Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseNeutralizingantibodiestargetedactivin A and______.Follistatin isknown toneutralizesignaling by______.In our study,follistatin wasadministered toSHRs over aperiod of______ weeks.Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________Whichhypertensiveanimal modeldid we use?___________What assay didwe use toquantifyhydrogenperoxide?_____________Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________What does theabbreviation'SHR' stand for? _____________The fluorescentproductproduced in theAmplexRedassay is called______.Follistatinincreases theexpression of______, which iscritical for nitricoxide production.What is theendogenousprotein we studiedthat antagonizesactivin?_____________"In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.Our study showsthat blockingactivin signalingreduces ______production.Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.Whichnormotensivestrain served asour control?_____________FST lowersROS levels, asindicated by adecrease in______ levels.FST’santioxidanteffect is lostif it is______.Follistatinmust maintainits proper______ to befunctional.Hypertension isdefined bysustained bloodpressure above____ mmHg.Enhanced NObioavailabilityindicatesimproved______.FST wasadministeredevery______ day.Our assaydirectlymeasureshydrogen______ levels.Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseNeutralizingantibodiestargetedactivin A and______.Follistatin isknown toneutralizesignaling by______.In our study,follistatin wasadministered toSHRs over aperiod of______ weeks.Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________Whichhypertensiveanimal modeldid we use?___________What assay didwe use toquantifyhydrogenperoxide?_____________Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________What does theabbreviation'SHR' stand for? _____________The fluorescentproductproduced in theAmplexRedassay is called______.Follistatinincreases theexpression of______, which iscritical for nitricoxide production.What is theendogenousprotein we studiedthat antagonizesactivin?_____________"In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.Our study showsthat blockingactivin signalingreduces ______production.Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.Whichnormotensivestrain served asour control?_____________FST lowersROS levels, asindicated by adecrease in______ levels.FST’santioxidanteffect is lostif it is______.Follistatinmust maintainits proper______ to befunctional.Hypertension isdefined bysustained bloodpressure above____ mmHg.Enhanced NObioavailabilityindicatesimproved______.FST wasadministeredevery______ day.Our assaydirectlymeasureshydrogen______ levels.Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.

Follistatin to the Rescue: Bingo for Lowering Pressure and Defeating ROS - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Follistatin retains its antioxidant effect even when denatured. True/False
  2. Neutralizing antibodies targeted activin A and ______.
  3. Follistatin is known to neutralize signaling by ______.
  4. In our study, follistatin was administered to SHRs over a period of ______ weeks.
  5. Which type of assay is used to evaluate NO levels in our study: fluorometric or colorimetric? _____________
  6. Which hypertensive animal model did we use? ___________
  7. What assay did we use to quantify hydrogen peroxide? _____________
  8. Which protein marker indicates a brown adipose phenotype in PVAT? _____________
  9. What does the abbreviation 'SHR' stand for? _____________
  10. The fluorescent product produced in the AmplexRed assay is called ______.
  11. Follistatin increases the expression of ______, which is critical for nitric oxide production.
  12. What is the endogenous protein we studied that antagonizes activin? _____________
  13. "In our experiments, the reduction in oxidative stress was measured as a decrease in the fold change of ______.
  14. Our study shows that blocking activin signaling reduces ______ production.
  15. Follistatin treatment reduced blood pressure by approximately ______ mmHg.
  16. Which normotensive strain served as our control? _____________
  17. FST lowers ROS levels, as indicated by a decrease in ______ levels.
  18. FST’s antioxidant effect is lost if it is ______.
  19. Follistatin must maintain its proper ______ to be functional.
  20. Hypertension is defined by sustained blood pressure above ____ mmHg.
  21. Enhanced NO bioavailability indicates improved ______.
  22. FST was administered every ______ day.
  23. Our assay directly measures hydrogen ______ levels.
  24. Follistatin treatment induces ______ of PVAT, a phenotype associated with improved vascular function.