LDLCholesterolThislipoprotein ismade up of50%cholesterolSmallIntestineChylomicronsare madehere<70mg/dLTargetLDL goalfor DM ptsXanthomasPhysicalexam findingc/whyperlipidemiaSmokingModifiableRisk Factor foratherosclerosisHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalityBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DTriglyceridesThismoleculemakes up55-65% ofVLDLSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblems<150mg/dLOptimallevel ofTGsModerate-IntensityPitavastatinisconsideredthis type ofintensitiesAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesFibratesGemfibrozilbelongs tothis class ofmedicationsFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsChylomicronsThislipoproteinprimarilytransportsTGsPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaLiverSynthesizesVLDL andHDL fromremants ofcholesterol>40mg/dLIdeal levelof HDL formenFattystreakThe initiallesion of anatheroscleroticplaqueHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensity>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisNiacinThis drug ismosteffective inincreasingHDLPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectable>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemiaHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseLDLCholesterolThislipoprotein ismade up of50%cholesterolSmallIntestineChylomicronsare madehere<70mg/dLTargetLDL goalfor DM ptsXanthomasPhysicalexam findingc/whyperlipidemiaSmokingModifiableRisk Factor foratherosclerosisHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalityBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DTriglyceridesThismoleculemakes up55-65% ofVLDLSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblems<150mg/dLOptimallevel ofTGsModerate-IntensityPitavastatinisconsideredthis type ofintensitiesAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesFibratesGemfibrozilbelongs tothis class ofmedicationsFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsChylomicronsThislipoproteinprimarilytransportsTGsPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaLiverSynthesizesVLDL andHDL fromremants ofcholesterol>40mg/dLIdeal levelof HDL formenFattystreakThe initiallesion of anatheroscleroticplaqueHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensity>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisNiacinThis drug ismosteffective inincreasingHDLPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectable>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemiaHDLCholesterol Thislipoprotein isprotectiveagainst heartdisease

PA518 Lipid Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. This lipoprotein is made up of 50% cholesterol
    LDL Cholesterol
  2. Chylomicrons are made here
    Small Intestine
  3. Target LDL goal for DM pts
    <70mg/dL
  4. Physical exam finding c/w hyperlipidemia
    Xanthomas
  5. Modifiable Risk Factor for atherosclerosis
    Smoking
  6. First line treatment for hyperlipidemia
    HMG-CoA Reductase Inhibitors
  7. The best prevention for the development of atherosclerosis
    Lifestyle Changes
  8. Although this drug can lower TGs, it does not reduce CV morbidity and mortality
    Omega-3 Fatty Acids
  9. This class of drugs can interfere with the absorption of Vit D
    Bile Acid Sequestrants
  10. This molecule makes up 55-65% of VLDL
    Triglycerides
  11. This disorder occurs due to other health problems
    Secondary dyslipidemia
  12. Optimal level of TGs
    <150mg/dL
  13. Pitavastatin is considered this type of intensities
    Moderate-Intensity
  14. Hardening of the arteries by way of cholesterol plaques
    Atherosclerosis
  15. Gemfibrozil belongs to this class of medications
    Fibrates
  16. This disorder causes gene mutations for LDL receptors
    Familial Hypercholesterolemia
  17. This lipoprotein primarily transports TGs
    Chylomicrons
  18. This disorder occurs independent of other health problems
    Primary dyslipidemia
  19. ADRs of this drug include elevated liver enzymes, hepatotoxicity, and myalgia
    Statin Side effects
  20. Synthesizes VLDL and HDL from remants of cholesterol
    Liver
  21. Ideal level of HDL for men
    >40mg/dL
  22. The initial lesion of an atherosclerotic plaque
    Fatty streak
  23. Atorvastatin 40mg is considered this type of intensity
    High-Intensity
  24. This level of TGs puts a patient at risk for developing pancreatitis
    >500mg/dL
  25. This drug is most effective in increasing HDL
    Niacin
  26. The most effective drug at lowering LDL and is an injectable
    PCSK9 Inhibitors
  27. This level of TC on a fasting panel is diagnostic of hyperlipidemia
    >200mg/dL
  28. This lipoprotein is protective against heart disease
    HDL Cholesterol