SinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASContractilityability ofheart topump,inotropyVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeNegativeinotropedecreases the heartpumping/contractionie. BBNegativechronotropedecreasesHR ie. BBPositivechronotropeincreasesHR ie.AtropineCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Troponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSAVnodeintrinsicHR 40-60Creatininekinase(CK)found inheart, brainand skeletalmusclePreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.StrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinPositiveinotropeincreasesheartpumping/contractionie. DigoxinEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVSAnodethe naturalpacemakerof the heartHR 60-100Complianceability ofventriclesto distendor expandSinusTachycardiacauses includestimulants,pain, fever,shock,hemorrhageNatriureticpeptidescausevasodilation,increasenatriuresis andinhibit SNS andRAASContractilityability ofheart topump,inotropyVentricularTachycardia(VT)wide and bizarrecomplexes, treatwith synchronizedcardioversion andmeds, pulselessrequires ACLS andcodeNegativeinotropedecreases the heartpumping/contractionie. BBNegativechronotropedecreasesHR ie. BBPositivechronotropeincreasesHR ie.AtropineCK-MBspecific to heart,elevates 3-12 hrsafter injury, peaksat 24 hr returns tobaseline 2-3 daysafterBaroreceptorsincrease ordecrease HR inresponse topressurechanges, ie:hypotensionAfterloadresistance LV mustovercome to circulateblood. ↑aorticstenosis, septalhypertrophy,vasoconstriction↓sepsis, vasodilationStarling’sLawthe more the LV isstretched the moreforceful thecontraction. Canonly stretch somuch and for solong.Troponinonly in heartmuscle, elevates3-6 hrs after injury,peaks at 12-48hrs, returns tobaseline 1-2weeks afterSupraventricularTachycardia(SVT)dysrhythmias thatstart above AVnode, treat withcarotid massage,Valsalva,adenosineEjectionFraction(EF)percent ofblood ejectedby left ventricle.SV/EDV > 50%is normalCompleteHeartBlockatria beatindependently ofventricles. 20-40BPM, Low CO,requirespacemakerChemoreceptorssensechanges inPaO2 orPaCO2,triggers SNSAVnodeintrinsicHR 40-60Creatininekinase(CK)found inheart, brainand skeletalmusclePreloadvolume of blood in LVat end of diastole.Affected by venousreturn to heart, totalblood volume, atrialkick and stiffness andthickness of heart.StrokeVolume(SV)volume ofblood ejectedby the heartwith eachbeatTorsadesdePointesVentricularTachycardia in thesetting of prolongedQT interval. Cancause VF. Causesinclude electrolytesand medicationsSinusBradycardiacausesinclude vagalresponses,ICP, MI, BB,digoxinPositiveinotropeincreasesheartpumping/contractionie. DigoxinEndDiastolicVolume(EDV)amount ofblood inventricles atend of diastolebeforecontractionCardiacOutput(CO)the volume ofblood ejectedby the heart in1 min. 4-6L atrest, HR x SVSAnodethe naturalpacemakerof the heartHR 60-100Complianceability ofventriclesto distendor expand

Cardiac Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. causes include stimulants, pain, fever, shock, hemorrhage
    Sinus Tachycardia
  2. cause vasodilation, increase natriuresis and inhibit SNS and RAAS
    Natriuretic peptides
  3. ability of heart to pump, inotropy
    Contractility
  4. wide and bizarre complexes, treat with synchronized cardioversion and meds, pulseless requires ACLS and code
    Ventricular Tachycardia (VT)
  5. decreases the heart pumping/contraction ie. BB
    Negative inotrope
  6. decreases HR ie. BB
    Negative chronotrope
  7. increases HR ie. Atropine
    Positive chronotrope
  8. specific to heart, elevates 3-12 hrs after injury, peaks at 24 hr returns to baseline 2-3 days after
    CK-MB
  9. increase or decrease HR in response to pressure changes, ie: hypotension
    Baroreceptors
  10. resistance LV must overcome to circulate blood. ↑aortic stenosis, septal hypertrophy, vasoconstriction ↓sepsis, vasodilation
    Afterload
  11. the more the LV is stretched the more forceful the contraction. Can only stretch so much and for so long.
    Starling’s Law
  12. only in heart muscle, elevates 3-6 hrs after injury, peaks at 12-48 hrs, returns to baseline 1-2 weeks after
    Troponin
  13. dysrhythmias that start above AV node, treat with carotid massage, Valsalva, adenosine
    Supraventricular Tachycardia (SVT)
  14. percent of blood ejected by left ventricle. SV/EDV > 50% is normal
    Ejection Fraction (EF)
  15. atria beat independently of ventricles. 20-40 BPM, Low CO, requires pacemaker
    Complete Heart Block
  16. sense changes in PaO2 or PaCO2, triggers SNS
    Chemoreceptors
  17. intrinsic HR 40-60
    AV node
  18. found in heart, brain and skeletal muscle
    Creatinine kinase (CK)
  19. volume of blood in LV at end of diastole. Affected by venous return to heart, total blood volume, atrial kick and stiffness and thickness of heart.
    Preload
  20. volume of blood ejected by the heart with each beat
    Stroke Volume (SV)
  21. Ventricular Tachycardia in the setting of prolonged QT interval. Can cause VF. Causes include electrolytes and medications
    Torsades de Pointes
  22. causes include vagal responses, ICP, MI, BB, digoxin
    Sinus Bradycardia
  23. increases heart pumping/ contraction ie. Digoxin
    Positive inotrope
  24. amount of blood in ventricles at end of diastole before contraction
    End Diastolic Volume (EDV)
  25. the volume of blood ejected by the heart in 1 min. 4-6L at rest, HR x SV
    Cardiac Output (CO)
  26. the natural pacemaker of the heart HR 60-100
    SA node
  27. ability of ventricles to distend or expand
    Compliance