StatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaFibratesGemfibrozilbelongs tothis class ofmedicationsBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit D<150mg/dLOptimallevel ofTGsNiacinThis drug ismosteffective inincreasingHDLLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsLDLCholesterolThislipoprotein ismade up of50%cholesterol>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisModerate-IntensityPitavastatinisconsideredthis type ofintensitiesAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensityLiverSynthesizesVLDL andHDL fromremants ofcholesterolChylomicronsThislipoproteinprimarilytransportsTGsSmokingModifiableRisk Factor foratherosclerosisHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemia>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemia>40mg/dLIdeal levelof HDL formenFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalitySecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectable<70mg/dLTargetLDL goalfor DM ptsHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseFattystreakThe initiallesion of anatheroscleroticplaqueXanthomasPhysicalexam findingc/whyperlipidemiaSmallIntestineChylomicronsare madehereTriglyceridesThismoleculemakes up55-65% ofVLDLStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaFibratesGemfibrozilbelongs tothis class ofmedicationsBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit D<150mg/dLOptimallevel ofTGsNiacinThis drug ismosteffective inincreasingHDLLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsLDLCholesterolThislipoprotein ismade up of50%cholesterol>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisModerate-IntensityPitavastatinisconsideredthis type ofintensitiesAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensityLiverSynthesizesVLDL andHDL fromremants ofcholesterolChylomicronsThislipoproteinprimarilytransportsTGsSmokingModifiableRisk Factor foratherosclerosisHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemia>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemia>40mg/dLIdeal levelof HDL formenFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalitySecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectable<70mg/dLTargetLDL goalfor DM ptsHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseFattystreakThe initiallesion of anatheroscleroticplaqueXanthomasPhysicalexam findingc/whyperlipidemiaSmallIntestineChylomicronsare madehereTriglyceridesThismoleculemakes up55-65% ofVLDL

PA518 Lipid Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. ADRs of this drug include elevated liver enzymes, hepatotoxicity, and myalgia
    Statin Side effects
  2. Gemfibrozil belongs to this class of medications
    Fibrates
  3. This class of drugs can interfere with the absorption of Vit D
    Bile Acid Sequestrants
  4. Optimal level of TGs
    <150mg/dL
  5. This drug is most effective in increasing HDL
    Niacin
  6. The best prevention for the development of atherosclerosis
    Lifestyle Changes
  7. This disorder occurs independent of other health problems
    Primary dyslipidemia
  8. This lipoprotein is made up of 50% cholesterol
    LDL Cholesterol
  9. This level of TGs puts a patient at risk for developing pancreatitis
    >500mg/dL
  10. Pitavastatin is considered this type of intensities
    Moderate-Intensity
  11. Hardening of the arteries by way of cholesterol plaques
    Atherosclerosis
  12. Atorvastatin 40mg is considered this type of intensity
    High-Intensity
  13. Synthesizes VLDL and HDL from remants of cholesterol
    Liver
  14. This lipoprotein primarily transports TGs
    Chylomicrons
  15. Modifiable Risk Factor for atherosclerosis
    Smoking
  16. First line treatment for hyperlipidemia
    HMG-CoA Reductase Inhibitors
  17. This level of TC on a fasting panel is diagnostic of hyperlipidemia
    >200mg/dL
  18. Ideal level of HDL for men
    >40mg/dL
  19. This disorder causes gene mutations for LDL receptors
    Familial Hypercholesterolemia
  20. Although this drug can lower TGs, it does not reduce CV morbidity and mortality
    Omega-3 Fatty Acids
  21. This disorder occurs due to other health problems
    Secondary dyslipidemia
  22. The most effective drug at lowering LDL and is an injectable
    PCSK9 Inhibitors
  23. Target LDL goal for DM pts
    <70mg/dL
  24. This lipoprotein is protective against heart disease
    HDL Cholesterol
  25. The initial lesion of an atherosclerotic plaque
    Fatty streak
  26. Physical exam finding c/w hyperlipidemia
    Xanthomas
  27. Chylomicrons are made here
    Small Intestine
  28. This molecule makes up 55-65% of VLDL
    Triglycerides