Troponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterContractilityability ofheart topump,inotropyAVnodeintrinsicHR 40-60StrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.EndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerNegativechronotropedecreasesHR ie. BBChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVPositivechronotropeincreasesHR ie.AtropineNegativeinotropedecreases the heartpumping/contractionie. BBCreatininekinase(CK)found inheart, brainand skeletalmuscleSAnodethe naturalpacemakerof the heartHR 60-100Complianceability ofventriclesto distendor expandSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinPositiveinotropeincreasesheartpumping/contractionie. DigoxinPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.Natriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterContractilityability ofheart topump,inotropyAVnodeintrinsicHR 40-60StrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.EndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerNegativechronotropedecreasesHR ie. BBChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVPositivechronotropeincreasesHR ie.AtropineNegativeinotropedecreases the heartpumping/contractionie. BBCreatininekinase(CK)found inheart, brainand skeletalmuscleSAnodethe naturalpacemakerof the heartHR 60-100Complianceability ofventriclesto distendor expandSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinPositiveinotropeincreasesheartpumping/contractionie. DigoxinPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.Natriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcode

Cardiac Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
    Troponin
  2. ability of heart to pump, inotropy
    Contractility
  3. intrinsic HR 40-60
    AV node
  4. volume of blood ejected by the heart with each beat
    Stroke Volume (SV)
  5. resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
    Afterload
  6. the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
    Starling’s Law
  7. amount of blood in ventricles at end of diastole before contraction
    End Diastolic Volume (EDV)
  8. increase or decrease HR in response to pressure changes, ie: hypotension
    Baroreceptors
  9. atria beat independently of ventricles. 20-40 BPM, Low CO, requires pacemaker
    Complete Heart Block
  10. decreases HR ie. BB
    Negative chronotrope
  11. sense changes in PaO2 or PaCO2, triggers SNS
    Chemoreceptors
  12. Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
    Torsades de Pointes
  13. the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
    Cardiac Output (CO)
  14. increases HR ie. Atropine
    Positive chronotrope
  15. decreases the heart pumping/contraction ie. BB
    Negative inotrope
  16. found in heart, brain and skeletal muscle
    Creatinine kinase (CK)
  17. the natural pacemaker of the heart HR 60-100
    SA node
  18. ability of ventricles to distend or expand
    Compliance
  19. causes include vagal responses, ICP, MI, BB, digoxin
    Sinus Bradycardia
  20. increases heart pumping/ contraction ie. Digoxin
    Positive inotrope
  21. volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
    Preload
  22. cause vasodilation, increase natriuresis and inhibit SNS and RAAS
    Natriuretic peptides
  23. percent of blood ejected by left ventricle. SV/EDV > 50% is normal
    Ejection Fraction (EF)
  24. specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
    CK-MB
  25. dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
    Supraventricular Tachycardia (SVT)
  26. causes include stimulants, pain, fever, shock, hemorrhage
    Sinus Tachycardia
  27. wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
    Ventricular Tachycardia (VT)