR in response to pressure changes, ie: hypotension
    Baroreceptors
  27. decreases HR ie. BB
    Negative chronotrope