Baroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerPositiveinotropeincreasesheartpumping/contractionie. DigoxinSAnodethe naturalpacemakerof the heartHR 60-100PositivechronotropeincreasesHR ie.AtropineEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionCreatininekinase(CK)found inheart, brainand skeletalmuscleAVnodeintrinsicHR 40-60Contractilityability ofheart topump,inotropyTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterNegativechronotropedecreasesHR ie. BBNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASComplianceability ofventriclesto distendor expandChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.EjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineNegativeinotropedecreases the heartpumping/contractionie. BBPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.Afterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerPositiveinotropeincreasesheartpumping/contractionie. DigoxinSAnodethe naturalpacemakerof the heartHR 60-100PositivechronotropeincreasesHR ie.AtropineEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionCreatininekinase(CK)found inheart, brainand skeletalmuscleAVnodeintrinsicHR 40-60Contractilityability ofheart topump,inotropyTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterNegativechronotropedecreasesHR ie. BBNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASComplianceability ofventriclesto distendor expandChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.EjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineNegativeinotropedecreases the heartpumping/contractionie. BBPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.Afterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilation

Cardiac Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. increase or decrease HR in response to pressure changes, ie: hypotension
    Baroreceptors
  2. the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
    Cardiac Output (CO)
  3. atria beat independently of ventricles. 20-40 BPM, Low CO, requires pacemaker
    Complete Heart Block
  4. increases heart pumping/ contraction ie. Digoxin
    Positive inotrope
  5. the natural pacemaker of the heart HR 60-100
    SA node
  6. increases HR ie. Atropine
    Positive chronotrope
  7. amount of blood in ventricles at end of diastole before contraction
    End Diastolic Volume (EDV)
  8. found in heart, brain and skeletal muscle
    Creatinine kinase (CK)
  9. intrinsic HR 40-60
    AV node
  10. ability of heart to pump, inotropy
    Contractility
  11. only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
    Troponin
  12. decreases HR ie. BB
    Negative chronotrope
  13. cause vasodilation, increase natriuresis and inhibit SNS and RAAS
    Natriuretic peptides
  14. ability of ventricles to distend or expand
    Compliance
  15. sense changes in PaO2 or PaCO2, triggers SNS
    Chemoreceptors
  16. the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
    Starling’s Law
  17. percent of blood ejected by left ventricle. SV/EDV > 50% is normal
    Ejection Fraction (EF)
  18. wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
    Ventricular Tachycardia (VT)
  19. specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
    CK-MB
  20. Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
    Torsades de Pointes
  21. causes include vagal responses, ICP, MI, BB, digoxin
    Sinus Bradycardia
  22. volume of blood ejected by the heart with each beat
    Stroke Volume (SV)
  23. causes include stimulants, pain, fever, shock, hemorrhage
    Sinus Tachycardia
  24. dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
    Supraventricular Tachycardia (SVT)
  25. decreases the heart pumping/contraction ie. BB
    Negative inotrope
  26. volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
    Preload
  27. resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
    Afterload