hypovolemicshock,ARDS, AKI,necrosis,pseudocystssystemic andlocalcomplicationsof acutepancreatitisCullen'sSign, GreyTurner'ssignlate signsofabdominalbleedingEsophagogastricVaricesEngorged anddistended bloodvessels of theesophagus andproximal stomach thatdevelop as a result ofportal hypertensionfrom cirrhosis of theliverStress-relatedmucosaldiseasedevelopmentof stress ulcersin critically illpatients due tohypoperfusionH.Pyloriinfection&NSAIDSTop twocauses ofPeptic UlcerDisease/UpperGI bleedEndoscopygoldstandard toisolate andtreat sourceof GI bleedViralhepatitis ,medication-inducedtop 2 causesof acute liverfailure inNorthAmericaAlcoholism&Gallstonemigrationtop twocauses ofacutepancreatitisasterixisliver flap, Bestrecognized by downwardflapping of the handswhen the pt extends thearms and dorsiflexes thewrists, indicatespresence of ammonia onthe brain/hepaticencephalopathy Octreotideas effective asvasopressin intreating varicealbleeding withminimal sideeffectsHematemesisbright red orbrown "coffeeground" emesis,indicatesbleeding fromupper GI tractCTabdomen,Ultrasoundabdomengoldstandarddiagnosticsfor acutepancreatitisElevatedamylase,lipase, andCRP,low mag,K, Ca,hyperglycemialab valuesseen inacutepancreatitisMelenablack, tarry, ordark red stools,indicatesbleeding fromupper GI tractcerebraledema,dysrhythmias,respiratoryfailure, sepsis,AKIComplicationsof acute liverfailurePerforation/PeritonitisComplication fromulcers, presents assevere, acutegeneralize abdominalpain, significantrebound tendernessand rigidityVasopressinnot a 1st lineagent fortreatment of ulcersand varicealbleeding due tovasoconstrictionside effectsreplaceelectrolytes andfluids, painmanagement,nutritionsupport,reverseacidosisManagementgoals foracutepancreatitisHematocheziabright redstools,indicatesbleeding fromlower GI tractLigamentof Treitzthe surgicallandmark thatdivides upperand lower GItractHigh PT/INR/aPTT,,increased bilirubin,AST, ALP,ammonia, lowalbumin, platelets,NA, K, and glucoselab valuesseen inacute liverfailureneomycin,metronidazole,rifaximin,lactulosedecreaseformation ofammonia oreliminate totreat hepaticencephalopathypainresolves andinflammatorymarkersdecreasingWhen oralfeeding canresume inpancreatitisPPIs & H2antagonistsorderedprophylacticallyto preventdevelopment ofstress ulcershypovolemicshock,ARDS, AKI,necrosis,pseudocystssystemic andlocalcomplicationsof acutepancreatitisCullen'sSign, GreyTurner'ssignlate signsofabdominalbleedingEsophagogastricVaricesEngorged anddistended bloodvessels of theesophagus andproximal stomach thatdevelop as a result ofportal hypertensionfrom cirrhosis of theliverStress-relatedmucosaldiseasedevelopmentof stress ulcersin critically illpatients due tohypoperfusionH.Pyloriinfection&NSAIDSTop twocauses ofPeptic UlcerDisease/UpperGI bleedEndoscopygoldstandard toisolate andtreat sourceof GI bleedViralhepatitis ,medication-inducedtop 2 causesof acute liverfailure inNorthAmericaAlcoholism&Gallstonemigrationtop twocauses ofacutepancreatitisasterixisliver flap, Bestrecognized by downwardflapping of the handswhen the pt extends thearms and dorsiflexes thewrists, indicatespresence of ammonia onthe brain/hepaticencephalopathy Octreotideas effective asvasopressin intreating varicealbleeding withminimal sideeffectsHematemesisbright red orbrown "coffeeground" emesis,indicatesbleeding fromupper GI tractCTabdomen,Ultrasoundabdomengoldstandarddiagnosticsfor acutepancreatitisElevatedamylase,lipase, andCRP,low mag,K, Ca,hyperglycemialab valuesseen inacutepancreatitisMelenablack, tarry, ordark red stools,indicatesbleeding fromupper GI tractcerebraledema,dysrhythmias,respiratoryfailure, sepsis,AKIComplicationsof acute liverfailurePerforation/PeritonitisComplication fromulcers, presents assevere, acutegeneralize abdominalpain, significantrebound tendernessand rigidityVasopressinnot a 1st lineagent fortreatment of ulcersand varicealbleeding due tovasoconstrictionside effectsreplaceelectrolytes andfluids, painmanagement,nutritionsupport,reverseacidosisManagementgoals foracutepancreatitisHematocheziabright redstools,indicatesbleeding fromlower GI tractLigamentof Treitzthe surgicallandmark thatdivides upperand lower GItractHigh PT/INR/aPTT,,increased bilirubin,AST, ALP,ammonia, lowalbumin, platelets,NA, K, and glucoselab valuesseen inacute liverfailureneomycin,metronidazole,rifaximin,lactulosedecreaseformation ofammonia oreliminate totreat hepaticencephalopathypainresolves andinflammatorymarkersdecreasingWhen oralfeeding canresume inpancreatitisPPIs & H2antagonistsorderedprophylacticallyto preventdevelopment ofstress ulcers

Gastrointestinal Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. systemic and local complications of acute pancreatitis
    hypovolemic shock, ARDS, AKI, necrosis, pseudocysts
  2. late signs of abdominal bleeding
    Cullen's Sign, Grey Turner's sign
  3. Engorged and distended blood vessels of the esophagus and proximal stomach that develop as a result of portal hypertension from cirrhosis of the liver
    Esophagogastric Varices
  4. development of stress ulcers in critically ill patients due to hypoperfusion
    Stress-related mucosal disease
  5. Top two causes of Peptic Ulcer Disease/Upper GI bleed
    H.Pylori infection & NSAIDS
  6. gold standard to isolate and treat source of GI bleed
    Endoscopy
  7. top 2 causes of acute liver failure in North America
    Viral hepatitis , medication-induced
  8. top two causes of acute pancreatitis
    Alcoholism & Gallstone migration
  9. liver flap, Best recognized by downward flapping of the hands when the pt extends the arms and dorsiflexes the wrists, indicates presence of ammonia on the brain/hepatic encephalopathy
    asterixis
  10. as effective as vasopressin in treating variceal bleeding with minimal side effects
    Octreotide
  11. bright red or brown "coffee ground" emesis, indicates bleeding from upper GI tract
    Hematemesis
  12. gold standard diagnostics for acute pancreatitis
    CT abdomen, Ultrasound abdomen
  13. lab values seen in acute pancreatitis
    Elevated amylase, lipase, and CRP,low mag, K, Ca, hyperglycemia
  14. black, tarry, or dark red stools, indicates bleeding from upper GI tract
    Melena
  15. Complications of acute liver failure
    cerebral edema, dysrhythmias, respiratory failure, sepsis, AKI
  16. Complication from ulcers, presents as severe, acute generalize abdominal pain, significant rebound tenderness and rigidity
    Perforation/Peritonitis
  17. not a 1st line agent for treatment of ulcers and variceal bleeding due to vasoconstriction side effects
    Vasopressin
  18. Management goals for acute pancreatitis
    replace electrolytes and fluids, pain management, nutrition support,reverse acidosis
  19. bright red stools, indicates bleeding from lower GI tract
    Hematochezia
  20. the surgical landmark that divides upper and lower GI tract
    Ligament of Treitz
  21. lab values seen in acute liver failure
    High PT/INR/aPTT,, increased bilirubin, AST, ALP, ammonia, low albumin, platelets, NA, K, and glucose
  22. decrease formation of ammonia or eliminate to treat hepatic encephalopathy
    neomycin, metronidazole, rifaximin, lactulose
  23. When oral feeding can resume in pancreatitis
    pain resolves and inflammatory markers decreasing
  24. ordered prophylactically to prevent development of stress ulcers
    PPIs & H2 antagonists