Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.The fluorescentproductproduced in theAmplexRedassay is called______.Enhanced NObioavailabilityindicatesimproved______.FST lowersROS levels, asindicated by adecrease in______ levels.Neutralizingantibodiestargetedactivin A and______.Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.What assay didwe use toquantifyhydrogenperoxide?_____________"In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.Hypertension isdefined bysustained bloodpressure above____ mmHg.Follistatinincreases theexpression of______, which iscritical for nitricoxide production.FST wasadministeredevery______ day.Our study showsthat blockingactivin signalingreduces ______production.Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________Whichnormotensivestrain served asour control?_____________Whichhypertensiveanimal modeldid we use?___________Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseFollistatinmust maintainits proper______ to befunctional.What is theendogenousprotein we studiedthat antagonizesactivin?_____________What does theabbreviation'SHR' stand for? _____________Follistatin isknown toneutralizesignaling by______.Our assaydirectlymeasureshydrogen______ levels.FST’santioxidanteffect is lostif it is______.In our study,follistatin wasadministered toSHRs over aperiod of______ weeks.Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.The fluorescentproductproduced in theAmplexRedassay is called______.Enhanced NObioavailabilityindicatesimproved______.FST lowersROS levels, asindicated by adecrease in______ levels.Neutralizingantibodiestargetedactivin A and______.Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.What assay didwe use toquantifyhydrogenperoxide?_____________"In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.Hypertension isdefined bysustained bloodpressure above____ mmHg.Follistatinincreases theexpression of______, which iscritical for nitricoxide production.FST wasadministeredevery______ day.Our study showsthat blockingactivin signalingreduces ______production.Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________Whichnormotensivestrain served asour control?_____________Whichhypertensiveanimal modeldid we use?___________Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseFollistatinmust maintainits proper______ to befunctional.What is theendogenousprotein we studiedthat antagonizesactivin?_____________What does theabbreviation'SHR' stand for? _____________Follistatin isknown toneutralizesignaling by______.Our assaydirectlymeasureshydrogen______ levels.FST’santioxidanteffect is lostif it is______.In our study,follistatin wasadministered toSHRs over aperiod of______ weeks.

Follistatin to the Rescue: Bingo for Lowering Pressure and Defeating ROS - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Follistatin treatment reduced blood pressure by approximately ______ mmHg.
  2. The fluorescent product produced in the AmplexRed assay is called ______.
  3. Enhanced NO bioavailability indicates improved ______.
  4. FST lowers ROS levels, as indicated by a decrease in ______ levels.
  5. Neutralizing antibodies targeted activin A and ______.
  6. Follistatin treatment induces ______ of PVAT, a phenotype associated with improved vascular function.
  7. What assay did we use to quantify hydrogen peroxide? _____________
  8. "In our experiments, the reduction in oxidative stress was measured as a decrease in the fold change of ______.
  9. Hypertension is defined by sustained blood pressure above ____ mmHg.
  10. Follistatin increases the expression of ______, which is critical for nitric oxide production.
  11. FST was administered every ______ day.
  12. Our study shows that blocking activin signaling reduces ______ production.
  13. Which protein marker indicates a brown adipose phenotype in PVAT? _____________
  14. Which type of assay is used to evaluate NO levels in our study: fluorometric or colorimetric? _____________
  15. Which normotensive strain served as our control? _____________
  16. Which hypertensive animal model did we use? ___________
  17. Follistatin retains its antioxidant effect even when denatured. True/False
  18. Follistatin must maintain its proper ______ to be functional.
  19. What is the endogenous protein we studied that antagonizes activin? _____________
  20. What does the abbreviation 'SHR' stand for? _____________
  21. Follistatin is known to neutralize signaling by ______.
  22. Our assay directly measures hydrogen ______ levels.
  23. FST’s antioxidant effect is lost if it is ______.
  24. In our study, follistatin was administered to SHRs over a period of ______ weeks.