CK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsNegativechronotropedecreasesHR ie. BBEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineComplianceability ofventriclesto distendor expandVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeContractilityability ofheart topump,inotropyNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Creatininekinase(CK)found inheart, brainand skeletalmusclePositivechronotropeincreasesHR ie.AtropineSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSSAnodethe naturalpacemakerof the heartHR 60-100Preloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.Troponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionAVnodeintrinsicHR 40-60Negativeinotropedecreases the heartpumping/contractionie. BBPositiveinotropeincreasesheartpumping/contractionie. DigoxinCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsNegativechronotropedecreasesHR ie. BBEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineComplianceability ofventriclesto distendor expandVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeContractilityability ofheart topump,inotropyNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Creatininekinase(CK)found inheart, brainand skeletalmusclePositivechronotropeincreasesHR ie.AtropineSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSSAnodethe naturalpacemakerof the heartHR 60-100Preloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.Troponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionAVnodeintrinsicHR 40-60Negativeinotropedecreases the heartpumping/contractionie. BBPositiveinotropeincreasesheartpumping/contractionie. Digoxin

Cardiac Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
    CK-MB
  2. Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
    Torsades de Pointes
  3. decreases HR ie. BB
    Negative chronotrope
  4. amount of blood in ventricles at end of diastole before contraction
    End Diastolic Volume (EDV)
  5. percent of blood ejected by left ventricle. SV/EDV > 50% is normal
    Ejection Fraction (EF)
  6. the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
    Cardiac Output (CO)
  7. volume of blood ejected by the heart with each beat
    Stroke Volume (SV)
  8. causes include stimulants, pain, fever, shock, hemorrhage
    Sinus Tachycardia
  9. dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
    Supraventricular Tachycardia (SVT)
  10. ability of ventricles to distend or expand
    Compliance
  11. wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
    Ventricular Tachycardia (VT)
  12. ability of heart to pump, inotropy
    Contractility
  13. cause vasodilation, increase natriuresis and inhibit SNS and RAAS
    Natriuretic peptides
  14. atria beat independently of ventricles. 20-40 BPM, Low CO, requires pacemaker
    Complete Heart Block
  15. resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
    Afterload
  16. the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
    Starling’s Law
  17. found in heart, brain and skeletal muscle
    Creatinine kinase (CK)
  18. increases HR ie. Atropine
    Positive chronotrope
  19. causes include vagal responses, ICP, MI, BB, digoxin
    Sinus Bradycardia
  20. sense changes in PaO2 or PaCO2, triggers SNS
    Chemoreceptors
  21. the natural pacemaker of the heart HR 60-100
    SA node
  22. volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
    Preload
  23. only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
    Troponin
  24. increase or decrease HR in response to pressure changes, ie: hypotension
    Baroreceptors
  25. intrinsic HR 40-60
    AV node
  26. decreases the heart pumping/contraction ie. BB
    Negative inotrope
  27. increases heart pumping/ contraction ie. Digoxin
    Positive inotrope