neomycin,metronidazole,rifaximin,lactulosedecreaseformation ofammonia oreliminate totreat hepaticencephalopathyPerforation/PeritonitisComplication fromulcers, presents assevere, acutegeneralize abdominalpain, significantrebound tendernessand rigidityCullen'sSign, GreyTurner'ssignlate signsofabdominalbleedingEndoscopygoldstandard toisolate andtreat sourceof GI bleedAlcoholism&Gallstonemigrationtop twocauses ofacutepancreatitisOctreotideas effective asvasopressin intreating varicealbleeding withminimal sideeffectsasterixisliver flap, Bestrecognized by downwardflapping of the handswhen the pt extends thearms and dorsiflexes thewrists, indicatespresence of ammonia onthe brain/hepaticencephalopathy Hematemesisbright red orbrown "coffeeground" emesis,indicatesbleeding fromupper GI tractHigh PT/INR/aPTT,,increased bilirubin,AST, ALP,ammonia, lowalbumin, platelets,NA, K, and glucoselab valuesseen inacute liverfailureCTabdomen,Ultrasoundabdomengoldstandarddiagnosticsfor acutepancreatitisreplaceelectrolytes andfluids, painmanagement,nutritionsupport,reverseacidosisManagementgoals foracutepancreatitisStress-relatedmucosaldiseasedevelopmentof stress ulcersin critically illpatients due tohypoperfusionElevatedamylase,lipase, andCRP,low mag,K, Ca,hyperglycemialab valuesseen inacutepancreatitiscerebraledema,dysrhythmias,respiratoryfailure, sepsis,AKIComplicationsof acute liverfailureHematocheziabright redstools,indicatesbleeding fromlower GI tracthypovolemicshock,ARDS, AKI,necrosis,pseudocystssystemic andlocalcomplicationsof acutepancreatitisVasopressinnot a 1st lineagent fortreatment of ulcersand varicealbleeding due tovasoconstrictionside effectsViralhepatitis ,medication-inducedtop 2 causesof acute liverfailure inNorthAmericapainresolves andinflammatorymarkersdecreasingWhen oralfeeding canresume inpancreatitisH.Pyloriinfection&NSAIDSTop twocauses ofPeptic UlcerDisease/UpperGI bleedEsophagogastricVaricesEngorged anddistended bloodvessels of theesophagus andproximal stomach thatdevelop as a result ofportal hypertensionfrom cirrhosis of theliverPPIs & H2antagonistsorderedprophylacticallyto preventdevelopment ofstress ulcersLigamentof Treitzthe surgicallandmark thatdivides upperand lower GItractMelenablack, tarry, ordark red stools,indicatesbleeding fromupper GI tractneomycin,metronidazole,rifaximin,lactulosedecreaseformation ofammonia oreliminate totreat hepaticencephalopathyPerforation/PeritonitisComplication fromulcers, presents assevere, acutegeneralize abdominalpain, significantrebound tendernessand rigidityCullen'sSign, GreyTurner'ssignlate signsofabdominalbleedingEndoscopygoldstandard toisolate andtreat sourceof GI bleedAlcoholism&Gallstonemigrationtop twocauses ofacutepancreatitisOctreotideas effective asvasopressin intreating varicealbleeding withminimal sideeffectsasterixisliver flap, Bestrecognized by downwardflapping of the handswhen the pt extends thearms and dorsiflexes thewrists, indicatespresence of ammonia onthe brain/hepaticencephalopathy Hematemesisbright red orbrown "coffeeground" emesis,indicatesbleeding fromupper GI tractHigh PT/INR/aPTT,,increased bilirubin,AST, ALP,ammonia, lowalbumin, platelets,NA, K, and glucoselab valuesseen inacute liverfailureCTabdomen,Ultrasoundabdomengoldstandarddiagnosticsfor acutepancreatitisreplaceelectrolytes andfluids, painmanagement,nutritionsupport,reverseacidosisManagementgoals foracutepancreatitisStress-relatedmucosaldiseasedevelopmentof stress ulcersin critically illpatients due tohypoperfusionElevatedamylase,lipase, andCRP,low mag,K, Ca,hyperglycemialab valuesseen inacutepancreatitiscerebraledema,dysrhythmias,respiratoryfailure, sepsis,AKIComplicationsof acute liverfailureHematocheziabright redstools,indicatesbleeding fromlower GI tracthypovolemicshock,ARDS, AKI,necrosis,pseudocystssystemic andlocalcomplicationsof acutepancreatitisVasopressinnot a 1st lineagent fortreatment of ulcersand varicealbleeding due tovasoconstrictionside effectsViralhepatitis ,medication-inducedtop 2 causesof acute liverfailure inNorthAmericapainresolves andinflammatorymarkersdecreasingWhen oralfeeding canresume inpancreatitisH.Pyloriinfection&NSAIDSTop twocauses ofPeptic UlcerDisease/UpperGI bleedEsophagogastricVaricesEngorged anddistended bloodvessels of theesophagus andproximal stomach thatdevelop as a result ofportal hypertensionfrom cirrhosis of theliverPPIs & H2antagonistsorderedprophylacticallyto preventdevelopment ofstress ulcersLigamentof Treitzthe surgicallandmark thatdivides upperand lower GItractMelenablack, tarry, ordark red stools,indicatesbleeding fromupper GI tract

Gastrointestinal Disorders - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. decrease formation of ammonia or eliminate to treat hepatic encephalopathy
    neomycin, metronidazole, rifaximin, lactulose
  2. Complication from ulcers, presents as severe, acute generalize abdominal pain, significant rebound tenderness and rigidity
    Perforation/Peritonitis
  3. late signs of abdominal bleeding
    Cullen's Sign, Grey Turner's sign
  4. gold standard to isolate and treat source of GI bleed
    Endoscopy
  5. top two causes of acute pancreatitis
    Alcoholism & Gallstone migration
  6. as effective as vasopressin in treating variceal bleeding with minimal side effects
    Octreotide
  7. liver flap, Best recognized by downward flapping of the hands when the pt extends the arms and dorsiflexes the wrists, indicates presence of ammonia on the brain/hepatic encephalopathy
    asterixis
  8. bright red or brown "coffee ground" emesis, indicates bleeding from upper GI tract
    Hematemesis
  9. lab values seen in acute liver failure
    High PT/INR/aPTT,, increased bilirubin, AST, ALP, ammonia, low albumin, platelets, NA, K, and glucose
  10. gold standard diagnostics for acute pancreatitis
    CT abdomen, Ultrasound abdomen
  11. Management goals for acute pancreatitis
    replace electrolytes and fluids, pain management, nutrition support,reverse acidosis
  12. development of stress ulcers in critically ill patients due to hypoperfusion
    Stress-related mucosal disease
  13. lab values seen in acute pancreatitis
    Elevated amylase, lipase, and CRP,low mag, K, Ca, hyperglycemia
  14. Complications of acute liver failure
    cerebral edema, dysrhythmias, respiratory failure, sepsis, AKI
  15. bright red stools, indicates bleeding from lower GI tract
    Hematochezia
  16. systemic and local complications of acute pancreatitis
    hypovolemic shock, ARDS, AKI, necrosis, pseudocysts
  17. not a 1st line agent for treatment of ulcers and variceal bleeding due to vasoconstriction side effects
    Vasopressin
  18. top 2 causes of acute liver failure in North America
    Viral hepatitis , medication-induced
  19. When oral feeding can resume in pancreatitis
    pain resolves and inflammatory markers decreasing
  20. Top two causes of Peptic Ulcer Disease/Upper GI bleed
    H.Pylori infection & NSAIDS
  21. Engorged and distended blood vessels of the esophagus and proximal stomach that develop as a result of portal hypertension from cirrhosis of the liver
    Esophagogastric Varices
  22. ordered prophylactically to prevent development of stress ulcers
    PPIs & H2 antagonists
  23. the surgical landmark that divides upper and lower GI tract
    Ligament of Treitz
  24. black, tarry, or dark red stools, indicates bleeding from upper GI tract
    Melena