Follistatinincreases theexpression of______, which iscritical for nitricoxide production.Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.The fluorescentproductproduced in theAmplexRedassay is called______.What is theendogenousprotein we studiedthat antagonizesactivin?_____________Whichnormotensivestrain served asour control?_____________Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________Follistatinmust maintainits proper______ to befunctional.Our study showsthat blockingactivin signalingreduces ______production.Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.Whichhypertensiveanimal modeldid we use?___________What does theabbreviation'SHR' stand for? _____________Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________FST wasadministeredevery______ day."In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.FST’santioxidanteffect is lostif it is______.Our assaydirectlymeasureshydrogen______ levels.FST lowersROS levels, asindicated by adecrease in______ levels.What assay didwe use toquantifyhydrogenperoxide?_____________Neutralizingantibodiestargetedactivin A and______.Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseIn our study,follistatin wasadministered toSHRs over aperiod of______ weeks.Follistatin isknown toneutralizesignaling by______.Hypertension isdefined bysustained bloodpressure above____ mmHg.Enhanced NObioavailabilityindicatesimproved______.Follistatinincreases theexpression of______, which iscritical for nitricoxide production.Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.The fluorescentproductproduced in theAmplexRedassay is called______.What is theendogenousprotein we studiedthat antagonizesactivin?_____________Whichnormotensivestrain served asour control?_____________Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________Follistatinmust maintainits proper______ to befunctional.Our study showsthat blockingactivin signalingreduces ______production.Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.Whichhypertensiveanimal modeldid we use?___________What does theabbreviation'SHR' stand for? _____________Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________FST wasadministeredevery______ day."In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.FST’santioxidanteffect is lostif it is______.Our assaydirectlymeasureshydrogen______ levels.FST lowersROS levels, asindicated by adecrease in______ levels.What assay didwe use toquantifyhydrogenperoxide?_____________Neutralizingantibodiestargetedactivin A and______.Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseIn our study,follistatin wasadministered toSHRs over aperiod of______ weeks.Follistatin isknown toneutralizesignaling by______.Hypertension isdefined bysustained bloodpressure above____ mmHg.Enhanced NObioavailabilityindicatesimproved______.

Follistatin to the Rescue: Bingo for Lowering Pressure and Defeating ROS - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Follistatin increases the expression of ______, which is critical for nitric oxide production.
  2. Follistatin treatment reduced blood pressure by approximately ______ mmHg.
  3. The fluorescent product produced in the AmplexRed assay is called ______.
  4. What is the endogenous protein we studied that antagonizes activin? _____________
  5. Which normotensive strain served as our control? _____________
  6. Which protein marker indicates a brown adipose phenotype in PVAT? _____________
  7. Follistatin must maintain its proper ______ to be functional.
  8. Our study shows that blocking activin signaling reduces ______ production.
  9. Follistatin treatment induces ______ of PVAT, a phenotype associated with improved vascular function.
  10. Which hypertensive animal model did we use? ___________
  11. What does the abbreviation 'SHR' stand for? _____________
  12. Which type of assay is used to evaluate NO levels in our study: fluorometric or colorimetric? _____________
  13. FST was administered every ______ day.
  14. "In our experiments, the reduction in oxidative stress was measured as a decrease in the fold change of ______.
  15. FST’s antioxidant effect is lost if it is ______.
  16. Our assay directly measures hydrogen ______ levels.
  17. FST lowers ROS levels, as indicated by a decrease in ______ levels.
  18. What assay did we use to quantify hydrogen peroxide? _____________
  19. Neutralizing antibodies targeted activin A and ______.
  20. Follistatin retains its antioxidant effect even when denatured. True/False
  21. In our study, follistatin was administered to SHRs over a period of ______ weeks.
  22. Follistatin is known to neutralize signaling by ______.
  23. Hypertension is defined by sustained blood pressure above ____ mmHg.
  24. Enhanced NO bioavailability indicates improved ______.