cerebraledema,dysrhythmias,respiratoryfailure, sepsis,AKIComplicationsof acute liverfailureEndoscopygoldstandard toisolate andtreat sourceof GI bleedEsophagogastricVaricesEngorged anddistended bloodvessels of theesophagus andproximal stomach thatdevelop as a result ofportal hypertensionfrom cirrhosis of theliverLigamentof Treitzthe surgicallandmark thatdivides upperand lower GItractHematemesisbright red orbrown "coffeeground" emesis,indicatesbleeding fromupper GI tractpainresolves andinflammatorymarkersdecreasingWhen oralfeeding canresume inpancreatitisStress-relatedmucosaldiseasedevelopmentof stress ulcersin critically illpatients due tohypoperfusionPPIs & H2antagonistsorderedprophylacticallyto preventdevelopment ofstress ulcersCullen'sSign, GreyTurner'ssignlate signsofabdominalbleedingasterixisliver flap, Bestrecognized by downwardflapping of the handswhen the pt extends thearms and dorsiflexes thewrists, indicatespresence of ammonia onthe brain/hepaticencephalopathy Vasopressinnot a 1st lineagent fortreatment of ulcersand varicealbleeding due tovasoconstrictionside effectsElevatedamylase,lipase, andCRP,low mag,K, Ca,hyperglycemialab valuesseen inacutepancreatitisCTabdomen,Ultrasoundabdomengoldstandarddiagnosticsfor acutepancreatitisAlcoholism&Gallstonemigrationtop twocauses ofacutepancreatitishypovolemicshock,ARDS, AKI,necrosis,pseudocystssystemic andlocalcomplicationsof acutepancreatitisHematocheziabright redstools,indicatesbleeding fromlower GI tractneomycin,metronidazole,rifaximin,lactulosedecreaseformation ofammonia oreliminate totreat hepaticencephalopathyHigh PT/INR/aPTT,,increased bilirubin,AST, ALP,ammonia, lowalbumin, platelets,NA, K, and glucoselab valuesseen inacute liverfailureOctreotideas effective asvasopressin intreating varicealbleeding withminimal sideeffectsPerforation/PeritonitisComplication fromulcers, presents assevere, acutegeneralize abdominalpain, significantrebound tendernessand rigidityMelenablack, tarry, ordark red stools,indicatesbleeding fromupper GI tractViralhepatitis ,medication-inducedtop 2 causesof acute liverfailure inNorthAmericaH.Pyloriinfection&NSAIDSTop twocauses ofPeptic UlcerDisease/UpperGI bleedreplaceelectrolytes andfluids, painmanagement,nutritionsupport,reverseacidosisManagementgoals foracutepancreatitiscerebraledema,dysrhythmias,respiratoryfailure, sepsis,AKIComplicationsof acute liverfailureEndoscopygoldstandard toisolate andtreat sourceof GI bleedEsophagogastricVaricesEngorged anddistended bloodvessels of theesophagus andproximal stomach thatdevelop as a result ofportal hypertensionfrom cirrhosis of theliverLigamentof Treitzthe surgicallandmark thatdivides upperand lower GItractHematemesisbright red orbrown "coffeeground" emesis,indicatesbleeding fromupper GI tractpainresolves andinflammatorymarkersdecreasingWhen oralfeeding canresume inpancreatitisStress-relatedmucosaldiseasedevelopmentof stress ulcersin critically illpatients due tohypoperfusionPPIs & H2antagonistsorderedprophylacticallyto preventdevelopment ofstress ulcersCullen'sSign, GreyTurner'ssignlate signsofabdominalbleedingasterixisliver flap, Bestrecognized by downwardflapping of the handswhen the pt extends thearms and dorsiflexes thewrists, indicatespresence of ammonia onthe brain/hepaticencephalopathy Vasopressinnot a 1st lineagent fortreatment of ulcersand varicealbleeding due tovasoconstrictionside effectsElevatedamylase,lipase, andCRP,low mag,K, Ca,hyperglycemialab valuesseen inacutepancreatitisCTabdomen,Ultrasoundabdomengoldstandarddiagnosticsfor acutepancreatitisAlcoholism&Gallstonemigrationtop twocauses ofacutepancreatitishypovolemicshock,ARDS, AKI,necrosis,pseudocystssystemic andlocalcomplicationsof acutepancreatitisHematocheziabright redstools,indicatesbleeding fromlower GI tractneomycin,metronidazole,rifaximin,lactulosedecreaseformation ofammonia oreliminate totreat hepaticencephalopathyHigh PT/INR/aPTT,,increased bilirubin,AST, ALP,ammonia, lowalbumin, platelets,NA, K, and glucoselab valuesseen inacute liverfailureOctreotideas effective asvasopressin intreating varicealbleeding withminimal sideeffectsPerforation/PeritonitisComplication fromulcers, presents assevere, acutegeneralize abdominalpain, significantrebound tendernessand rigidityMelenablack, tarry, ordark red stools,indicatesbleeding fromupper GI tractViralhepatitis ,medication-inducedtop 2 causesof acute liverfailure inNorthAmericaH.Pyloriinfection&NSAIDSTop twocauses ofPeptic UlcerDisease/UpperGI bleedreplaceelectrolytes andfluids, painmanagement,nutritionsupport,reverseacidosisManagementgoals foracutepancreatitis

Gastrointestinal Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Complications of acute liver failure
    cerebral edema, dysrhythmias, respiratory failure, sepsis, AKI
  2. gold standard to isolate and treat source of GI bleed
    Endoscopy
  3. Engorged and distended blood vessels of the esophagus and proximal stomach that develop as a result of portal hypertension from cirrhosis of the liver
    Esophagogastric Varices
  4. the surgical landmark that divides upper and lower GI tract
    Ligament of Treitz
  5. bright red or brown "coffee ground" emesis, indicates bleeding from upper GI tract
    Hematemesis
  6. When oral feeding can resume in pancreatitis
    pain resolves and inflammatory markers decreasing
  7. development of stress ulcers in critically ill patients due to hypoperfusion
    Stress-related mucosal disease
  8. ordered prophylactically to prevent development of stress ulcers
    PPIs & H2 antagonists
  9. late signs of abdominal bleeding
    Cullen's Sign, Grey Turner's sign
  10. liver flap, Best recognized by downward flapping of the hands when the pt extends the arms and dorsiflexes the wrists, indicates presence of ammonia on the brain/hepatic encephalopathy
    asterixis
  11. not a 1st line agent for treatment of ulcers and variceal bleeding due to vasoconstriction side effects
    Vasopressin
  12. lab values seen in acute pancreatitis
    Elevated amylase, lipase, and CRP,low mag, K, Ca, hyperglycemia
  13. gold standard diagnostics for acute pancreatitis
    CT abdomen, Ultrasound abdomen
  14. top two causes of acute pancreatitis
    Alcoholism & Gallstone migration
  15. systemic and local complications of acute pancreatitis
    hypovolemic shock, ARDS, AKI, necrosis, pseudocysts
  16. bright red stools, indicates bleeding from lower GI tract
    Hematochezia
  17. decrease formation of ammonia or eliminate to treat hepatic encephalopathy
    neomycin, metronidazole, rifaximin, lactulose
  18. lab values seen in acute liver failure
    High PT/INR/aPTT,, increased bilirubin, AST, ALP, ammonia, low albumin, platelets, NA, K, and glucose
  19. as effective as vasopressin in treating variceal bleeding with minimal side effects
    Octreotide
  20. Complication from ulcers, presents as severe, acute generalize abdominal pain, significant rebound tenderness and rigidity
    Perforation/Peritonitis
  21. black, tarry, or dark red stools, indicates bleeding from upper GI tract
    Melena
  22. top 2 causes of acute liver failure in North America
    Viral hepatitis , medication-induced
  23. Top two causes of Peptic Ulcer Disease/Upper GI bleed
    H.Pylori infection & NSAIDS
  24. Management goals for acute pancreatitis
    replace electrolytes and fluids, pain management, nutrition support,reverse acidosis