NiacinThis drug ismosteffective inincreasingHDLFattystreakThe initiallesion of anatheroscleroticplaqueSmallIntestineChylomicronsare madehere<150mg/dLOptimallevel ofTGsFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsPrimarydyslipidemiaThis disorderoccursindependentof other healthproblems>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisXanthomasPhysicalexam findingc/whyperlipidemiaChylomicronsThislipoproteinprimarilytransportsTGsOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalityBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DLiverSynthesizesVLDL andHDL fromremants ofcholesterolLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisSmokingModifiableRisk Factor foratherosclerosisLDLCholesterolThislipoprotein ismade up of50%cholesterolFibratesGemfibrozilbelongs tothis class ofmedications<70mg/dLTargetLDL goalfor DM ptsPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseTriglyceridesThismoleculemakes up55-65% ofVLDLHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensity>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemiaStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemia>40mg/dLIdeal levelof HDL formenModerate-IntensityPitavastatinisconsideredthis type ofintensitiesNiacinThis drug ismosteffective inincreasingHDLFattystreakThe initiallesion of anatheroscleroticplaqueSmallIntestineChylomicronsare madehere<150mg/dLOptimallevel ofTGsFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsPrimarydyslipidemiaThis disorderoccursindependentof other healthproblems>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisXanthomasPhysicalexam findingc/whyperlipidemiaChylomicronsThislipoproteinprimarilytransportsTGsOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalityBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DLiverSynthesizesVLDL andHDL fromremants ofcholesterolLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisSmokingModifiableRisk Factor foratherosclerosisLDLCholesterolThislipoprotein ismade up of50%cholesterolFibratesGemfibrozilbelongs tothis class ofmedications<70mg/dLTargetLDL goalfor DM ptsPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseTriglyceridesThismoleculemakes up55-65% ofVLDLHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensity>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemiaStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemia>40mg/dLIdeal levelof HDL formenModerate-IntensityPitavastatinisconsideredthis type ofintensities

PA518 Lipid Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. This drug is most effective in increasing HDL
    Niacin
  2. The initial lesion of an atherosclerotic plaque
    Fatty streak
  3. Chylomicrons are made here
    Small Intestine
  4. Optimal level of TGs
    <150mg/dL
  5. This disorder causes gene mutations for LDL receptors
    Familial Hypercholesterolemia
  6. This disorder occurs due to other health problems
    Secondary dyslipidemia
  7. This disorder occurs independent of other health problems
    Primary dyslipidemia
  8. This level of TGs puts a patient at risk for developing pancreatitis
    >500mg/dL
  9. Physical exam finding c/w hyperlipidemia
    Xanthomas
  10. This lipoprotein primarily transports TGs
    Chylomicrons
  11. Although this drug can lower TGs, it does not reduce CV morbidity and mortality
    Omega-3 Fatty Acids
  12. This class of drugs can interfere with the absorption of Vit D
    Bile Acid Sequestrants
  13. Synthesizes VLDL and HDL from remants of cholesterol
    Liver
  14. The best prevention for the development of atherosclerosis
    Lifestyle Changes
  15. Modifiable Risk Factor for atherosclerosis
    Smoking
  16. This lipoprotein is made up of 50% cholesterol
    LDL Cholesterol
  17. Gemfibrozil belongs to this class of medications
    Fibrates
  18. Target LDL goal for DM pts
    <70mg/dL
  19. The most effective drug at lowering LDL and is an injectable
    PCSK9 Inhibitors
  20. This lipoprotein is protective against heart disease
    HDL Cholesterol
  21. This molecule makes up 55-65% of VLDL
    Triglycerides
  22. Atorvastatin 40mg is considered this type of intensity
    High-Intensity
  23. This level of TC on a fasting panel is diagnostic of hyperlipidemia
    >200mg/dL
  24. ADRs of this drug include elevated liver enzymes, hepatotoxicity, and myalgia
    Statin Side effects
  25. Hardening of the arteries by way of cholesterol plaques
    Atherosclerosis
  26. First line treatment for hyperlipidemia
    HMG-CoA Reductase Inhibitors
  27. Ideal level of HDL for men
    >40mg/dL
  28. Pitavastatin is considered this type of intensities
    Moderate-Intensity