PositivechronotropeincreasesHR ie.AtropineVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinAVnodeintrinsicHR 40-60Starling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.SinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageSAnodethe naturalpacemakerof the heartHR 60-100Complianceability ofventriclesto distendor expandNegativechronotropedecreasesHR ie. BBTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsContractilityability ofheart topump,inotropyCreatininekinase(CK)found inheart, brainand skeletalmuscleChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.CardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosinePositiveinotropeincreasesheartpumping/contractionie. DigoxinNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionNegativeinotropedecreases the heartpumping/contractionie. BBCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerPositivechronotropeincreasesHR ie.AtropineVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinAVnodeintrinsicHR 40-60Starling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.SinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageSAnodethe naturalpacemakerof the heartHR 60-100Complianceability ofventriclesto distendor expandNegativechronotropedecreasesHR ie. BBTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsContractilityability ofheart topump,inotropyCreatininekinase(CK)found inheart, brainand skeletalmuscleChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.CardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosinePositiveinotropeincreasesheartpumping/contractionie. DigoxinNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionNegativeinotropedecreases the heartpumping/contractionie. BBCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemaker

Cardiac Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. increases HR ie. Atropine
    Positive chronotrope
  2. wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
    Ventricular Tachycardia (VT)
  3. causes include vagal responses, ICP, MI, BB, digoxin
    Sinus Bradycardia
  4. intrinsic HR 40-60
    AV node
  5. the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
    Starling’s Law
  6. causes include stimulants, pain, fever, shock, hemorrhage
    Sinus Tachycardia
  7. the natural pacemaker of the heart HR 60-100
    SA node
  8. ability of ventricles to distend or expand
    Compliance
  9. decreases HR ie. BB
    Negative chronotrope
  10. Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
    Torsades de Pointes
  11. ability of heart to pump, inotropy
    Contractility
  12. found in heart, brain and skeletal muscle
    Creatinine kinase (CK)
  13. sense changes in PaO2 or PaCO2, triggers SNS
    Chemoreceptors
  14. specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
    CK-MB
  15. percent of blood ejected by left ventricle. SV/EDV > 50% is normal
    Ejection Fraction (EF)
  16. volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
    Preload
  17. the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
    Cardiac Output (CO)
  18. amount of blood in ventricles at end of diastole before contraction
    End Diastolic Volume (EDV)
  19. volume of blood ejected by the heart with each beat
    Stroke Volume (SV)
  20. only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
    Troponin
  21. dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
    Supraventricular Tachycardia (SVT)
  22. increases heart pumping/ contraction ie. Digoxin
    Positive inotrope
  23. cause vasodilation, increase natriuresis and inhibit SNS and RAAS
    Natriuretic peptides
  24. resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
    Afterload
  25. increase or decrease HR in response to pressure changes, ie: hypotension
    Baroreceptors
  26. decreases the heart pumping/contraction ie. BB
    Negative inotrope
  27. atria beat independently of ventricles. 20-40 BPM, Low CO, requires pacemaker
    Complete Heart Block