Type 2Diabetes:Blood sugar drops duringthe night (often due toexcess insulin before bed),and the body compensatesby raising blood glucose,resulting in high morningglucose. Managed byadjusting insulin dosage oradding a bedtime snack.Insulinstimulators (e.g.,glipizide) helpthe pancreasrelease moreinsulin.Symptoms: Suddenweight loss, frequenturination (polyuria),extreme thirst(polydipsia), andincreased hunger(polyphagia).Type 2Diabetes:Common inpeople under30 and oftendiagnosedabruptly.Biguanides(e.g., metformin)decreaseglucoseproduction in theliver.Intermediate-Acting Insulin(e.g., NPH): Takes1-2 hours to startworking; used tocover blood sugarbetween meals.Type 1: Family history,genetic predisposition,autoimmuneconditions, andsometimes followingviral infections. It’smore common in thoseunder 30.Symptoms:Often similar toType 1 (polyuria,polydipsia,polyphagia), butless noticeable.The liver stores glucose inthe form of glycogen. Whenblood glucose levels drop,glucagon from thepancreas tells the liver torelease stored glucose,raising blood glucoselevels. When blood glucoseis high, insulin encouragesthe liver to storUrineScreening:PancreasFunctions:HyperosmolarHyperglycemicState (HHS):DawnPhenomenon:ExocrineFunction: Thisinvolvesproducingenzymes thathelp digest food.MedicationTypes andInsulinAdministrationDiet: Emphasizebalanced meals,carb counting,and limitingsugar intake.InsulinSensitizers (e.g.,pioglitazone)make cells moreresponsive toinsulin.Types ofDiabetes:Beta cells: These cellsrelease insulin and amylinwhen blood glucose ishigh. Insulin helps cellsabsorb glucose for energy,while amylin slows downthe absorption of glucoseand controls blood sugarspikes.Checks for glucoseand ketones in theurine, as elevatedlevels may indicatediabetes oruncontrolled bloodglucose.Sick Day Rules:Maintain bloodsugar checks, evenif not eating much,as stress andillness can increaseblood sugar.Treatment:Insulin IV,fluids, andmonitoringelectrolytes.Higher risk forHyperosmolarHyperglycemic State(HHS), where bloodsugar risesdangerously highwithout the presence ofketones (no acidosis).Hyperglycemia (HighBlood Sugar): Whenblood glucose rises,the pancreasreleases insulin tohelp cells absorbglucose and lowerblood sugar levels.Type 2Diabetes:Type 1Diabetes:Reflects averageblood glucoselevels over thepast 3 months. Anormal A1C isbetween 4-6%.Insulin Resistance:The body producesinsulin but doesn'tuse it effectively,often due to obesity,inactivity, andgenetic factors.Blood GlucoseMonitoring:Educate onfrequency andtechniques.Rapid-ActingInsulin (e.g.,Humalog): Workswithin 15 minutesand is givenbefore or withmeals.Blood sugar risesin the earlymorning due tohormone release.Managed byadjusting insulintiming.TypesofInsulin:Signs andSymptomsQuick onset ofsymptoms, includingweight loss,increased appetite,and the "3 Ps"(polyuria, polydipsia,polyphagia).Type 1Diabetes:DiabeticKetoacidosis(DKA):Symptoms:Extreme thirst,confusion, drymouth, noketones.Weight gain iscommon, andHHS is a riskdue to highblood glucosewithout ketones.Short-Acting Insulin(e.g., Regularinsulin): Takes 30-60 minutes to workand is used formeal coverage andDKA emergencies.Diet, exercise,and potentiallyoralmedicationsthat help lowerblood glucose.Common onsetis around age50 and tends todevelopgradually.Type 1Diabetes:Key LabValues andDiagnosticTestsSomepatients mayeventuallyneed insulin.Treatment:Insulin IV,fluids, andelectrolytereplacement.NormalFastingLevel: Lessthan 100mg/dL.Alpha cells: These cellsrelease glucagon whenblood glucose is low.Glucagon signals theliver to convert storedglycogen back intoglucose and release itinto the bloodstream,raising blood sugar.Should bebelow 140mg/dL after 2hours ofconsumingglucose.Cause: Extremelyhigh bloodglucose withdehydration,typically in Type2.Liver'sRole:Focus onlifestylechangesfirst:Foot Care: Inspectfeet daily for cuts,blisters, orinfections; wearproper footwear;avoid barefootwalking.NursingInterventions:Hypoglycemia (LowBlood Sugar): Whenblood glucose drops,the pancreasreleases glucagon tosignal the liver torelease glucose.Regularinsulininjections oran insulinpump.GlucoseToleranceTest:Complicationsof DiabetesDiabetesRiskFactorsEndocrine Function:The pancreas alsoregulates blood sugar(glucose) levels. Thishappens through tinyclusters called theIslets of Langerhans,which contain twocritical types of cells:BloodGlucoseBasics:What isDiabetesMellitus?Exercise: Canlower bloodglucose butrequires carefulplanning to avoidhypoglycemia.Insulin isessential.Treatmentinvolves:Normal Range: Bloodglucose should bebetween 70-110 mg/dL.The brain relies on asteady glucose supply,as it cannot storeglucose, making bloodglucose regulationcrucial for brain function.Cause: Highblood glucosewith ketoneproduction (dueto lack ofinsulin).OralHypoglycemicsfor Type 2:AutoimmuneCondition: Theimmune systemattacks and destroysbeta cells in thepancreas, whichstops the productionof insulin.Type 2: Obesity (high BMI),sedentary lifestyle, familyhistory, certain ethnicities(Hispanic, AfricanAmerican, NativeAmerican, Asian American,Pacific Islander), andhistory of gestationaldiabetes. Symptomsusually develop gradually.Higher risk forDiabetic Ketoacidosis(DKA), a life-threatening conditionwhere bloodbecomes acidic dueto a lack of insulin.BloodGlucose:Long-Acting Insulin(e.g., Lantus):Works slowlywithout peaks,providing steadyinsulin levels over12-24 hours.Symptoms:Rapid breathing(Kussmaul),fruity breath,nausea,dehydration.SomogyiEffect:UnderstandingDiabetes andthe Pancreas'RoleGradualonset, withsymptomsoften lessnoticeable.Diet controlthroughcarbohydratecounting.Diabetes Mellitus is acondition where thebody cannot regulateblood glucoseproperly, resulting inchronichyperglycemia (highblood sugar).UnderstandingDawnPhenomenonvs. SomogyiEffectHemoglobinA1C:Higher riskfor DKA dueto thecomplete lackof insulin.TherapeuticManagementand NursingInterventionsMonitoringblood sugarmultipletimes daily.DiabetesMellitus(DM): TypesandDifferencesType 2Diabetes:Blood sugar drops duringthe night (often due toexcess insulin before bed),and the body compensatesby raising blood glucose,resulting in high morningglucose. Managed byadjusting insulin dosage oradding a bedtime snack.Insulinstimulators (e.g.,glipizide) helpthe pancreasrelease moreinsulin.Symptoms: Suddenweight loss, frequenturination (polyuria),extreme thirst(polydipsia), andincreased hunger(polyphagia).Type 2Diabetes:Common inpeople under30 and oftendiagnosedabruptly.Biguanides(e.g., metformin)decreaseglucoseproduction in theliver.Intermediate-Acting Insulin(e.g., NPH): Takes1-2 hours to startworking; used tocover blood sugarbetween meals.Type 1: Family history,genetic predisposition,autoimmuneconditions, andsometimes followingviral infections. It’smore common in thoseunder 30.Symptoms:Often similar toType 1 (polyuria,polydipsia,polyphagia), butless noticeable.The liver stores glucose inthe form of glycogen. Whenblood glucose levels drop,glucagon from thepancreas tells the liver torelease stored glucose,raising blood glucoselevels. When blood glucoseis high, insulin encouragesthe liver to storUrineScreening:PancreasFunctions:HyperosmolarHyperglycemicState (HHS):DawnPhenomenon:ExocrineFunction: Thisinvolvesproducingenzymes thathelp digest food.MedicationTypes andInsulinAdministrationDiet: Emphasizebalanced meals,carb counting,and limitingsugar intake.InsulinSensitizers (e.g.,pioglitazone)make cells moreresponsive toinsulin.Types ofDiabetes:Beta cells: These cellsrelease insulin and amylinwhen blood glucose ishigh. Insulin helps cellsabsorb glucose for energy,while amylin slows downthe absorption of glucoseand controls blood sugarspikes.Checks for glucoseand ketones in theurine, as elevatedlevels may indicatediabetes oruncontrolled bloodglucose.Sick Day Rules:Maintain bloodsugar checks, evenif not eating much,as stress andillness can increaseblood sugar.Treatment:Insulin IV,fluids, andmonitoringelectrolytes.Higher risk forHyperosmolarHyperglycemic State(HHS), where bloodsugar risesdangerously highwithout the presence ofketones (no acidosis).Hyperglycemia (HighBlood Sugar): Whenblood glucose rises,the pancreasreleases insulin tohelp cells absorbglucose and lowerblood sugar levels.Type 2Diabetes:Type 1Diabetes:Reflects averageblood glucoselevels over thepast 3 months. Anormal A1C isbetween 4-6%.Insulin Resistance:The body producesinsulin but doesn'tuse it effectively,often due to obesity,inactivity, andgenetic factors.Blood GlucoseMonitoring:Educate onfrequency andtechniques.Rapid-ActingInsulin (e.g.,Humalog): Workswithin 15 minutesand is givenbefore or withmeals.Blood sugar risesin the earlymorning due tohormone release.Managed byadjusting insulintiming.TypesofInsulin:Signs andSymptomsQuick onset ofsymptoms, includingweight loss,increased appetite,and the "3 Ps"(polyuria, polydipsia,polyphagia).Type 1Diabetes:DiabeticKetoacidosis(DKA):Symptoms:Extreme thirst,confusion, drymouth, noketones.Weight gain iscommon, andHHS is a riskdue to highblood glucosewithout ketones.Short-Acting Insulin(e.g., Regularinsulin): Takes 30-60 minutes to workand is used formeal coverage andDKA emergencies.Diet, exercise,and potentiallyoralmedicationsthat help lowerblood glucose.Common onsetis around age50 and tends todevelopgradually.Type 1Diabetes:Key LabValues andDiagnosticTestsSomepatients mayeventuallyneed insulin.Treatment:Insulin IV,fluids, andelectrolytereplacement.NormalFastingLevel: Lessthan 100mg/dL.Alpha cells: These cellsrelease glucagon whenblood glucose is low.Glucagon signals theliver to convert storedglycogen back intoglucose and release itinto the bloodstream,raising blood sugar.Should bebelow 140mg/dL after 2hours ofconsumingglucose.Cause: Extremelyhigh bloodglucose withdehydration,typically in Type2.Liver'sRole:Focus onlifestylechangesfirst:Foot Care: Inspectfeet daily for cuts,blisters, orinfections; wearproper footwear;avoid barefootwalking.NursingInterventions:Hypoglycemia (LowBlood Sugar): Whenblood glucose drops,the pancreasreleases glucagon tosignal the liver torelease glucose.Regularinsulininjections oran insulinpump.GlucoseToleranceTest:Complicationsof DiabetesDiabetesRiskFactorsEndocrine Function:The pancreas alsoregulates blood sugar(glucose) levels. Thishappens through tinyclusters called theIslets of Langerhans,which contain twocritical types of cells:BloodGlucoseBasics:What isDiabetesMellitus?Exercise: Canlower bloodglucose butrequires carefulplanning to avoidhypoglycemia.Insulin isessential.Treatmentinvolves:Normal Range: Bloodglucose should bebetween 70-110 mg/dL.The brain relies on asteady glucose supply,as it cannot storeglucose, making bloodglucose regulationcrucial for brain function.Cause: Highblood glucosewith ketoneproduction (dueto lack ofinsulin).OralHypoglycemicsfor Type 2:AutoimmuneCondition: Theimmune systemattacks and destroysbeta cells in thepancreas, whichstops the productionof insulin.Type 2: Obesity (high BMI),sedentary lifestyle, familyhistory, certain ethnicities(Hispanic, AfricanAmerican, NativeAmerican, Asian American,Pacific Islander), andhistory of gestationaldiabetes. Symptomsusually develop gradually.Higher risk forDiabetic Ketoacidosis(DKA), a life-threatening conditionwhere bloodbecomes acidic dueto a lack of insulin.BloodGlucose:Long-Acting Insulin(e.g., Lantus):Works slowlywithout peaks,providing steadyinsulin levels over12-24 hours.Symptoms:Rapid breathing(Kussmaul),fruity breath,nausea,dehydration.SomogyiEffect:UnderstandingDiabetes andthe Pancreas'RoleGradualonset, withsymptomsoften lessnoticeable.Diet controlthroughcarbohydratecounting.Diabetes Mellitus is acondition where thebody cannot regulateblood glucoseproperly, resulting inchronichyperglycemia (highblood sugar).UnderstandingDawnPhenomenonvs. SomogyiEffectHemoglobinA1C:Higher riskfor DKA dueto thecomplete lackof insulin.TherapeuticManagementand NursingInterventionsMonitoringblood sugarmultipletimes daily.DiabetesMellitus(DM): TypesandDifferences

Untitled Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


1
2
3
4
5
6
7
8
9
10
11
12
13
14
15
16
17
18
19
20
21
22
23
24
25
26
27
28
29
30
31
32
33
34
35
36
37
38
39
40
41
42
43
44
45
46
47
48
49
50
51
52
53
54
55
56
57
58
59
60
61
62
63
64
65
66
67
68
69
70
71
72
73
74
75
76
77
78
79
80
81
82
83
84
85
  1. Type 2 Diabetes:
  2. Blood sugar drops during the night (often due to excess insulin before bed), and the body compensates by raising blood glucose, resulting in high morning glucose. Managed by adjusting insulin dosage or adding a bedtime snack.
  3. Insulin stimulators (e.g., glipizide) help the pancreas release more insulin.
  4. Symptoms: Sudden weight loss, frequent urination (polyuria), extreme thirst (polydipsia), and increased hunger (polyphagia).
  5. Type 2 Diabetes:
  6. Common in people under 30 and often diagnosed abruptly.
  7. Biguanides (e.g., metformin) decrease glucose production in the liver.
  8. Intermediate-Acting Insulin (e.g., NPH): Takes 1-2 hours to start working; used to cover blood sugar between meals.
  9. Type 1: Family history, genetic predisposition, autoimmune conditions, and sometimes following viral infections. It’s more common in those under 30.
  10. Symptoms: Often similar to Type 1 (polyuria, polydipsia, polyphagia), but less noticeable.
  11. The liver stores glucose in the form of glycogen. When blood glucose levels drop, glucagon from the pancreas tells the liver to release stored glucose, raising blood glucose levels. When blood glucose is high, insulin encourages the liver to stor
  12. Urine Screening:
  13. Pancreas Functions:
  14. Hyperosmolar Hyperglycemic State (HHS):
  15. Dawn Phenomenon:
  16. Exocrine Function: This involves producing enzymes that help digest food.
  17. Medication Types and Insulin Administration
  18. Diet: Emphasize balanced meals, carb counting, and limiting sugar intake.
  19. Insulin Sensitizers (e.g., pioglitazone) make cells more responsive to insulin.
  20. Types of Diabetes:
  21. Beta cells: These cells release insulin and amylin when blood glucose is high. Insulin helps cells absorb glucose for energy, while amylin slows down the absorption of glucose and controls blood sugar spikes.
  22. Checks for glucose and ketones in the urine, as elevated levels may indicate diabetes or uncontrolled blood glucose.
  23. Sick Day Rules: Maintain blood sugar checks, even if not eating much, as stress and illness can increase blood sugar.
  24. Treatment: Insulin IV, fluids, and monitoring electrolytes.
  25. Higher risk for Hyperosmolar Hyperglycemic State (HHS), where blood sugar rises dangerously high without the presence of ketones (no acidosis).
  26. Hyperglycemia (High Blood Sugar): When blood glucose rises, the pancreas releases insulin to help cells absorb glucose and lower blood sugar levels.
  27. Type 2 Diabetes:
  28. Type 1 Diabetes:
  29. Reflects average blood glucose levels over the past 3 months. A normal A1C is between 4-6%.
  30. Insulin Resistance: The body produces insulin but doesn't use it effectively, often due to obesity, inactivity, and genetic factors.
  31. Blood Glucose Monitoring: Educate on frequency and techniques.
  32. Rapid-Acting Insulin (e.g., Humalog): Works within 15 minutes and is given before or with meals.
  33. Blood sugar rises in the early morning due to hormone release. Managed by adjusting insulin timing.
  34. Types of Insulin:
  35. Signs and Symptoms
  36. Quick onset of symptoms, including weight loss, increased appetite, and the "3 Ps" (polyuria, polydipsia, polyphagia).
  37. Type 1 Diabetes:
  38. Diabetic Ketoacidosis (DKA):
  39. Symptoms: Extreme thirst, confusion, dry mouth, no ketones.
  40. Weight gain is common, and HHS is a risk due to high blood glucose without ketones.
  41. Short-Acting Insulin (e.g., Regular insulin): Takes 30-60 minutes to work and is used for meal coverage and DKA emergencies.
  42. Diet, exercise, and potentially oral medications that help lower blood glucose.
  43. Common onset is around age 50 and tends to develop gradually.
  44. Type 1 Diabetes:
  45. Key Lab Values and Diagnostic Tests
  46. Some patients may eventually need insulin.
  47. Treatment: Insulin IV, fluids, and electrolyte replacement.
  48. Normal Fasting Level: Less than 100 mg/dL.
  49. Alpha cells: These cells release glucagon when blood glucose is low. Glucagon signals the liver to convert stored glycogen back into glucose and release it into the bloodstream, raising blood sugar.
  50. Should be below 140 mg/dL after 2 hours of consuming glucose.
  51. Cause: Extremely high blood glucose with dehydration, typically in Type 2.
  52. Liver's Role:
  53. Focus on lifestyle changes first:
  54. Foot Care: Inspect feet daily for cuts, blisters, or infections; wear proper footwear; avoid barefoot walking.
  55. Nursing Interventions:
  56. Hypoglycemia (Low Blood Sugar): When blood glucose drops, the pancreas releases glucagon to signal the liver to release glucose.
  57. Regular insulin injections or an insulin pump.
  58. Glucose Tolerance Test:
  59. Complications of Diabetes
  60. Diabetes Risk Factors
  61. Endocrine Function: The pancreas also regulates blood sugar (glucose) levels. This happens through tiny clusters called the Islets of Langerhans, which contain two critical types of cells:
  62. Blood Glucose Basics:
  63. What is Diabetes Mellitus?
  64. Exercise: Can lower blood glucose but requires careful planning to avoid hypoglycemia.
  65. Insulin is essential. Treatment involves:
  66. Normal Range: Blood glucose should be between 70-110 mg/dL. The brain relies on a steady glucose supply, as it cannot store glucose, making blood glucose regulation crucial for brain function.
  67. Cause: High blood glucose with ketone production (due to lack of insulin).
  68. Oral Hypoglycemics for Type 2:
  69. Autoimmune Condition: The immune system attacks and destroys beta cells in the pancreas, which stops the production of insulin.
  70. Type 2: Obesity (high BMI), sedentary lifestyle, family history, certain ethnicities (Hispanic, African American, Native American, Asian American, Pacific Islander), and history of gestational diabetes. Symptoms usually develop gradually.
  71. Higher risk for Diabetic Ketoacidosis (DKA), a life-threatening condition where blood becomes acidic due to a lack of insulin.
  72. Blood Glucose:
  73. Long-Acting Insulin (e.g., Lantus): Works slowly without peaks, providing steady insulin levels over 12-24 hours.
  74. Symptoms: Rapid breathing (Kussmaul), fruity breath, nausea, dehydration.
  75. Somogyi Effect:
  76. Understanding Diabetes and the Pancreas' Role
  77. Gradual onset, with symptoms often less noticeable.
  78. Diet control through carbohydrate counting.
  79. Diabetes Mellitus is a condition where the body cannot regulate blood glucose properly, resulting in chronic hyperglycemia (high blood sugar).
  80. Understanding Dawn Phenomenon vs. Somogyi Effect
  81. Hemoglobin A1C:
  82. Higher risk for DKA due to the complete lack of insulin.
  83. Therapeutic Management and Nursing Interventions
  84. Monitoring blood sugar multiple times daily.
  85. Diabetes Mellitus (DM): Types and Differences