EndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationPositivechronotropeincreasesHR ie.AtropineSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterComplianceability ofventriclesto distendor expandNegativeinotropedecreases the heartpumping/contractionie. BBSAnodethe naturalpacemakerof the heartHR 60-100VentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineCreatininekinase(CK)found inheart, brainand skeletalmuscleTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatAVnodeintrinsicHR 40-60SinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Preloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.Baroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalPositiveinotropeincreasesheartpumping/contractionie. DigoxinNegativechronotropedecreasesHR ie. BBTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASContractilityability ofheart topump,inotropyEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationPositivechronotropeincreasesHR ie.AtropineSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterComplianceability ofventriclesto distendor expandNegativeinotropedecreases the heartpumping/contractionie. BBSAnodethe naturalpacemakerof the heartHR 60-100VentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineCreatininekinase(CK)found inheart, brainand skeletalmuscleTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatAVnodeintrinsicHR 40-60SinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Preloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.Baroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalPositiveinotropeincreasesheartpumping/contractionie. DigoxinNegativechronotropedecreasesHR ie. BBTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASContractilityability ofheart topump,inotropy

Cardiac Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. amount of blood in ventricles at end of diastole before contraction
    End Diastolic Volume (EDV)
  2. resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
    Afterload
  3. increases HR ie. Atropine
    Positive chronotrope
  4. causes include stimulants, pain, fever, shock, hemorrhage
    Sinus Tachycardia
  5. specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
    CK-MB
  6. ability of ventricles to distend or expand
    Compliance
  7. decreases the heart pumping/contraction ie. BB
    Negative inotrope
  8. the natural pacemaker of the heart HR 60-100
    SA node
  9. wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
    Ventricular Tachycardia (VT)
  10. sense changes in PaO2 or PaCO2, triggers SNS
    Chemoreceptors
  11. dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
    Supraventricular Tachycardia (SVT)
  12. found in heart, brain and skeletal muscle
    Creatinine kinase (CK)
  13. only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
    Troponin
  14. volume of blood ejected by the heart with each beat
    Stroke Volume (SV)
  15. intrinsic HR 40-60
    AV node
  16. causes include vagal responses, ICP, MI, BB, digoxin
    Sinus Bradycardia
  17. the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
    Cardiac Output (CO)
  18. the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
    Starling’s Law
  19. volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
    Preload
  20. increase or decrease HR in response to pressure changes, ie: hypotension
    Baroreceptors
  21. atria beat independently of ventricles. 20-40 BPM, Low CO, requires pacemaker
    Complete Heart Block
  22. percent of blood ejected by left ventricle. SV/EDV > 50% is normal
    Ejection Fraction (EF)
  23. increases heart pumping/ contraction ie. Digoxin
    Positive inotrope
  24. decreases HR ie. BB
    Negative chronotrope
  25. Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
    Torsades de Pointes
  26. cause vasodilation, increase natriuresis and inhibit SNS and RAAS
    Natriuretic peptides
  27. ability of heart to pump, inotropy
    Contractility