TorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterCreatininekinase(CK)found inheart, brainand skeletalmuscleEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationNegativechronotropedecreasesHR ie. BBEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionPositiveinotropeincreasesheartpumping/contractionie. DigoxinStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.VentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodePositivechronotropeincreasesHR ie.AtropineNegativeinotropedecreases the heartpumping/contractionie. BBBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionComplianceability ofventriclesto distendor expandStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Contractilityability ofheart topump,inotropySAnodethe naturalpacemakerof the heartHR 60-100SinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinAVnodeintrinsicHR 40-60SinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterCreatininekinase(CK)found inheart, brainand skeletalmuscleEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationNegativechronotropedecreasesHR ie. BBEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionPositiveinotropeincreasesheartpumping/contractionie. DigoxinStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.VentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodePositivechronotropeincreasesHR ie.AtropineNegativeinotropedecreases the heartpumping/contractionie. BBBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionComplianceability ofventriclesto distendor expandStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Contractilityability ofheart topump,inotropySAnodethe naturalpacemakerof the heartHR 60-100SinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinAVnodeintrinsicHR 40-60SinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhage

Cardiac Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
    Torsades de Pointes
  2. the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
    Cardiac Output (CO)
  3. specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
    CK-MB
  4. only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
    Troponin
  5. found in heart, brain and skeletal muscle
    Creatinine kinase (CK)
  6. percent of blood ejected by left ventricle. SV/EDV > 50% is normal
    Ejection Fraction (EF)
  7. atria beat independently of ventricles. 20-40 BPM, Low CO, requires pacemaker
    Complete Heart Block
  8. cause vasodilation, increase natriuresis and inhibit SNS and RAAS
    Natriuretic peptides
  9. dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
    Supraventricular Tachycardia (SVT)
  10. resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
    Afterload
  11. decreases HR ie. BB
    Negative chronotrope
  12. amount of blood in ventricles at end of diastole before contraction
    End Diastolic Volume (EDV)
  13. increases heart pumping/ contraction ie. Digoxin
    Positive inotrope
  14. volume of blood ejected by the heart with each beat
    Stroke Volume (SV)
  15. sense changes in PaO2 or PaCO2, triggers SNS
    Chemoreceptors
  16. volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
    Preload
  17. wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
    Ventricular Tachycardia (VT)
  18. increases HR ie. Atropine
    Positive chronotrope
  19. decreases the heart pumping/contraction ie. BB
    Negative inotrope
  20. increase or decrease HR in response to pressure changes, ie: hypotension
    Baroreceptors
  21. ability of ventricles to distend or expand
    Compliance
  22. the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
    Starling’s Law
  23. ability of heart to pump, inotropy
    Contractility
  24. the natural pacemaker of the heart HR 60-100
    SA node
  25. causes include vagal responses, ICP, MI, BB, digoxin
    Sinus Bradycardia
  26. intrinsic HR 40-60
    AV node
  27. causes include stimulants, pain, fever, shock, hemorrhage
    Sinus Tachycardia