Hematocheziabright redstools,indicatesbleeding fromlower GI tractneomycin,metronidazole,rifaximin,lactulosedecreaseformation ofammonia oreliminate totreat hepaticencephalopathyLigamentof Treitzthe surgicallandmark thatdivides upperand lower GItractPPIs & H2antagonistsorderedprophylacticallyto preventdevelopment ofstress ulcersViralhepatitis ,medication-inducedtop 2 causesof acute liverfailure inNorthAmericacerebraledema,dysrhythmias,respiratoryfailure, sepsis,AKIComplicationsof acute liverfailureAlcoholism&Gallstonemigrationtop twocauses ofacutepancreatitisasterixisliver flap, Bestrecognized by downwardflapping of the handswhen the pt extends thearms and dorsiflexes thewrists, indicatespresence of ammonia onthe brain/hepaticencephalopathy Cullen'sSign, GreyTurner'ssignlate signsofabdominalbleedingEsophagogastricVaricesEngorged anddistended bloodvessels of theesophagus andproximal stomach thatdevelop as a result ofportal hypertensionfrom cirrhosis of theliverpainresolves andinflammatorymarkersdecreasingWhen oralfeeding canresume inpancreatitishypovolemicshock,ARDS, AKI,necrosis,pseudocystssystemic andlocalcomplicationsof acutepancreatitisElevatedamylase,lipase, andCRP,low mag,K, Ca,hyperglycemialab valuesseen inacutepancreatitisCTabdomen,Ultrasoundabdomengoldstandarddiagnosticsfor acutepancreatitisreplaceelectrolytes andfluids, painmanagement,nutritionsupport,reverseacidosisManagementgoals foracutepancreatitisStress-relatedmucosaldiseasedevelopmentof stress ulcersin critically illpatients due tohypoperfusionHematemesisbright red orbrown "coffeeground" emesis,indicatesbleeding fromupper GI tractMelenablack, tarry, ordark red stools,indicatesbleeding fromupper GI tractH.Pyloriinfection&NSAIDSTop twocauses ofPeptic UlcerDisease/UpperGI bleedOctreotideas effective asvasopressin intreating varicealbleeding withminimal sideeffectsEndoscopygoldstandard toisolate andtreat sourceof GI bleedHigh PT/INR/aPTT,,increased bilirubin,AST, ALP,ammonia, lowalbumin, platelets,NA, K, and glucoselab valuesseen inacute liverfailurePerforation/PeritonitisComplication fromulcers, presents assevere, acutegeneralize abdominalpain, significantrebound tendernessand rigidityVasopressinnot a 1st lineagent fortreatment of ulcersand varicealbleeding due tovasoconstrictionside effectsHematocheziabright redstools,indicatesbleeding fromlower GI tractneomycin,metronidazole,rifaximin,lactulosedecreaseformation ofammonia oreliminate totreat hepaticencephalopathyLigamentof Treitzthe surgicallandmark thatdivides upperand lower GItractPPIs & H2antagonistsorderedprophylacticallyto preventdevelopment ofstress ulcersViralhepatitis ,medication-inducedtop 2 causesof acute liverfailure inNorthAmericacerebraledema,dysrhythmias,respiratoryfailure, sepsis,AKIComplicationsof acute liverfailureAlcoholism&Gallstonemigrationtop twocauses ofacutepancreatitisasterixisliver flap, Bestrecognized by downwardflapping of the handswhen the pt extends thearms and dorsiflexes thewrists, indicatespresence of ammonia onthe brain/hepaticencephalopathy Cullen'sSign, GreyTurner'ssignlate signsofabdominalbleedingEsophagogastricVaricesEngorged anddistended bloodvessels of theesophagus andproximal stomach thatdevelop as a result ofportal hypertensionfrom cirrhosis of theliverpainresolves andinflammatorymarkersdecreasingWhen oralfeeding canresume inpancreatitishypovolemicshock,ARDS, AKI,necrosis,pseudocystssystemic andlocalcomplicationsof acutepancreatitisElevatedamylase,lipase, andCRP,low mag,K, Ca,hyperglycemialab valuesseen inacutepancreatitisCTabdomen,Ultrasoundabdomengoldstandarddiagnosticsfor acutepancreatitisreplaceelectrolytes andfluids, painmanagement,nutritionsupport,reverseacidosisManagementgoals foracutepancreatitisStress-relatedmucosaldiseasedevelopmentof stress ulcersin critically illpatients due tohypoperfusionHematemesisbright red orbrown "coffeeground" emesis,indicatesbleeding fromupper GI tractMelenablack, tarry, ordark red stools,indicatesbleeding fromupper GI tractH.Pyloriinfection&NSAIDSTop twocauses ofPeptic UlcerDisease/UpperGI bleedOctreotideas effective asvasopressin intreating varicealbleeding withminimal sideeffectsEndoscopygoldstandard toisolate andtreat sourceof GI bleedHigh PT/INR/aPTT,,increased bilirubin,AST, ALP,ammonia, lowalbumin, platelets,NA, K, and glucoselab valuesseen inacute liverfailurePerforation/PeritonitisComplication fromulcers, presents assevere, acutegeneralize abdominalpain, significantrebound tendernessand rigidityVasopressinnot a 1st lineagent fortreatment of ulcersand varicealbleeding due tovasoconstrictionside effects

Gastrointestinal Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. bright red stools, indicates bleeding from lower GI tract
    Hematochezia
  2. decrease formation of ammonia or eliminate to treat hepatic encephalopathy
    neomycin, metronidazole, rifaximin, lactulose
  3. the surgical landmark that divides upper and lower GI tract
    Ligament of Treitz
  4. ordered prophylactically to prevent development of stress ulcers
    PPIs & H2 antagonists
  5. top 2 causes of acute liver failure in North America
    Viral hepatitis , medication-induced
  6. Complications of acute liver failure
    cerebral edema, dysrhythmias, respiratory failure, sepsis, AKI
  7. top two causes of acute pancreatitis
    Alcoholism & Gallstone migration
  8. liver flap, Best recognized by downward flapping of the hands when the pt extends the arms and dorsiflexes the wrists, indicates presence of ammonia on the brain/hepatic encephalopathy
    asterixis
  9. late signs of abdominal bleeding
    Cullen's Sign, Grey Turner's sign
  10. Engorged and distended blood vessels of the esophagus and proximal stomach that develop as a result of portal hypertension from cirrhosis of the liver
    Esophagogastric Varices
  11. When oral feeding can resume in pancreatitis
    pain resolves and inflammatory markers decreasing
  12. systemic and local complications of acute pancreatitis
    hypovolemic shock, ARDS, AKI, necrosis, pseudocysts
  13. lab values seen in acute pancreatitis
    Elevated amylase, lipase, and CRP,low mag, K, Ca, hyperglycemia
  14. gold standard diagnostics for acute pancreatitis
    CT abdomen, Ultrasound abdomen
  15. Management goals for acute pancreatitis
    replace electrolytes and fluids, pain management, nutrition support,reverse acidosis
  16. development of stress ulcers in critically ill patients due to hypoperfusion
    Stress-related mucosal disease
  17. bright red or brown "coffee ground" emesis, indicates bleeding from upper GI tract
    Hematemesis
  18. black, tarry, or dark red stools, indicates bleeding from upper GI tract
    Melena
  19. Top two causes of Peptic Ulcer Disease/Upper GI bleed
    H.Pylori infection & NSAIDS
  20. as effective as vasopressin in treating variceal bleeding with minimal side effects
    Octreotide
  21. gold standard to isolate and treat source of GI bleed
    Endoscopy
  22. lab values seen in acute liver failure
    High PT/INR/aPTT,, increased bilirubin, AST, ALP, ammonia, low albumin, platelets, NA, K, and glucose
  23. Complication from ulcers, presents as severe, acute generalize abdominal pain, significant rebound tenderness and rigidity
    Perforation/Peritonitis
  24. not a 1st line agent for treatment of ulcers and variceal bleeding due to vasoconstriction side effects
    Vasopressin