HMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsSmokingModifiableRisk Factor foratherosclerosisFibratesGemfibrozilbelongs tothis class ofmedicationsPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalityLDLCholesterolThislipoprotein ismade up of50%cholesterolFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsSmallIntestineChylomicronsare madehereXanthomasPhysicalexam findingc/whyperlipidemiaSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsNiacinThis drug ismosteffective inincreasingHDL<150mg/dLOptimallevel ofTGsHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensity>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisLiverSynthesizesVLDL andHDL fromremants ofcholesterolChylomicronsThislipoproteinprimarilytransportsTGsLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosis>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemiaStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaFattystreakThe initiallesion of anatheroscleroticplaqueModerate-IntensityPitavastatinisconsideredthis type ofintensities>40mg/dLIdeal levelof HDL formen<70mg/dLTargetLDL goalfor DM ptsTriglyceridesThismoleculemakes up55-65% ofVLDLHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsSmokingModifiableRisk Factor foratherosclerosisFibratesGemfibrozilbelongs tothis class ofmedicationsPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalityLDLCholesterolThislipoprotein ismade up of50%cholesterolFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsSmallIntestineChylomicronsare madehereXanthomasPhysicalexam findingc/whyperlipidemiaSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsNiacinThis drug ismosteffective inincreasingHDL<150mg/dLOptimallevel ofTGsHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensity>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisLiverSynthesizesVLDL andHDL fromremants ofcholesterolChylomicronsThislipoproteinprimarilytransportsTGsLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosis>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemiaStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaFattystreakThe initiallesion of anatheroscleroticplaqueModerate-IntensityPitavastatinisconsideredthis type ofintensities>40mg/dLIdeal levelof HDL formen<70mg/dLTargetLDL goalfor DM ptsTriglyceridesThismoleculemakes up55-65% ofVLDL

PA518 Lipid Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. First line treatment for hyperlipidemia
    HMG-CoA Reductase Inhibitors
  2. This disorder occurs independent of other health problems
    Primary dyslipidemia
  3. Modifiable Risk Factor for atherosclerosis
    Smoking
  4. Gemfibrozil belongs to this class of medications
    Fibrates
  5. The most effective drug at lowering LDL and is an injectable
    PCSK9 Inhibitors
  6. This class of drugs can interfere with the absorption of Vit D
    Bile Acid Sequestrants
  7. This lipoprotein is protective against heart disease
    HDL Cholesterol
  8. Hardening of the arteries by way of cholesterol plaques
    Atherosclerosis
  9. Although this drug can lower TGs, it does not reduce CV morbidity and mortality
    Omega-3 Fatty Acids
  10. This lipoprotein is made up of 50% cholesterol
    LDL Cholesterol
  11. This disorder causes gene mutations for LDL receptors
    Familial Hypercholesterolemia
  12. Chylomicrons are made here
    Small Intestine
  13. Physical exam finding c/w hyperlipidemia
    Xanthomas
  14. This disorder occurs due to other health problems
    Secondary dyslipidemia
  15. This drug is most effective in increasing HDL
    Niacin
  16. Optimal level of TGs
    <150mg/dL
  17. Atorvastatin 40mg is considered this type of intensity
    High-Intensity
  18. This level of TGs puts a patient at risk for developing pancreatitis
    >500mg/dL
  19. Synthesizes VLDL and HDL from remants of cholesterol
    Liver
  20. This lipoprotein primarily transports TGs
    Chylomicrons
  21. The best prevention for the development of atherosclerosis
    Lifestyle Changes
  22. This level of TC on a fasting panel is diagnostic of hyperlipidemia
    >200mg/dL
  23. ADRs of this drug include elevated liver enzymes, hepatotoxicity, and myalgia
    Statin Side effects
  24. The initial lesion of an atherosclerotic plaque
    Fatty streak
  25. Pitavastatin is considered this type of intensities
    Moderate-Intensity
  26. Ideal level of HDL for men
    >40mg/dL
  27. Target LDL goal for DM pts
    <70mg/dL
  28. This molecule makes up 55-65% of VLDL
    Triglycerides