SupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationAVnodeintrinsicHR 40-60Positiveinotropeincreasesheartpumping/contractionie. DigoxinEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionSAnodethe naturalpacemakerof the heartHR 60-100Complianceability ofventriclesto distendor expandNegativechronotropedecreasesHR ie. BBVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeNegativeinotropedecreases the heartpumping/contractionie. BBPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.StrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Creatininekinase(CK)found inheart, brainand skeletalmuscleSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinContractilityability ofheart topump,inotropyPositivechronotropeincreasesHR ie.AtropineSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationAVnodeintrinsicHR 40-60Positiveinotropeincreasesheartpumping/contractionie. DigoxinEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionSAnodethe naturalpacemakerof the heartHR 60-100Complianceability ofventriclesto distendor expandNegativechronotropedecreasesHR ie. BBVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeNegativeinotropedecreases the heartpumping/contractionie. BBPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.StrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Creatininekinase(CK)found inheart, brainand skeletalmuscleSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinContractilityability ofheart topump,inotropyPositivechronotropeincreasesHR ie.Atropine

Cardiac Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
    Supraventricular Tachycardia (SVT)
  2. specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
    CK-MB
  3. atria beat independently of ventricles. 20-40 BPM, Low CO, requires pacemaker
    Complete Heart Block
  4. cause vasodilation, increase natriuresis and inhibit SNS and RAAS
    Natriuretic peptides
  5. resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
    Afterload
  6. intrinsic HR 40-60
    AV node
  7. increases heart pumping/ contraction ie. Digoxin
    Positive inotrope
  8. amount of blood in ventricles at end of diastole before contraction
    End Diastolic Volume (EDV)
  9. the natural pacemaker of the heart HR 60-100
    SA node
  10. ability of ventricles to distend or expand
    Compliance
  11. decreases HR ie. BB
    Negative chronotrope
  12. wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
    Ventricular Tachycardia (VT)
  13. decreases the heart pumping/contraction ie. BB
    Negative inotrope
  14. volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
    Preload
  15. volume of blood ejected by the heart with each beat
    Stroke Volume (SV)
  16. Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
    Torsades de Pointes
  17. sense changes in PaO2 or PaCO2, triggers SNS
    Chemoreceptors
  18. the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
    Cardiac Output (CO)
  19. causes include stimulants, pain, fever, shock, hemorrhage
    Sinus Tachycardia
  20. only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
    Troponin
  21. increase or decrease HR in response to pressure changes, ie: hypotension
    Baroreceptors
  22. percent of blood ejected by left ventricle. SV/EDV > 50% is normal
    Ejection Fraction (EF)
  23. the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
    Starling’s Law
  24. found in heart, brain and skeletal muscle
    Creatinine kinase (CK)
  25. causes include vagal responses, ICP, MI, BB, digoxin
    Sinus Bradycardia
  26. ability of heart to pump, inotropy
    Contractility
  27. increases HR ie. Atropine
    Positive chronotrope