Follistatinincreases theexpression of______, which iscritical for nitricoxide production."In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.FST wasadministeredevery______ day.Whichhypertensiveanimal modeldid we use?___________The fluorescentproductproduced in theAmplexRedassay is called______.What is theendogenousprotein we studiedthat antagonizesactivin?_____________Enhanced NObioavailabilityindicatesimproved______.Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.FST’santioxidanteffect is lostif it is______.What assay didwe use toquantifyhydrogenperoxide?_____________Hypertension isdefined bysustained bloodpressure above____ mmHg.What does theabbreviation'SHR' stand for? _____________Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.Follistatin isknown toneutralizesignaling by______.Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________Whichnormotensivestrain served asour control?_____________FST lowersROS levels, asindicated by adecrease in______ levels.In our study,follistatin wasadministered toSHRs over aperiod of______ weeks.Our study showsthat blockingactivin signalingreduces ______production.Neutralizingantibodiestargetedactivin A and______.Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseFollistatinmust maintainits proper______ to befunctional.Our assaydirectlymeasureshydrogen______ levels.Follistatinincreases theexpression of______, which iscritical for nitricoxide production."In our experiments,the reduction inoxidative stress wasmeasured as adecrease in the foldchange of ______.FST wasadministeredevery______ day.Whichhypertensiveanimal modeldid we use?___________The fluorescentproductproduced in theAmplexRedassay is called______.What is theendogenousprotein we studiedthat antagonizesactivin?_____________Enhanced NObioavailabilityindicatesimproved______.Follistatintreatmentreduced bloodpressure byapproximately______ mmHg.FST’santioxidanteffect is lostif it is______.What assay didwe use toquantifyhydrogenperoxide?_____________Hypertension isdefined bysustained bloodpressure above____ mmHg.What does theabbreviation'SHR' stand for? _____________Which type of assayis used to evaluateNO levels in ourstudy: fluorometricor colorimetric? _____________Follistatintreatment induces______ of PVAT, aphenotypeassociated withimproved vascularfunction.Follistatin isknown toneutralizesignaling by______.Which proteinmarker indicatesa brown adiposephenotype inPVAT?_____________Whichnormotensivestrain served asour control?_____________FST lowersROS levels, asindicated by adecrease in______ levels.In our study,follistatin wasadministered toSHRs over aperiod of______ weeks.Our study showsthat blockingactivin signalingreduces ______production.Neutralizingantibodiestargetedactivin A and______.Follistatinretains itsantioxidanteffect evenwhen denatured.True/FalseFollistatinmust maintainits proper______ to befunctional.Our assaydirectlymeasureshydrogen______ levels.

Follistatin to the Rescue: Bingo for Lowering Pressure and Defeating ROS - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Follistatin increases the expression of ______, which is critical for nitric oxide production.
  2. "In our experiments, the reduction in oxidative stress was measured as a decrease in the fold change of ______.
  3. FST was administered every ______ day.
  4. Which hypertensive animal model did we use? ___________
  5. The fluorescent product produced in the AmplexRed assay is called ______.
  6. What is the endogenous protein we studied that antagonizes activin? _____________
  7. Enhanced NO bioavailability indicates improved ______.
  8. Follistatin treatment reduced blood pressure by approximately ______ mmHg.
  9. FST’s antioxidant effect is lost if it is ______.
  10. What assay did we use to quantify hydrogen peroxide? _____________
  11. Hypertension is defined by sustained blood pressure above ____ mmHg.
  12. What does the abbreviation 'SHR' stand for? _____________
  13. Which type of assay is used to evaluate NO levels in our study: fluorometric or colorimetric? _____________
  14. Follistatin treatment induces ______ of PVAT, a phenotype associated with improved vascular function.
  15. Follistatin is known to neutralize signaling by ______.
  16. Which protein marker indicates a brown adipose phenotype in PVAT? _____________
  17. Which normotensive strain served as our control? _____________
  18. FST lowers ROS levels, as indicated by a decrease in ______ levels.
  19. In our study, follistatin was administered to SHRs over a period of ______ weeks.
  20. Our study shows that blocking activin signaling reduces ______ production.
  21. Neutralizing antibodies targeted activin A and ______.
  22. Follistatin retains its antioxidant effect even when denatured. True/False
  23. Follistatin must maintain its proper ______ to be functional.
  24. Our assay directly measures hydrogen ______ levels.