High-IntensityAtorvastatin40mg isconsideredthis type ofintensityXanthomasPhysicalexam findingc/whyperlipidemiaPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisFibratesGemfibrozilbelongs tothis class ofmedicationsNiacinThis drug ismosteffective inincreasingHDLAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableSmokingModifiableRisk Factor foratherosclerosisSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblems<70mg/dLTargetLDL goalfor DM ptsTriglyceridesThismoleculemakes up55-65% ofVLDLModerate-IntensityPitavastatinisconsideredthis type ofintensitiesLDLCholesterolThislipoprotein ismade up of50%cholesterolFattystreakThe initiallesion of anatheroscleroticplaque<150mg/dLOptimallevel ofTGsHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalityLiverSynthesizesVLDL andHDL fromremants ofcholesterol>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsSmallIntestineChylomicronsare madehere>40mg/dLIdeal levelof HDL formenBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit D>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemiaHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaChylomicronsThislipoproteinprimarilytransportsTGsHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensityXanthomasPhysicalexam findingc/whyperlipidemiaPrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisFibratesGemfibrozilbelongs tothis class ofmedicationsNiacinThis drug ismosteffective inincreasingHDLAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectableSmokingModifiableRisk Factor foratherosclerosisSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblems<70mg/dLTargetLDL goalfor DM ptsTriglyceridesThismoleculemakes up55-65% ofVLDLModerate-IntensityPitavastatinisconsideredthis type ofintensitiesLDLCholesterolThislipoprotein ismade up of50%cholesterolFattystreakThe initiallesion of anatheroscleroticplaque<150mg/dLOptimallevel ofTGsHDLCholesterol Thislipoprotein isprotectiveagainst heartdiseaseOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortalityLiverSynthesizesVLDL andHDL fromremants ofcholesterol>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptorsSmallIntestineChylomicronsare madehere>40mg/dLIdeal levelof HDL formenBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit D>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemiaHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemiaChylomicronsThislipoproteinprimarilytransportsTGs

PA518 Lipid Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Atorvastatin 40mg is considered this type of intensity
    High-Intensity
  2. Physical exam finding c/w hyperlipidemia
    Xanthomas
  3. This disorder occurs independent of other health problems
    Primary dyslipidemia
  4. The best prevention for the development of atherosclerosis
    Lifestyle Changes
  5. Gemfibrozil belongs to this class of medications
    Fibrates
  6. This drug is most effective in increasing HDL
    Niacin
  7. Hardening of the arteries by way of cholesterol plaques
    Atherosclerosis
  8. The most effective drug at lowering LDL and is an injectable
    PCSK9 Inhibitors
  9. Modifiable Risk Factor for atherosclerosis
    Smoking
  10. This disorder occurs due to other health problems
    Secondary dyslipidemia
  11. Target LDL goal for DM pts
    <70mg/dL
  12. This molecule makes up 55-65% of VLDL
    Triglycerides
  13. Pitavastatin is considered this type of intensities
    Moderate-Intensity
  14. This lipoprotein is made up of 50% cholesterol
    LDL Cholesterol
  15. The initial lesion of an atherosclerotic plaque
    Fatty streak
  16. Optimal level of TGs
    <150mg/dL
  17. This lipoprotein is protective against heart disease
    HDL Cholesterol
  18. Although this drug can lower TGs, it does not reduce CV morbidity and mortality
    Omega-3 Fatty Acids
  19. Synthesizes VLDL and HDL from remants of cholesterol
    Liver
  20. This level of TGs puts a patient at risk for developing pancreatitis
    >500mg/dL
  21. ADRs of this drug include elevated liver enzymes, hepatotoxicity, and myalgia
    Statin Side effects
  22. This disorder causes gene mutations for LDL receptors
    Familial Hypercholesterolemia
  23. Chylomicrons are made here
    Small Intestine
  24. Ideal level of HDL for men
    >40mg/dL
  25. This class of drugs can interfere with the absorption of Vit D
    Bile Acid Sequestrants
  26. This level of TC on a fasting panel is diagnostic of hyperlipidemia
    >200mg/dL
  27. First line treatment for hyperlipidemia
    HMG-CoA Reductase Inhibitors
  28. This lipoprotein primarily transports TGs
    Chylomicrons