High-IntensityAtorvastatin40mg isconsideredthis type ofintensityChylomicronsThislipoproteinprimarilytransportsTGsFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsTriglyceridesThismoleculemakes up55-65% ofVLDLSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsModerate-IntensityPitavastatinisconsideredthis type ofintensitiesPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableNiacinThis drug ismosteffective inincreasingHDL>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisLiverSynthesizesVLDL andHDL fromremants ofcholesterolBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesSmokingModifiableRisk Factor foratherosclerosis>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemia<70mg/dLTargetLDL goalfor DM ptsLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortality>40mg/dLIdeal levelof HDL formenLDLCholesterolThislipoprotein ismade up of50%cholesterolStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaFattystreakThe initiallesion of anatheroscleroticplaqueSmallIntestineChylomicronsare madehere<150mg/dLOptimallevel ofTGsPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsXanthomasPhysicalexam findingc/whyperlipidemiaFibratesGemfibrozilbelongs tothis class ofmedicationsHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensityChylomicronsThislipoproteinprimarilytransportsTGsFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsTriglyceridesThismoleculemakes up55-65% ofVLDLSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsModerate-IntensityPitavastatinisconsideredthis type ofintensitiesPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableNiacinThis drug ismosteffective inincreasingHDL>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisLiverSynthesizesVLDL andHDL fromremants ofcholesterolBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesSmokingModifiableRisk Factor foratherosclerosis>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemia<70mg/dLTargetLDL goalfor DM ptsLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortality>40mg/dLIdeal levelof HDL formenLDLCholesterolThislipoprotein ismade up of50%cholesterolStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaFattystreakThe initiallesion of anatheroscleroticplaqueSmallIntestineChylomicronsare madehere<150mg/dLOptimallevel ofTGsPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsXanthomasPhysicalexam findingc/whyperlipidemiaFibratesGemfibrozilbelongs tothis class ofmedications

PA518 Lipid Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Atorvastatin 40mg is considered this type of intensity
    High-Intensity
  2. This lipoprotein primarily transports TGs
    Chylomicrons
  3. This disorder causes gene mutations for LDL receptors
    Familial Hypercholesterolemia
  4. This molecule makes up 55-65% of VLDL
    Triglycerides
  5. This disorder occurs due to other health problems
    Secondary dyslipidemia
  6. Pitavastatin is considered this type of intensities
    Moderate-Intensity
  7. The most effective drug at lowering LDL and is an injectable
    PCSK9 Inhibitors
  8. This drug is most effective in increasing HDL
    Niacin
  9. This level of TGs puts a patient at risk for developing pancreatitis
    >500mg/dL
  10. Synthesizes VLDL and HDL from remants of cholesterol
    Liver
  11. This class of drugs can interfere with the absorption of Vit D
    Bile Acid Sequestrants
  12. First line treatment for hyperlipidemia
    HMG-CoA Reductase Inhibitors
  13. Hardening of the arteries by way of cholesterol plaques
    Atherosclerosis
  14. Modifiable Risk Factor for atherosclerosis
    Smoking
  15. This level of TC on a fasting panel is diagnostic of hyperlipidemia
    >200mg/dL
  16. Target LDL goal for DM pts
    <70mg/dL
  17. The best prevention for the development of atherosclerosis
    Lifestyle Changes
  18. This lipoprotein is protective against heart disease
    HDL Cholesterol
  19. Although this drug can lower TGs, it does not reduce CV morbidity and mortality
    Omega-3 Fatty Acids
  20. Ideal level of HDL for men
    >40mg/dL
  21. This lipoprotein is made up of 50% cholesterol
    LDL Cholesterol
  22. ADRs of this drug include elevated liver enzymes, hepatotoxicity, and myalgia
    Statin Side effects
  23. The initial lesion of an atherosclerotic plaque
    Fatty streak
  24. Chylomicrons are made here
    Small Intestine
  25. Optimal level of TGs
    <150mg/dL
  26. This disorder occurs independent of other health problems
    Primary dyslipidemia
  27. Physical exam finding c/w hyperlipidemia
    Xanthomas
  28. Gemfibrozil belongs to this class of medications
    Fibrates