Follistatin isknown toneutralizesignaling by______.In our study,follistatin wasadministered toSHRs over aperiod of______ weeks.Whichnormotensivestrain served asour control?_____________Enhanced NObioavailabilityindicatesimproved______.Our assaydirectlymeasureshydrogen______ levels.What assay didwe use toquantifyhydrogenperoxide?_____________Follistatinmust maintainits proper______ to befunctional.Whichhypertensiveanimal modeldid we use?___________The fluorescentproductproduced in theAmplexRedassay is called______.Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.FST’santioxidanteffect is lostif it is______.Hypertension isdefined bysustained bloodpressure above____ mmHg.Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________Follistatinincreases theexpression of______, which iscritical for nitricoxide production."In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseWhat does theabbreviation'SHR' stand for? _____________Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________Neutralizingantibodiestargetedactivin A and______.FST lowersROS levels, asindicated by adecrease in______ levels.What is theendogenousprotein we studiedthat antagonizesactivin?_____________FST wasadministeredevery______ day.Our study showsthat blockingactivin signalingreduces ______production.Follistatin isknown toneutralizesignaling by______.In our study,follistatin wasadministered toSHRs over aperiod of______ weeks.Whichnormotensivestrain served asour control?_____________Enhanced NObioavailabilityindicatesimproved______.Our assaydirectlymeasureshydrogen______ levels.What assay didwe use toquantifyhydrogenperoxide?_____________Follistatinmust maintainits proper______ to befunctional.Whichhypertensiveanimal modeldid we use?___________The fluorescentproductproduced in theAmplexRedassay is called______.Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.FST’santioxidanteffect is lostif it is______.Hypertension isdefined bysustained bloodpressure above____ mmHg.Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________Follistatinincreases theexpression of______, which iscritical for nitricoxide production."In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseWhat does theabbreviation'SHR' stand for? _____________Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________Neutralizingantibodiestargetedactivin A and______.FST lowersROS levels, asindicated by adecrease in______ levels.What is theendogenousprotein we studiedthat antagonizesactivin?_____________FST wasadministeredevery______ day.Our study showsthat blockingactivin signalingreduces ______production.

Follistatin to the Rescue: Bingo for Lowering Pressure and Defeating ROS - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Follistatin is known to neutralize signaling by ______.
  2. In our study, follistatin was administered to SHRs over a period of ______ weeks.
  3. Which normotensive strain served as our control? _____________
  4. Enhanced NO bioavailability indicates improved ______.
  5. Our assay directly measures hydrogen ______ levels.
  6. What assay did we use to quantify hydrogen peroxide? _____________
  7. Follistatin must maintain its proper ______ to be functional.
  8. Which hypertensive animal model did we use? ___________
  9. The fluorescent product produced in the AmplexRed assay is called ______.
  10. Follistatin treatment induces ______ of PVAT, a phenotype associated with improved vascular function.
  11. FST’s antioxidant effect is lost if it is ______.
  12. Hypertension is defined by sustained blood pressure above ____ mmHg.
  13. Which protein marker indicates a brown adipose phenotype in PVAT? _____________
  14. Follistatin increases the expression of ______, which is critical for nitric oxide production.
  15. "In our experiments, the reduction in oxidative stress was measured as a decrease in the fold change of ______.
  16. Follistatin retains its antioxidant effect even when denatured. True/False
  17. What does the abbreviation 'SHR' stand for? _____________
  18. Follistatin treatment reduced blood pressure by approximately ______ mmHg.
  19. Which type of assay is used to evaluate NO levels in our study: fluorometric or colorimetric? _____________
  20. Neutralizing antibodies targeted activin A and ______.
  21. FST lowers ROS levels, as indicated by a decrease in ______ levels.
  22. What is the endogenous protein we studied that antagonizes activin? _____________
  23. FST was administered every ______ day.
  24. Our study shows that blocking activin signaling reduces ______ production.