EsophagogastricVaricesEngorged anddistended bloodvessels of theesophagus andproximal stomach thatdevelop as a result ofportal hypertensionfrom cirrhosis of theliverAlcoholism&Gallstonemigrationtop twocauses ofacutepancreatitisHematocheziabright redstools,indicatesbleeding fromlower GI tractCullen'sSign, GreyTurner'ssignlate signsofabdominalbleedingElevatedamylase,lipase, andCRP,low mag,K, Ca,hyperglycemialab valuesseen inacutepancreatitisLigamentof Treitzthe surgicallandmark thatdivides upperand lower GItractH.Pyloriinfection&NSAIDSTop twocauses ofPeptic UlcerDisease/UpperGI bleedVasopressinnot a 1st lineagent fortreatment of ulcersand varicealbleeding due tovasoconstrictionside effectsPerforation/PeritonitisComplication fromulcers, presents assevere, acutegeneralize abdominalpain, significantrebound tendernessand rigidityEndoscopygoldstandard toisolate andtreat sourceof GI bleedOctreotideas effective asvasopressin intreating varicealbleeding withminimal sideeffectshypovolemicshock,ARDS, AKI,necrosis,pseudocystssystemic andlocalcomplicationsof acutepancreatitisHigh PT/INR/aPTT,,increased bilirubin,AST, ALP,ammonia, lowalbumin, platelets,NA, K, and glucoselab valuesseen inacute liverfailureCTabdomen,Ultrasoundabdomengoldstandarddiagnosticsfor acutepancreatitisViralhepatitis ,medication-inducedtop 2 causesof acute liverfailure inNorthAmericareplaceelectrolytes andfluids, painmanagement,nutritionsupport,reverseacidosisManagementgoals foracutepancreatitisStress-relatedmucosaldiseasedevelopmentof stress ulcersin critically illpatients due tohypoperfusionHematemesisbright red orbrown "coffeeground" emesis,indicatesbleeding fromupper GI tractMelenablack, tarry, ordark red stools,indicatesbleeding fromupper GI tractneomycin,metronidazole,rifaximin,lactulosedecreaseformation ofammonia oreliminate totreat hepaticencephalopathypainresolves andinflammatorymarkersdecreasingWhen oralfeeding canresume inpancreatitisPPIs & H2antagonistsorderedprophylacticallyto preventdevelopment ofstress ulcerscerebraledema,dysrhythmias,respiratoryfailure, sepsis,AKIComplicationsof acute liverfailureasterixisliver flap, Bestrecognized by downwardflapping of the handswhen the pt extends thearms and dorsiflexes thewrists, indicatespresence of ammonia onthe brain/hepaticencephalopathy EsophagogastricVaricesEngorged anddistended bloodvessels of theesophagus andproximal stomach thatdevelop as a result ofportal hypertensionfrom cirrhosis of theliverAlcoholism&Gallstonemigrationtop twocauses ofacutepancreatitisHematocheziabright redstools,indicatesbleeding fromlower GI tractCullen'sSign, GreyTurner'ssignlate signsofabdominalbleedingElevatedamylase,lipase, andCRP,low mag,K, Ca,hyperglycemialab valuesseen inacutepancreatitisLigamentof Treitzthe surgicallandmark thatdivides upperand lower GItractH.Pyloriinfection&NSAIDSTop twocauses ofPeptic UlcerDisease/UpperGI bleedVasopressinnot a 1st lineagent fortreatment of ulcersand varicealbleeding due tovasoconstrictionside effectsPerforation/PeritonitisComplication fromulcers, presents assevere, acutegeneralize abdominalpain, significantrebound tendernessand rigidityEndoscopygoldstandard toisolate andtreat sourceof GI bleedOctreotideas effective asvasopressin intreating varicealbleeding withminimal sideeffectshypovolemicshock,ARDS, AKI,necrosis,pseudocystssystemic andlocalcomplicationsof acutepancreatitisHigh PT/INR/aPTT,,increased bilirubin,AST, ALP,ammonia, lowalbumin, platelets,NA, K, and glucoselab valuesseen inacute liverfailureCTabdomen,Ultrasoundabdomengoldstandarddiagnosticsfor acutepancreatitisViralhepatitis ,medication-inducedtop 2 causesof acute liverfailure inNorthAmericareplaceelectrolytes andfluids, painmanagement,nutritionsupport,reverseacidosisManagementgoals foracutepancreatitisStress-relatedmucosaldiseasedevelopmentof stress ulcersin critically illpatients due tohypoperfusionHematemesisbright red orbrown "coffeeground" emesis,indicatesbleeding fromupper GI tractMelenablack, tarry, ordark red stools,indicatesbleeding fromupper GI tractneomycin,metronidazole,rifaximin,lactulosedecreaseformation ofammonia oreliminate totreat hepaticencephalopathypainresolves andinflammatorymarkersdecreasingWhen oralfeeding canresume inpancreatitisPPIs & H2antagonistsorderedprophylacticallyto preventdevelopment ofstress ulcerscerebraledema,dysrhythmias,respiratoryfailure, sepsis,AKIComplicationsof acute liverfailureasterixisliver flap, Bestrecognized by downwardflapping of the handswhen the pt extends thearms and dorsiflexes thewrists, indicatespresence of ammonia onthe brain/hepaticencephalopathy 

Gastrointestinal Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Engorged and distended blood vessels of the esophagus and proximal stomach that develop as a result of portal hypertension from cirrhosis of the liver
    Esophagogastric Varices
  2. top two causes of acute pancreatitis
    Alcoholism & Gallstone migration
  3. bright red stools, indicates bleeding from lower GI tract
    Hematochezia
  4. late signs of abdominal bleeding
    Cullen's Sign, Grey Turner's sign
  5. lab values seen in acute pancreatitis
    Elevated amylase, lipase, and CRP,low mag, K, Ca, hyperglycemia
  6. the surgical landmark that divides upper and lower GI tract
    Ligament of Treitz
  7. Top two causes of Peptic Ulcer Disease/Upper GI bleed
    H.Pylori infection & NSAIDS
  8. not a 1st line agent for treatment of ulcers and variceal bleeding due to vasoconstriction side effects
    Vasopressin
  9. Complication from ulcers, presents as severe, acute generalize abdominal pain, significant rebound tenderness and rigidity
    Perforation/Peritonitis
  10. gold standard to isolate and treat source of GI bleed
    Endoscopy
  11. as effective as vasopressin in treating variceal bleeding with minimal side effects
    Octreotide
  12. systemic and local complications of acute pancreatitis
    hypovolemic shock, ARDS, AKI, necrosis, pseudocysts
  13. lab values seen in acute liver failure
    High PT/INR/aPTT,, increased bilirubin, AST, ALP, ammonia, low albumin, platelets, NA, K, and glucose
  14. gold standard diagnostics for acute pancreatitis
    CT abdomen, Ultrasound abdomen
  15. top 2 causes of acute liver failure in North America
    Viral hepatitis , medication-induced
  16. Management goals for acute pancreatitis
    replace electrolytes and fluids, pain management, nutrition support,reverse acidosis
  17. development of stress ulcers in critically ill patients due to hypoperfusion
    Stress-related mucosal disease
  18. bright red or brown "coffee ground" emesis, indicates bleeding from upper GI tract
    Hematemesis
  19. black, tarry, or dark red stools, indicates bleeding from upper GI tract
    Melena
  20. decrease formation of ammonia or eliminate to treat hepatic encephalopathy
    neomycin, metronidazole, rifaximin, lactulose
  21. When oral feeding can resume in pancreatitis
    pain resolves and inflammatory markers decreasing
  22. ordered prophylactically to prevent development of stress ulcers
    PPIs & H2 antagonists
  23. Complications of acute liver failure
    cerebral edema, dysrhythmias, respiratory failure, sepsis, AKI
  24. liver flap, Best recognized by downward flapping of the hands when the pt extends the arms and dorsiflexes the wrists, indicates presence of ammonia on the brain/hepatic encephalopathy
    asterixis