(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.
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the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
Starling’s Law
causes include vagal responses, ICP, MI, BB, digoxin
Sinus Bradycardia
decreases the heart pumping/contraction ie. BB
Negative inotrope
resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
Afterload
volume of blood ejected by the heart with each beat
Stroke Volume (SV)
increase or decrease HR in response to pressure changes, ie: hypotension
Baroreceptors
intrinsic HR 40-60
AV node
amount of blood in ventricles at end of diastole before contraction
End Diastolic Volume (EDV)
sense changes in PaO2 or PaCO2, triggers SNS
Chemoreceptors
cause vasodilation, increase natriuresis and inhibit SNS and RAAS
Natriuretic peptides
dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
Supraventricular Tachycardia (SVT)
Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
Torsades de Pointes
the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
Cardiac Output (CO)
decreases HR ie. BB
Negative chronotrope
increases HR ie. Atropine
Positive chronotrope
percent of blood ejected by left ventricle. SV/EDV > 50% is normal
Ejection Fraction (EF)
volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
Preload
the natural pacemaker of the heart HR 60-100
SA node
