Type 2Diabetes:TherapeuticManagementand NursingInterventionsRapid-ActingInsulin (e.g.,Humalog): Workswithin 15 minutesand is givenbefore or withmeals.Checks for glucoseand ketones in theurine, as elevatedlevels may indicatediabetes oruncontrolled bloodglucose.GlucoseToleranceTest:Reflects averageblood glucoselevels over thepast 3 months. Anormal A1C isbetween 4-6%.Higher risk forDiabetic Ketoacidosis(DKA), a life-threatening conditionwhere bloodbecomes acidic dueto a lack of insulin.Symptoms: Suddenweight loss, frequenturination (polyuria),extreme thirst(polydipsia), andincreased hunger(polyphagia).Insulin Resistance:The body producesinsulin but doesn'tuse it effectively,often due to obesity,inactivity, andgenetic factors.Weight gain iscommon, andHHS is a riskdue to highblood glucosewithout ketones.Long-Acting Insulin(e.g., Lantus):Works slowlywithout peaks,providing steadyinsulin levels over12-24 hours.Higher risk forHyperosmolarHyperglycemic State(HHS), where bloodsugar risesdangerously highwithout the presence ofketones (no acidosis).Complicationsof DiabetesBloodGlucose:SomogyiEffect:Normal Range: Bloodglucose should bebetween 70-110 mg/dL.The brain relies on asteady glucose supply,as it cannot storeglucose, making bloodglucose regulationcrucial for brain function.Hyperglycemia (HighBlood Sugar): Whenblood glucose rises,the pancreasreleases insulin tohelp cells absorbglucose and lowerblood sugar levels.Hypoglycemia (LowBlood Sugar): Whenblood glucose drops,the pancreasreleases glucagon tosignal the liver torelease glucose.Key LabValues andDiagnosticTestsOralHypoglycemicsfor Type 2:Gradualonset, withsymptomsoften lessnoticeable.Alpha cells: These cellsrelease glucagon whenblood glucose is low.Glucagon signals theliver to convert storedglycogen back intoglucose and release itinto the bloodstream,raising blood sugar.Type 2Diabetes:Liver'sRole:Type 2: Obesity (high BMI),sedentary lifestyle, familyhistory, certain ethnicities(Hispanic, AfricanAmerican, NativeAmerican, Asian American,Pacific Islander), andhistory of gestationaldiabetes. Symptomsusually develop gradually.Should bebelow 140mg/dL after 2hours ofconsumingglucose.Common inpeople under30 and oftendiagnosedabruptly.UrineScreening:Biguanides(e.g., metformin)decreaseglucoseproduction in theliver.Treatment:Insulin IV,fluids, andelectrolytereplacement.The liver stores glucose inthe form of glycogen. Whenblood glucose levels drop,glucagon from thepancreas tells the liver torelease stored glucose,raising blood glucoselevels. When blood glucoseis high, insulin encouragesthe liver to storSigns andSymptomsQuick onset ofsymptoms, includingweight loss,increased appetite,and the "3 Ps"(polyuria, polydipsia,polyphagia).Type 1Diabetes:TypesofInsulin:Treatment:Insulin IV,fluids, andmonitoringelectrolytes.BloodGlucoseBasics:Blood GlucoseMonitoring:Educate onfrequency andtechniques.Insulin isessential.Treatmentinvolves:MedicationTypes andInsulinAdministrationDiet: Emphasizebalanced meals,carb counting,and limitingsugar intake.NursingInterventions:Regularinsulininjections oran insulinpump.Symptoms:Rapid breathing(Kussmaul),fruity breath,nausea,dehydration.Foot Care: Inspectfeet daily for cuts,blisters, orinfections; wearproper footwear;avoid barefootwalking.Symptoms:Often similar toType 1 (polyuria,polydipsia,polyphagia), butless noticeable.NormalFastingLevel: Lessthan 100mg/dL.AutoimmuneCondition: Theimmune systemattacks and destroysbeta cells in thepancreas, whichstops the productionof insulin.Type 1Diabetes:Beta cells: These cellsrelease insulin and amylinwhen blood glucose ishigh. Insulin helps cellsabsorb glucose for energy,while amylin slows downthe absorption of glucoseand controls blood sugarspikes.Blood sugar drops duringthe night (often due toexcess insulin before bed),and the body compensatesby raising blood glucose,resulting in high morningglucose. Managed byadjusting insulin dosage oradding a bedtime snack.Short-Acting Insulin(e.g., Regularinsulin): Takes 30-60 minutes to workand is used formeal coverage andDKA emergencies.UnderstandingDiabetes andthe Pancreas'RoleWhat isDiabetesMellitus?Types ofDiabetes:PancreasFunctions:DiabetesMellitus(DM): TypesandDifferencesExocrineFunction: Thisinvolvesproducingenzymes thathelp digest food.Common onsetis around age50 and tends todevelopgradually.Somepatients mayeventuallyneed insulin.Blood sugar risesin the earlymorning due tohormone release.Managed byadjusting insulintiming.DawnPhenomenon:Higher riskfor DKA dueto thecomplete lackof insulin.Intermediate-Acting Insulin(e.g., NPH): Takes1-2 hours to startworking; used tocover blood sugarbetween meals.Cause: Extremelyhigh bloodglucose withdehydration,typically in Type2.Sick Day Rules:Maintain bloodsugar checks, evenif not eating much,as stress andillness can increaseblood sugar.Insulinstimulators (e.g.,glipizide) helpthe pancreasrelease moreinsulin.Type 2Diabetes:DiabetesRiskFactorsMonitoringblood sugarmultipletimes daily.DiabeticKetoacidosis(DKA):HyperosmolarHyperglycemicState (HHS):Exercise: Canlower bloodglucose butrequires carefulplanning to avoidhypoglycemia.Diet controlthroughcarbohydratecounting.Diet, exercise,and potentiallyoralmedicationsthat help lowerblood glucose.Cause: Highblood glucosewith ketoneproduction (dueto lack ofinsulin).Type 1: Family history,genetic predisposition,autoimmuneconditions, andsometimes followingviral infections. It’smore common in thoseunder 30.Symptoms:Extreme thirst,confusion, drymouth, noketones.HemoglobinA1C:Focus onlifestylechangesfirst:Diabetes Mellitus is acondition where thebody cannot regulateblood glucoseproperly, resulting inchronichyperglycemia (highblood sugar).UnderstandingDawnPhenomenonvs. SomogyiEffectInsulinSensitizers (e.g.,pioglitazone)make cells moreresponsive toinsulin.Type 1Diabetes:Endocrine Function:The pancreas alsoregulates blood sugar(glucose) levels. Thishappens through tinyclusters called theIslets of Langerhans,which contain twocritical types of cells:Type 2Diabetes:TherapeuticManagementand NursingInterventionsRapid-ActingInsulin (e.g.,Humalog): Workswithin 15 minutesand is givenbefore or withmeals.Checks for glucoseand ketones in theurine, as elevatedlevels may indicatediabetes oruncontrolled bloodglucose.GlucoseToleranceTest:Reflects averageblood glucoselevels over thepast 3 months. Anormal A1C isbetween 4-6%.Higher risk forDiabetic Ketoacidosis(DKA), a life-threatening conditionwhere bloodbecomes acidic dueto a lack of insulin.Symptoms: Suddenweight loss, frequenturination (polyuria),extreme thirst(polydipsia), andincreased hunger(polyphagia).Insulin Resistance:The body producesinsulin but doesn'tuse it effectively,often due to obesity,inactivity, andgenetic factors.Weight gain iscommon, andHHS is a riskdue to highblood glucosewithout ketones.Long-Acting Insulin(e.g., Lantus):Works slowlywithout peaks,providing steadyinsulin levels over12-24 hours.Higher risk forHyperosmolarHyperglycemic State(HHS), where bloodsugar risesdangerously highwithout the presence ofketones (no acidosis).Complicationsof DiabetesBloodGlucose:SomogyiEffect:Normal Range: Bloodglucose should bebetween 70-110 mg/dL.The brain relies on asteady glucose supply,as it cannot storeglucose, making bloodglucose regulationcrucial for brain function.Hyperglycemia (HighBlood Sugar): Whenblood glucose rises,the pancreasreleases insulin tohelp cells absorbglucose and lowerblood sugar levels.Hypoglycemia (LowBlood Sugar): Whenblood glucose drops,the pancreasreleases glucagon tosignal the liver torelease glucose.Key LabValues andDiagnosticTestsOralHypoglycemicsfor Type 2:Gradualonset, withsymptomsoften lessnoticeable.Alpha cells: These cellsrelease glucagon whenblood glucose is low.Glucagon signals theliver to convert storedglycogen back intoglucose and release itinto the bloodstream,raising blood sugar.Type 2Diabetes:Liver'sRole:Type 2: Obesity (high BMI),sedentary lifestyle, familyhistory, certain ethnicities(Hispanic, AfricanAmerican, NativeAmerican, Asian American,Pacific Islander), andhistory of gestationaldiabetes. Symptomsusually develop gradually.Should bebelow 140mg/dL after 2hours ofconsumingglucose.Common inpeople under30 and oftendiagnosedabruptly.UrineScreening:Biguanides(e.g., metformin)decreaseglucoseproduction in theliver.Treatment:Insulin IV,fluids, andelectrolytereplacement.The liver stores glucose inthe form of glycogen. Whenblood glucose levels drop,glucagon from thepancreas tells the liver torelease stored glucose,raising blood glucoselevels. When blood glucoseis high, insulin encouragesthe liver to storSigns andSymptomsQuick onset ofsymptoms, includingweight loss,increased appetite,and the "3 Ps"(polyuria, polydipsia,polyphagia).Type 1Diabetes:TypesofInsulin:Treatment:Insulin IV,fluids, andmonitoringelectrolytes.BloodGlucoseBasics:Blood GlucoseMonitoring:Educate onfrequency andtechniques.Insulin isessential.Treatmentinvolves:MedicationTypes andInsulinAdministrationDiet: Emphasizebalanced meals,carb counting,and limitingsugar intake.NursingInterventions:Regularinsulininjections oran insulinpump.Symptoms:Rapid breathing(Kussmaul),fruity breath,nausea,dehydration.Foot Care: Inspectfeet daily for cuts,blisters, orinfections; wearproper footwear;avoid barefootwalking.Symptoms:Often similar toType 1 (polyuria,polydipsia,polyphagia), butless noticeable.NormalFastingLevel: Lessthan 100mg/dL.AutoimmuneCondition: Theimmune systemattacks and destroysbeta cells in thepancreas, whichstops the productionof insulin.Type 1Diabetes:Beta cells: These cellsrelease insulin and amylinwhen blood glucose ishigh. Insulin helps cellsabsorb glucose for energy,while amylin slows downthe absorption of glucoseand controls blood sugarspikes.Blood sugar drops duringthe night (often due toexcess insulin before bed),and the body compensatesby raising blood glucose,resulting in high morningglucose. Managed byadjusting insulin dosage oradding a bedtime snack.Short-Acting Insulin(e.g., Regularinsulin): Takes 30-60 minutes to workand is used formeal coverage andDKA emergencies.UnderstandingDiabetes andthe Pancreas'RoleWhat isDiabetesMellitus?Types ofDiabetes:PancreasFunctions:DiabetesMellitus(DM): TypesandDifferencesExocrineFunction: Thisinvolvesproducingenzymes thathelp digest food.Common onsetis around age50 and tends todevelopgradually.Somepatients mayeventuallyneed insulin.Blood sugar risesin the earlymorning due tohormone release.Managed byadjusting insulintiming.DawnPhenomenon:Higher riskfor DKA dueto thecomplete lackof insulin.Intermediate-Acting Insulin(e.g., NPH): Takes1-2 hours to startworking; used tocover blood sugarbetween meals.Cause: Extremelyhigh bloodglucose withdehydration,typically in Type2.Sick Day Rules:Maintain bloodsugar checks, evenif not eating much,as stress andillness can increaseblood sugar.Insulinstimulators (e.g.,glipizide) helpthe pancreasrelease moreinsulin.Type 2Diabetes:DiabetesRiskFactorsMonitoringblood sugarmultipletimes daily.DiabeticKetoacidosis(DKA):HyperosmolarHyperglycemicState (HHS):Exercise: Canlower bloodglucose butrequires carefulplanning to avoidhypoglycemia.Diet controlthroughcarbohydratecounting.Diet, exercise,and potentiallyoralmedicationsthat help lowerblood glucose.Cause: Highblood glucosewith ketoneproduction (dueto lack ofinsulin).Type 1: Family history,genetic predisposition,autoimmuneconditions, andsometimes followingviral infections. It’smore common in thoseunder 30.Symptoms:Extreme thirst,confusion, drymouth, noketones.HemoglobinA1C:Focus onlifestylechangesfirst:Diabetes Mellitus is acondition where thebody cannot regulateblood glucoseproperly, resulting inchronichyperglycemia (highblood sugar).UnderstandingDawnPhenomenonvs. SomogyiEffectInsulinSensitizers (e.g.,pioglitazone)make cells moreresponsive toinsulin.Type 1Diabetes:Endocrine Function:The pancreas alsoregulates blood sugar(glucose) levels. Thishappens through tinyclusters called theIslets of Langerhans,which contain twocritical types of cells:

Untitled Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Type 2 Diabetes:
  2. Therapeutic Management and Nursing Interventions
  3. Rapid-Acting Insulin (e.g., Humalog): Works within 15 minutes and is given before or with meals.
  4. Checks for glucose and ketones in the urine, as elevated levels may indicate diabetes or uncontrolled blood glucose.
  5. Glucose Tolerance Test:
  6. Reflects average blood glucose levels over the past 3 months. A normal A1C is between 4-6%.
  7. Higher risk for Diabetic Ketoacidosis (DKA), a life-threatening condition where blood becomes acidic due to a lack of insulin.
  8. Symptoms: Sudden weight loss, frequent urination (polyuria), extreme thirst (polydipsia), and increased hunger (polyphagia).
  9. Insulin Resistance: The body produces insulin but doesn't use it effectively, often due to obesity, inactivity, and genetic factors.
  10. Weight gain is common, and HHS is a risk due to high blood glucose without ketones.
  11. Long-Acting Insulin (e.g., Lantus): Works slowly without peaks, providing steady insulin levels over 12-24 hours.
  12. Higher risk for Hyperosmolar Hyperglycemic State (HHS), where blood sugar rises dangerously high without the presence of ketones (no acidosis).
  13. Complications of Diabetes
  14. Blood Glucose:
  15. Somogyi Effect:
  16. Normal Range: Blood glucose should be between 70-110 mg/dL. The brain relies on a steady glucose supply, as it cannot store glucose, making blood glucose regulation crucial for brain function.
  17. Hyperglycemia (High Blood Sugar): When blood glucose rises, the pancreas releases insulin to help cells absorb glucose and lower blood sugar levels.
  18. Hypoglycemia (Low Blood Sugar): When blood glucose drops, the pancreas releases glucagon to signal the liver to release glucose.
  19. Key Lab Values and Diagnostic Tests
  20. Oral Hypoglycemics for Type 2:
  21. Gradual onset, with symptoms often less noticeable.
  22. Alpha cells: These cells release glucagon when blood glucose is low. Glucagon signals the liver to convert stored glycogen back into glucose and release it into the bloodstream, raising blood sugar.
  23. Type 2 Diabetes:
  24. Liver's Role:
  25. Type 2: Obesity (high BMI), sedentary lifestyle, family history, certain ethnicities (Hispanic, African American, Native American, Asian American, Pacific Islander), and history of gestational diabetes. Symptoms usually develop gradually.
  26. Should be below 140 mg/dL after 2 hours of consuming glucose.
  27. Common in people under 30 and often diagnosed abruptly.
  28. Urine Screening:
  29. Biguanides (e.g., metformin) decrease glucose production in the liver.
  30. Treatment: Insulin IV, fluids, and electrolyte replacement.
  31. The liver stores glucose in the form of glycogen. When blood glucose levels drop, glucagon from the pancreas tells the liver to release stored glucose, raising blood glucose levels. When blood glucose is high, insulin encourages the liver to stor
  32. Signs and Symptoms
  33. Quick onset of symptoms, including weight loss, increased appetite, and the "3 Ps" (polyuria, polydipsia, polyphagia).
  34. Type 1 Diabetes:
  35. Types of Insulin:
  36. Treatment: Insulin IV, fluids, and monitoring electrolytes.
  37. Blood Glucose Basics:
  38. Blood Glucose Monitoring: Educate on frequency and techniques.
  39. Insulin is essential. Treatment involves:
  40. Medication Types and Insulin Administration
  41. Diet: Emphasize balanced meals, carb counting, and limiting sugar intake.
  42. Nursing Interventions:
  43. Regular insulin injections or an insulin pump.
  44. Symptoms: Rapid breathing (Kussmaul), fruity breath, nausea, dehydration.
  45. Foot Care: Inspect feet daily for cuts, blisters, or infections; wear proper footwear; avoid barefoot walking.
  46. Symptoms: Often similar to Type 1 (polyuria, polydipsia, polyphagia), but less noticeable.
  47. Normal Fasting Level: Less than 100 mg/dL.
  48. Autoimmune Condition: The immune system attacks and destroys beta cells in the pancreas, which stops the production of insulin.
  49. Type 1 Diabetes:
  50. Beta cells: These cells release insulin and amylin when blood glucose is high. Insulin helps cells absorb glucose for energy, while amylin slows down the absorption of glucose and controls blood sugar spikes.
  51. Blood sugar drops during the night (often due to excess insulin before bed), and the body compensates by raising blood glucose, resulting in high morning glucose. Managed by adjusting insulin dosage or adding a bedtime snack.
  52. Short-Acting Insulin (e.g., Regular insulin): Takes 30-60 minutes to work and is used for meal coverage and DKA emergencies.
  53. Understanding Diabetes and the Pancreas' Role
  54. What is Diabetes Mellitus?
  55. Types of Diabetes:
  56. Pancreas Functions:
  57. Diabetes Mellitus (DM): Types and Differences
  58. Exocrine Function: This involves producing enzymes that help digest food.
  59. Common onset is around age 50 and tends to develop gradually.
  60. Some patients may eventually need insulin.
  61. Blood sugar rises in the early morning due to hormone release. Managed by adjusting insulin timing.
  62. Dawn Phenomenon:
  63. Higher risk for DKA due to the complete lack of insulin.
  64. Intermediate-Acting Insulin (e.g., NPH): Takes 1-2 hours to start working; used to cover blood sugar between meals.
  65. Cause: Extremely high blood glucose with dehydration, typically in Type 2.
  66. Sick Day Rules: Maintain blood sugar checks, even if not eating much, as stress and illness can increase blood sugar.
  67. Insulin stimulators (e.g., glipizide) help the pancreas release more insulin.
  68. Type 2 Diabetes:
  69. Diabetes Risk Factors
  70. Monitoring blood sugar multiple times daily.
  71. Diabetic Ketoacidosis (DKA):
  72. Hyperosmolar Hyperglycemic State (HHS):
  73. Exercise: Can lower blood glucose but requires careful planning to avoid hypoglycemia.
  74. Diet control through carbohydrate counting.
  75. Diet, exercise, and potentially oral medications that help lower blood glucose.
  76. Cause: High blood glucose with ketone production (due to lack of insulin).
  77. Type 1: Family history, genetic predisposition, autoimmune conditions, and sometimes following viral infections. It’s more common in those under 30.
  78. Symptoms: Extreme thirst, confusion, dry mouth, no ketones.
  79. Hemoglobin A1C:
  80. Focus on lifestyle changes first:
  81. Diabetes Mellitus is a condition where the body cannot regulate blood glucose properly, resulting in chronic hyperglycemia (high blood sugar).
  82. Understanding Dawn Phenomenon vs. Somogyi Effect
  83. Insulin Sensitizers (e.g., pioglitazone) make cells more responsive to insulin.
  84. Type 1 Diabetes:
  85. Endocrine Function: The pancreas also regulates blood sugar (glucose) levels. This happens through tiny clusters called the Islets of Langerhans, which contain two critical types of cells: