EndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVAVnodeintrinsicHR 40-60CK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterContractilityability ofheart topump,inotropySinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinComplianceability ofventriclesto distendor expandSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsCreatininekinase(CK)found inheart, brainand skeletalmuscleChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationNegativechronotropedecreasesHR ie. BBStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerNegativeinotropedecreases the heartpumping/contractionie. BBPositivechronotropeincreasesHR ie.AtropinePositiveinotropeincreasesheartpumping/contractionie. DigoxinStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Preloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.SAnodethe naturalpacemakerof the heartHR 60-100Baroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVAVnodeintrinsicHR 40-60CK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterContractilityability ofheart topump,inotropySinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinComplianceability ofventriclesto distendor expandSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsCreatininekinase(CK)found inheart, brainand skeletalmuscleChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationNegativechronotropedecreasesHR ie. BBStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatTroponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerNegativeinotropedecreases the heartpumping/contractionie. BBPositivechronotropeincreasesHR ie.AtropinePositiveinotropeincreasesheartpumping/contractionie. DigoxinStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Preloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.SAnodethe naturalpacemakerof the heartHR 60-100Baroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAAS

Cardiac Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. amount of blood in ventricles at end of diastole before contraction
    End Diastolic Volume (EDV)
  2. percent of blood ejected by left ventricle. SV/EDV > 50% is normal
    Ejection Fraction (EF)
  3. the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
    Cardiac Output (CO)
  4. intrinsic HR 40-60
    AV node
  5. specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
    CK-MB
  6. ability of heart to pump, inotropy
    Contractility
  7. causes include vagal responses, ICP, MI, BB, digoxin
    Sinus Bradycardia
  8. ability of ventricles to distend or expand
    Compliance
  9. dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
    Supraventricular Tachycardia (SVT)
  10. Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
    Torsades de Pointes
  11. found in heart, brain and skeletal muscle
    Creatinine kinase (CK)
  12. sense changes in PaO2 or PaCO2, triggers SNS
    Chemoreceptors
  13. causes include stimulants, pain, fever, shock, hemorrhage
    Sinus Tachycardia
  14. resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
    Afterload
  15. decreases HR ie. BB
    Negative chronotrope
  16. volume of blood ejected by the heart with each beat
    Stroke Volume (SV)
  17. only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
    Troponin
  18. atria beat independently of ventricles. 20-40 BPM, Low CO, requires pacemaker
    Complete Heart Block
  19. decreases the heart pumping/contraction ie. BB
    Negative inotrope
  20. increases HR ie. Atropine
    Positive chronotrope
  21. increases heart pumping/ contraction ie. Digoxin
    Positive inotrope
  22. the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
    Starling’s Law
  23. volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
    Preload
  24. the natural pacemaker of the heart HR 60-100
    SA node
  25. increase or decrease HR in response to pressure changes, ie: hypotension
    Baroreceptors
  26. wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
    Ventricular Tachycardia (VT)
  27. cause vasodilation, increase natriuresis and inhibit SNS and RAAS
    Natriuretic peptides