The fluorescentproductproduced in theAmplexRedassay is called______.Follistatin isknown toneutralizesignaling by______.Our study showsthat blockingactivin signalingreduces ______production.Whichnormotensivestrain served asour control?_____________Follistatinincreases theexpression of______, which iscritical for nitricoxide production.Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.FST wasadministeredevery______ day.Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseWhat does theabbreviation'SHR' stand for? _____________Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.FST lowersROS levels, asindicated by adecrease in______ levels.What is theendogenousprotein we studiedthat antagonizesactivin?_____________Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________Hypertension isdefined bysustained bloodpressure above____ mmHg.Whichhypertensiveanimal modeldid we use?___________Enhanced NObioavailabilityindicatesimproved______."In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.FST’santioxidanteffect is lostif it is______.Our assaydirectlymeasureshydrogen______ levels.In our study,follistatin wasadministered toSHRs over aperiod of______ weeks.What assay didwe use toquantifyhydrogenperoxide?_____________Follistatinmust maintainits proper______ to befunctional.Neutralizingantibodiestargetedactivin A and______.The fluorescentproductproduced in theAmplexRedassay is called______.Follistatin isknown toneutralizesignaling by______.Our study showsthat blockingactivin signalingreduces ______production.Whichnormotensivestrain served asour control?_____________Follistatinincreases theexpression of______, which iscritical for nitricoxide production.Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.FST wasadministeredevery______ day.Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseWhat does theabbreviation'SHR' stand for? _____________Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.FST lowersROS levels, asindicated by adecrease in______ levels.What is theendogenousprotein we studiedthat antagonizesactivin?_____________Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________Hypertension isdefined bysustained bloodpressure above____ mmHg.Whichhypertensiveanimal modeldid we use?___________Enhanced NObioavailabilityindicatesimproved______."In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.FST’santioxidanteffect is lostif it is______.Our assaydirectlymeasureshydrogen______ levels.In our study,follistatin wasadministered toSHRs over aperiod of______ weeks.What assay didwe use toquantifyhydrogenperoxide?_____________Follistatinmust maintainits proper______ to befunctional.Neutralizingantibodiestargetedactivin A and______.

Follistatin to the Rescue: Bingo for Lowering Pressure and Defeating ROS - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. The fluorescent product produced in the AmplexRed assay is called ______.
  2. Follistatin is known to neutralize signaling by ______.
  3. Our study shows that blocking activin signaling reduces ______ production.
  4. Which normotensive strain served as our control? _____________
  5. Follistatin increases the expression of ______, which is critical for nitric oxide production.
  6. Which type of assay is used to evaluate NO levels in our study: fluorometric or colorimetric? _____________
  7. Follistatin treatment reduced blood pressure by approximately ______ mmHg.
  8. FST was administered every ______ day.
  9. Follistatin retains its antioxidant effect even when denatured. True/False
  10. What does the abbreviation 'SHR' stand for? _____________
  11. Follistatin treatment induces ______ of PVAT, a phenotype associated with improved vascular function.
  12. FST lowers ROS levels, as indicated by a decrease in ______ levels.
  13. What is the endogenous protein we studied that antagonizes activin? _____________
  14. Which protein marker indicates a brown adipose phenotype in PVAT? _____________
  15. Hypertension is defined by sustained blood pressure above ____ mmHg.
  16. Which hypertensive animal model did we use? ___________
  17. Enhanced NO bioavailability indicates improved ______.
  18. "In our experiments, the reduction in oxidative stress was measured as a decrease in the fold change of ______.
  19. FST’s antioxidant effect is lost if it is ______.
  20. Our assay directly measures hydrogen ______ levels.
  21. In our study, follistatin was administered to SHRs over a period of ______ weeks.
  22. What assay did we use to quantify hydrogen peroxide? _____________
  23. Follistatin must maintain its proper ______ to be functional.
  24. Neutralizing antibodies targeted activin A and ______.