StatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgia>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemia<150mg/dLOptimallevel ofTGs>40mg/dLIdeal levelof HDL formenLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DTriglyceridesThismoleculemakes up55-65% ofVLDLHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseXanthomasPhysicalexam findingc/whyperlipidemiaNiacinThis drug ismosteffective inincreasingHDLPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensityFattystreakThe initiallesion of anatheroscleroticplaqueFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsFibratesGemfibrozilbelongs tothis class ofmedicationsSmallIntestineChylomicronsare madehereLDLCholesterolThislipoprotein ismade up of50%cholesterolModerate-IntensityPitavastatinisconsideredthis type ofintensitiesAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaques<70mg/dLTargetLDL goalfor DM ptsSmokingModifiableRisk Factor foratherosclerosisSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsChylomicronsThislipoproteinprimarilytransportsTGsOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalityLiverSynthesizesVLDL andHDL fromremants ofcholesterol>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgia>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemia<150mg/dLOptimallevel ofTGs>40mg/dLIdeal levelof HDL formenLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DTriglyceridesThismoleculemakes up55-65% ofVLDLHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseXanthomasPhysicalexam findingc/whyperlipidemiaNiacinThis drug ismosteffective inincreasingHDLPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensityFattystreakThe initiallesion of anatheroscleroticplaqueFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsFibratesGemfibrozilbelongs tothis class ofmedicationsSmallIntestineChylomicronsare madehereLDLCholesterolThislipoprotein ismade up of50%cholesterolModerate-IntensityPitavastatinisconsideredthis type ofintensitiesAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaques<70mg/dLTargetLDL goalfor DM ptsSmokingModifiableRisk Factor foratherosclerosisSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsChylomicronsThislipoproteinprimarilytransportsTGsOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalityLiverSynthesizesVLDL andHDL fromremants ofcholesterol>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitis

PA518 Lipid Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. ADRs of this drug include elevated liver enzymes, hepatotoxicity, and myalgia
    Statin Side effects
  2. This level of TC on a fasting panel is diagnostic of hyperlipidemia
    >200mg/dL
  3. Optimal level of TGs
    <150mg/dL
  4. Ideal level of HDL for men
    >40mg/dL
  5. The best prevention for the development of atherosclerosis
    Lifestyle Changes
  6. This class of drugs can interfere with the absorption of Vit D
    Bile Acid Sequestrants
  7. This molecule makes up 55-65% of VLDL
    Triglycerides
  8. This lipoprotein is protective against heart disease
    HDL Cholesterol
  9. Physical exam finding c/w hyperlipidemia
    Xanthomas
  10. This drug is most effective in increasing HDL
    Niacin
  11. The most effective drug at lowering LDL and is an injectable
    PCSK9 Inhibitors
  12. First line treatment for hyperlipidemia
    HMG-CoA Reductase Inhibitors
  13. This disorder occurs independent of other health problems
    Primary dyslipidemia
  14. Atorvastatin 40mg is considered this type of intensity
    High-Intensity
  15. The initial lesion of an atherosclerotic plaque
    Fatty streak
  16. This disorder causes gene mutations for LDL receptors
    Familial Hypercholesterolemia
  17. Gemfibrozil belongs to this class of medications
    Fibrates
  18. Chylomicrons are made here
    Small Intestine
  19. This lipoprotein is made up of 50% cholesterol
    LDL Cholesterol
  20. Pitavastatin is considered this type of intensities
    Moderate-Intensity
  21. Hardening of the arteries by way of cholesterol plaques
    Atherosclerosis
  22. Target LDL goal for DM pts
    <70mg/dL
  23. Modifiable Risk Factor for atherosclerosis
    Smoking
  24. This disorder occurs due to other health problems
    Secondary dyslipidemia
  25. This lipoprotein primarily transports TGs
    Chylomicrons
  26. Although this drug can lower TGs, it does not reduce CV morbidity and mortality
    Omega-3 Fatty Acids
  27. Synthesizes VLDL and HDL from remants of cholesterol
    Liver
  28. This level of TGs puts a patient at risk for developing pancreatitis
    >500mg/dL