Bile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit D>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisSmallIntestineChylomicronsare madehereHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsModerate-IntensityPitavastatinisconsideredthis type ofintensitiesSmokingModifiableRisk Factor foratherosclerosisStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaFattystreakThe initiallesion of anatheroscleroticplaquePrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortality>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemia<150mg/dLOptimallevel ofTGs>40mg/dLIdeal levelof HDL formenAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesFibratesGemfibrozilbelongs tothis class ofmedicationsTriglyceridesThismoleculemakes up55-65% ofVLDLLiverSynthesizesVLDL andHDL fromremants ofcholesterolLDLCholesterolThislipoprotein ismade up of50%cholesterolPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisNiacinThis drug ismosteffective inincreasingHDL<70mg/dLTargetLDL goalfor DM ptsXanthomasPhysicalexam findingc/whyperlipidemiaHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseChylomicronsThislipoproteinprimarilytransportsTGsHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensitySecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit D>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisSmallIntestineChylomicronsare madehereHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsModerate-IntensityPitavastatinisconsideredthis type ofintensitiesSmokingModifiableRisk Factor foratherosclerosisStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaFattystreakThe initiallesion of anatheroscleroticplaquePrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortality>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemia<150mg/dLOptimallevel ofTGs>40mg/dLIdeal levelof HDL formenAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesFibratesGemfibrozilbelongs tothis class ofmedicationsTriglyceridesThismoleculemakes up55-65% ofVLDLLiverSynthesizesVLDL andHDL fromremants ofcholesterolLDLCholesterolThislipoprotein ismade up of50%cholesterolPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisNiacinThis drug ismosteffective inincreasingHDL<70mg/dLTargetLDL goalfor DM ptsXanthomasPhysicalexam findingc/whyperlipidemiaHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseChylomicronsThislipoproteinprimarilytransportsTGsHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensitySecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblems

PA518 Lipid Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. This class of drugs can interfere with the absorption of Vit D
    Bile Acid Sequestrants
  2. This level of TGs puts a patient at risk for developing pancreatitis
    >500mg/dL
  3. Chylomicrons are made here
    Small Intestine
  4. First line treatment for hyperlipidemia
    HMG-CoA Reductase Inhibitors
  5. This disorder causes gene mutations for LDL receptors
    Familial Hypercholesterolemia
  6. Pitavastatin is considered this type of intensities
    Moderate-Intensity
  7. Modifiable Risk Factor for atherosclerosis
    Smoking
  8. ADRs of this drug include elevated liver enzymes, hepatotoxicity, and myalgia
    Statin Side effects
  9. The initial lesion of an atherosclerotic plaque
    Fatty streak
  10. This disorder occurs independent of other health problems
    Primary dyslipidemia
  11. Although this drug can lower TGs, it does not reduce CV morbidity and mortality
    Omega-3 Fatty Acids
  12. This level of TC on a fasting panel is diagnostic of hyperlipidemia
    >200mg/dL
  13. Optimal level of TGs
    <150mg/dL
  14. Ideal level of HDL for men
    >40mg/dL
  15. Hardening of the arteries by way of cholesterol plaques
    Atherosclerosis
  16. Gemfibrozil belongs to this class of medications
    Fibrates
  17. This molecule makes up 55-65% of VLDL
    Triglycerides
  18. Synthesizes VLDL and HDL from remants of cholesterol
    Liver
  19. This lipoprotein is made up of 50% cholesterol
    LDL Cholesterol
  20. The most effective drug at lowering LDL and is an injectable
    PCSK9 Inhibitors
  21. The best prevention for the development of atherosclerosis
    Lifestyle Changes
  22. This drug is most effective in increasing HDL
    Niacin
  23. Target LDL goal for DM pts
    <70mg/dL
  24. Physical exam finding c/w hyperlipidemia
    Xanthomas
  25. This lipoprotein is protective against heart disease
    HDL Cholesterol
  26. This lipoprotein primarily transports TGs
    Chylomicrons
  27. Atorvastatin 40mg is considered this type of intensity
    High-Intensity
  28. This disorder occurs due to other health problems
    Secondary dyslipidemia