(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.
1
2
3
4
5
6
7
8
9
10
11
12
13
14
15
16
17
18
19
20
21
22
23
24
25
26
27
amount of blood in ventricles at end of diastole before contraction
End Diastolic Volume (EDV)
percent of blood ejected by left ventricle. SV/EDV > 50% is normal
Ejection Fraction (EF)
the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
Cardiac Output (CO)
intrinsic HR 40-60
AV node
specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
CK-MB
ability of heart to pump, inotropy
Contractility
causes include vagal responses, ICP, MI, BB, digoxin
Sinus Bradycardia
ability of ventricles to distend or expand
Compliance
dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
Supraventricular Tachycardia (SVT)
Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
Torsades de Pointes
found in heart, brain and skeletal muscle
Creatinine kinase (CK)
sense changes in PaO2 or PaCO2, triggers SNS
Chemoreceptors
causes include stimulants, pain, fever, shock, hemorrhage
Sinus Tachycardia
resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
Afterload
decreases HR ie. BB
Negative chronotrope
volume of blood ejected by the heart with each beat
Stroke Volume (SV)
only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
Troponin
the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
Starling’s Law
volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
Preload
the natural pacemaker of the heart HR 60-100
SA node
increase or decrease HR in response to pressure changes, ie: hypotension
Baroreceptors
wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
Ventricular Tachycardia (VT)
cause vasodilation, increase natriuresis and inhibit SNS and RAAS
Natriuretic peptides