Positiveinotropeincreasesheartpumping/contractionie. DigoxinNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.SupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationCreatininekinase(CK)found inheart, brainand skeletalmuscleEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.SAnodethe naturalpacemakerof the heartHR 60-100Troponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionAVnodeintrinsicHR 40-60Negativeinotropedecreases the heartpumping/contractionie. BBPositivechronotropeincreasesHR ie.AtropineNegativechronotropedecreasesHR ie. BBCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerContractilityability ofheart topump,inotropyComplianceability ofventriclesto distendor expandChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodePositiveinotropeincreasesheartpumping/contractionie. DigoxinNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsPreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.SupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationCreatininekinase(CK)found inheart, brainand skeletalmuscleEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.SAnodethe naturalpacemakerof the heartHR 60-100Troponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionAVnodeintrinsicHR 40-60Negativeinotropedecreases the heartpumping/contractionie. BBPositivechronotropeincreasesHR ie.AtropineNegativechronotropedecreasesHR ie. BBCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVStrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerContractilityability ofheart topump,inotropyComplianceability ofventriclesto distendor expandChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcode

Cardiac Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. increases heart pumping/ contraction ie. Digoxin
    Positive inotrope
  2. cause vasodilation, increase natriuresis and inhibit SNS and RAAS
    Natriuretic peptides
  3. causes include vagal responses, ICP, MI, BB, digoxin
    Sinus Bradycardia
  4. Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
    Torsades de Pointes
  5. volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
    Preload
  6. dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
    Supraventricular Tachycardia (SVT)
  7. resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
    Afterload
  8. found in heart, brain and skeletal muscle
    Creatinine kinase (CK)
  9. percent of blood ejected by left ventricle. SV/EDV > 50% is normal
    Ejection Fraction (EF)
  10. amount of blood in ventricles at end of diastole before contraction
    End Diastolic Volume (EDV)
  11. the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
    Starling’s Law
  12. the natural pacemaker of the heart HR 60-100
    SA node
  13. only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
    Troponin
  14. increase or decrease HR in response to pressure changes, ie: hypotension
    Baroreceptors
  15. intrinsic HR 40-60
    AV node
  16. decreases the heart pumping/contraction ie. BB
    Negative inotrope
  17. increases HR ie. Atropine
    Positive chronotrope
  18. decreases HR ie. BB
    Negative chronotrope
  19. the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
    Cardiac Output (CO)
  20. volume of blood ejected by the heart with each beat
    Stroke Volume (SV)
  21. causes include stimulants, pain, fever, shock, hemorrhage
    Sinus Tachycardia
  22. atria beat independently of ventricles. 20-40 BPM, Low CO, requires pacemaker
    Complete Heart Block
  23. ability of heart to pump, inotropy
    Contractility
  24. ability of ventricles to distend or expand
    Compliance
  25. sense changes in PaO2 or PaCO2, triggers SNS
    Chemoreceptors
  26. specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
    CK-MB
  27. wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
    Ventricular Tachycardia (VT)