NiacinThis drug ismosteffective inincreasingHDLLDLCholesterolThislipoprotein ismade up of50%cholesterolHDLCholesterol Thislipoprotein isprotectiveagainst heartdisease<150mg/dLOptimallevel ofTGsStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemia>40mg/dLIdeal levelof HDL formenBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptors<70mg/dLTargetLDL goalfor DM ptsModerate-IntensityPitavastatinisconsideredthis type ofintensitiesSmallIntestineChylomicronsare madehereOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortality>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemiaPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectablePrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisTriglyceridesThismoleculemakes up55-65% ofVLDLSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsChylomicronsThislipoproteinprimarilytransportsTGsFibratesGemfibrozilbelongs tothis class ofmedicationsLiverSynthesizesVLDL andHDL fromremants ofcholesterolXanthomasPhysicalexam findingc/whyperlipidemiaHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensitySmokingModifiableRisk Factor foratherosclerosis>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisFattystreakThe initiallesion of anatheroscleroticplaqueAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaquesNiacinThis drug ismosteffective inincreasingHDLLDLCholesterolThislipoprotein ismade up of50%cholesterolHDLCholesterol Thislipoprotein isprotectiveagainst heartdisease<150mg/dLOptimallevel ofTGsStatinSideeffectsADRs of thisdrug includeelevated liverenzymes,hepatotoxicity,and myalgiaHMG-CoAReductaseInhibitorsFirst linetreatment forhyperlipidemia>40mg/dLIdeal levelof HDL formenBile AcidSequestrantsThis class ofdrugs caninterfere withthe absorptionof Vit DFamilialHypercholesterolemia This disordercauses genemutations forLDLreceptors<70mg/dLTargetLDL goalfor DM ptsModerate-IntensityPitavastatinisconsideredthis type ofintensitiesSmallIntestineChylomicronsare madehereOmega-3 FattyAcidsAlthough thisdrug can lowerTGs, it does notreduce CVmorbidity andmortality>200mg/dLThis level ofTC on afasting panel isdiagnostic ofhyperlipidemiaPCSK9InhibitorsThe mosteffective drugat loweringLDL and is aninjectablePrimarydyslipidemiaThis disorderoccursindependentof other healthproblemsLifestyleChangesThe bestprevention forthedevelopment ofatherosclerosisTriglyceridesThismoleculemakes up55-65% ofVLDLSecondarydyslipidemiaThis disorderoccurs dueto otherhealthproblemsChylomicronsThislipoproteinprimarilytransportsTGsFibratesGemfibrozilbelongs tothis class ofmedicationsLiverSynthesizesVLDL andHDL fromremants ofcholesterolXanthomasPhysicalexam findingc/whyperlipidemiaHigh-IntensityAtorvastatin40mg isconsideredthis type ofintensitySmokingModifiableRisk Factor foratherosclerosis>500mg/dLThis level ofTGs puts apatient at riskfor developingpancreatitisFattystreakThe initiallesion of anatheroscleroticplaqueAtherosclerosisHardening ofthe arteriesby way ofcholesterolplaques

PA518 Lipid Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. This drug is most effective in increasing HDL
    Niacin
  2. This lipoprotein is made up of 50% cholesterol
    LDL Cholesterol
  3. This lipoprotein is protective against heart disease
    HDL Cholesterol
  4. Optimal level of TGs
    <150mg/dL
  5. ADRs of this drug include elevated liver enzymes, hepatotoxicity, and myalgia
    Statin Side effects
  6. First line treatment for hyperlipidemia
    HMG-CoA Reductase Inhibitors
  7. Ideal level of HDL for men
    >40mg/dL
  8. This class of drugs can interfere with the absorption of Vit D
    Bile Acid Sequestrants
  9. This disorder causes gene mutations for LDL receptors
    Familial Hypercholesterolemia
  10. Target LDL goal for DM pts
    <70mg/dL
  11. Pitavastatin is considered this type of intensities
    Moderate-Intensity
  12. Chylomicrons are made here
    Small Intestine
  13. Although this drug can lower TGs, it does not reduce CV morbidity and mortality
    Omega-3 Fatty Acids
  14. This level of TC on a fasting panel is diagnostic of hyperlipidemia
    >200mg/dL
  15. The most effective drug at lowering LDL and is an injectable
    PCSK9 Inhibitors
  16. This disorder occurs independent of other health problems
    Primary dyslipidemia
  17. The best prevention for the development of atherosclerosis
    Lifestyle Changes
  18. This molecule makes up 55-65% of VLDL
    Triglycerides
  19. This disorder occurs due to other health problems
    Secondary dyslipidemia
  20. This lipoprotein primarily transports TGs
    Chylomicrons
  21. Gemfibrozil belongs to this class of medications
    Fibrates
  22. Synthesizes VLDL and HDL from remants of cholesterol
    Liver
  23. Physical exam finding c/w hyperlipidemia
    Xanthomas
  24. Atorvastatin 40mg is considered this type of intensity
    High-Intensity
  25. Modifiable Risk Factor for atherosclerosis
    Smoking
  26. This level of TGs puts a patient at risk for developing pancreatitis
    >500mg/dL
  27. The initial lesion of an atherosclerotic plaque
    Fatty streak
  28. Hardening of the arteries by way of cholesterol plaques
    Atherosclerosis