InsulinSensitizers (e.g.,pioglitazone)make cells moreresponsive toinsulin.Diet, exercise,and potentiallyoralmedicationsthat help lowerblood glucose.TypesofInsulin:GlucoseToleranceTest:Long-Acting Insulin(e.g., Lantus):Works slowlywithout peaks,providing steadyinsulin levels over12-24 hours.Symptoms: Suddenweight loss, frequenturination (polyuria),extreme thirst(polydipsia), andincreased hunger(polyphagia).Symptoms:Extreme thirst,confusion, drymouth, noketones.NormalFastingLevel: Lessthan 100mg/dL.Checks for glucoseand ketones in theurine, as elevatedlevels may indicatediabetes oruncontrolled bloodglucose.Symptoms:Rapid breathing(Kussmaul),fruity breath,nausea,dehydration.UnderstandingDawnPhenomenonvs. SomogyiEffectSymptoms:Often similar toType 1 (polyuria,polydipsia,polyphagia), butless noticeable.Foot Care: Inspectfeet daily for cuts,blisters, orinfections; wearproper footwear;avoid barefootwalking.PancreasFunctions:Insulinstimulators (e.g.,glipizide) helpthe pancreasrelease moreinsulin.Monitoringblood sugarmultipletimes daily.Blood sugar drops duringthe night (often due toexcess insulin before bed),and the body compensatesby raising blood glucose,resulting in high morningglucose. Managed byadjusting insulin dosage oradding a bedtime snack.Treatment:Insulin IV,fluids, andmonitoringelectrolytes.Focus onlifestylechangesfirst:Rapid-ActingInsulin (e.g.,Humalog): Workswithin 15 minutesand is givenbefore or withmeals.Common inpeople under30 and oftendiagnosedabruptly.DiabetesRiskFactorsInsulin isessential.Treatmentinvolves:Complicationsof DiabetesExercise: Canlower bloodglucose butrequires carefulplanning to avoidhypoglycemia.Type 2Diabetes:DiabeticKetoacidosis(DKA):Higher risk forDiabetic Ketoacidosis(DKA), a life-threatening conditionwhere bloodbecomes acidic dueto a lack of insulin.Weight gain iscommon, andHHS is a riskdue to highblood glucosewithout ketones.Blood sugar risesin the earlymorning due tohormone release.Managed byadjusting insulintiming.Blood GlucoseMonitoring:Educate onfrequency andtechniques.HemoglobinA1C:BloodGlucose:Regularinsulininjections oran insulinpump.MedicationTypes andInsulinAdministrationDiet: Emphasizebalanced meals,carb counting,and limitingsugar intake.ExocrineFunction: Thisinvolvesproducingenzymes thathelp digest food.Type 1Diabetes:SomogyiEffect:Hyperglycemia (HighBlood Sugar): Whenblood glucose rises,the pancreasreleases insulin tohelp cells absorbglucose and lowerblood sugar levels.Gradualonset, withsymptomsoften lessnoticeable.OralHypoglycemicsfor Type 2:Type 1: Family history,genetic predisposition,autoimmuneconditions, andsometimes followingviral infections. It’smore common in thoseunder 30.Quick onset ofsymptoms, includingweight loss,increased appetite,and the "3 Ps"(polyuria, polydipsia,polyphagia).Short-Acting Insulin(e.g., Regularinsulin): Takes 30-60 minutes to workand is used formeal coverage andDKA emergencies.Treatment:Insulin IV,fluids, andelectrolytereplacement.Diabetes Mellitus is acondition where thebody cannot regulateblood glucoseproperly, resulting inchronichyperglycemia (highblood sugar).TherapeuticManagementand NursingInterventionsHigher risk forHyperosmolarHyperglycemic State(HHS), where bloodsugar risesdangerously highwithout the presence ofketones (no acidosis).Alpha cells: These cellsrelease glucagon whenblood glucose is low.Glucagon signals theliver to convert storedglycogen back intoglucose and release itinto the bloodstream,raising blood sugar.Types ofDiabetes:Diet controlthroughcarbohydratecounting.Normal Range: Bloodglucose should bebetween 70-110 mg/dL.The brain relies on asteady glucose supply,as it cannot storeglucose, making bloodglucose regulationcrucial for brain function.Hypoglycemia (LowBlood Sugar): Whenblood glucose drops,the pancreasreleases glucagon tosignal the liver torelease glucose.DawnPhenomenon:NursingInterventions:Signs andSymptomsIntermediate-Acting Insulin(e.g., NPH): Takes1-2 hours to startworking; used tocover blood sugarbetween meals.Cause: Extremelyhigh bloodglucose withdehydration,typically in Type2.Key LabValues andDiagnosticTestsEndocrine Function:The pancreas alsoregulates blood sugar(glucose) levels. Thishappens through tinyclusters called theIslets of Langerhans,which contain twocritical types of cells:What isDiabetesMellitus?Insulin Resistance:The body producesinsulin but doesn'tuse it effectively,often due to obesity,inactivity, andgenetic factors.DiabetesMellitus(DM): TypesandDifferencesType 2Diabetes:Somepatients mayeventuallyneed insulin.Type 1Diabetes:HyperosmolarHyperglycemicState (HHS):The liver stores glucose inthe form of glycogen. Whenblood glucose levels drop,glucagon from thepancreas tells the liver torelease stored glucose,raising blood glucoselevels. When blood glucoseis high, insulin encouragesthe liver to storLiver'sRole:Beta cells: These cellsrelease insulin and amylinwhen blood glucose ishigh. Insulin helps cellsabsorb glucose for energy,while amylin slows downthe absorption of glucoseand controls blood sugarspikes.Type 2: Obesity (high BMI),sedentary lifestyle, familyhistory, certain ethnicities(Hispanic, AfricanAmerican, NativeAmerican, Asian American,Pacific Islander), andhistory of gestationaldiabetes. Symptomsusually develop gradually.Cause: Highblood glucosewith ketoneproduction (dueto lack ofinsulin).UnderstandingDiabetes andthe Pancreas'RoleBiguanides(e.g., metformin)decreaseglucoseproduction in theliver.Type 2Diabetes:BloodGlucoseBasics:Should bebelow 140mg/dL after 2hours ofconsumingglucose.Common onsetis around age50 and tends todevelopgradually.Higher riskfor DKA dueto thecomplete lackof insulin.Type 1Diabetes:AutoimmuneCondition: Theimmune systemattacks and destroysbeta cells in thepancreas, whichstops the productionof insulin.UrineScreening:Sick Day Rules:Maintain bloodsugar checks, evenif not eating much,as stress andillness can increaseblood sugar.Reflects averageblood glucoselevels over thepast 3 months. Anormal A1C isbetween 4-6%.InsulinSensitizers (e.g.,pioglitazone)make cells moreresponsive toinsulin.Diet, exercise,and potentiallyoralmedicationsthat help lowerblood glucose.TypesofInsulin:GlucoseToleranceTest:Long-Acting Insulin(e.g., Lantus):Works slowlywithout peaks,providing steadyinsulin levels over12-24 hours.Symptoms: Suddenweight loss, frequenturination (polyuria),extreme thirst(polydipsia), andincreased hunger(polyphagia).Symptoms:Extreme thirst,confusion, drymouth, noketones.NormalFastingLevel: Lessthan 100mg/dL.Checks for glucoseand ketones in theurine, as elevatedlevels may indicatediabetes oruncontrolled bloodglucose.Symptoms:Rapid breathing(Kussmaul),fruity breath,nausea,dehydration.UnderstandingDawnPhenomenonvs. SomogyiEffectSymptoms:Often similar toType 1 (polyuria,polydipsia,polyphagia), butless noticeable.Foot Care: Inspectfeet daily for cuts,blisters, orinfections; wearproper footwear;avoid barefootwalking.PancreasFunctions:Insulinstimulators (e.g.,glipizide) helpthe pancreasrelease moreinsulin.Monitoringblood sugarmultipletimes daily.Blood sugar drops duringthe night (often due toexcess insulin before bed),and the body compensatesby raising blood glucose,resulting in high morningglucose. Managed byadjusting insulin dosage oradding a bedtime snack.Treatment:Insulin IV,fluids, andmonitoringelectrolytes.Focus onlifestylechangesfirst:Rapid-ActingInsulin (e.g.,Humalog): Workswithin 15 minutesand is givenbefore or withmeals.Common inpeople under30 and oftendiagnosedabruptly.DiabetesRiskFactorsInsulin isessential.Treatmentinvolves:Complicationsof DiabetesExercise: Canlower bloodglucose butrequires carefulplanning to avoidhypoglycemia.Type 2Diabetes:DiabeticKetoacidosis(DKA):Higher risk forDiabetic Ketoacidosis(DKA), a life-threatening conditionwhere bloodbecomes acidic dueto a lack of insulin.Weight gain iscommon, andHHS is a riskdue to highblood glucosewithout ketones.Blood sugar risesin the earlymorning due tohormone release.Managed byadjusting insulintiming.Blood GlucoseMonitoring:Educate onfrequency andtechniques.HemoglobinA1C:BloodGlucose:Regularinsulininjections oran insulinpump.MedicationTypes andInsulinAdministrationDiet: Emphasizebalanced meals,carb counting,and limitingsugar intake.ExocrineFunction: Thisinvolvesproducingenzymes thathelp digest food.Type 1Diabetes:SomogyiEffect:Hyperglycemia (HighBlood Sugar): Whenblood glucose rises,the pancreasreleases insulin tohelp cells absorbglucose and lowerblood sugar levels.Gradualonset, withsymptomsoften lessnoticeable.OralHypoglycemicsfor Type 2:Type 1: Family history,genetic predisposition,autoimmuneconditions, andsometimes followingviral infections. It’smore common in thoseunder 30.Quick onset ofsymptoms, includingweight loss,increased appetite,and the "3 Ps"(polyuria, polydipsia,polyphagia).Short-Acting Insulin(e.g., Regularinsulin): Takes 30-60 minutes to workand is used formeal coverage andDKA emergencies.Treatment:Insulin IV,fluids, andelectrolytereplacement.Diabetes Mellitus is acondition where thebody cannot regulateblood glucoseproperly, resulting inchronichyperglycemia (highblood sugar).TherapeuticManagementand NursingInterventionsHigher risk forHyperosmolarHyperglycemic State(HHS), where bloodsugar risesdangerously highwithout the presence ofketones (no acidosis).Alpha cells: These cellsrelease glucagon whenblood glucose is low.Glucagon signals theliver to convert storedglycogen back intoglucose and release itinto the bloodstream,raising blood sugar.Types ofDiabetes:Diet controlthroughcarbohydratecounting.Normal Range: Bloodglucose should bebetween 70-110 mg/dL.The brain relies on asteady glucose supply,as it cannot storeglucose, making bloodglucose regulationcrucial for brain function.Hypoglycemia (LowBlood Sugar): Whenblood glucose drops,the pancreasreleases glucagon tosignal the liver torelease glucose.DawnPhenomenon:NursingInterventions:Signs andSymptomsIntermediate-Acting Insulin(e.g., NPH): Takes1-2 hours to startworking; used tocover blood sugarbetween meals.Cause: Extremelyhigh bloodglucose withdehydration,typically in Type2.Key LabValues andDiagnosticTestsEndocrine Function:The pancreas alsoregulates blood sugar(glucose) levels. Thishappens through tinyclusters called theIslets of Langerhans,which contain twocritical types of cells:What isDiabetesMellitus?Insulin Resistance:The body producesinsulin but doesn'tuse it effectively,often due to obesity,inactivity, andgenetic factors.DiabetesMellitus(DM): TypesandDifferencesType 2Diabetes:Somepatients mayeventuallyneed insulin.Type 1Diabetes:HyperosmolarHyperglycemicState (HHS):The liver stores glucose inthe form of glycogen. Whenblood glucose levels drop,glucagon from thepancreas tells the liver torelease stored glucose,raising blood glucoselevels. When blood glucoseis high, insulin encouragesthe liver to storLiver'sRole:Beta cells: These cellsrelease insulin and amylinwhen blood glucose ishigh. Insulin helps cellsabsorb glucose for energy,while amylin slows downthe absorption of glucoseand controls blood sugarspikes.Type 2: Obesity (high BMI),sedentary lifestyle, familyhistory, certain ethnicities(Hispanic, AfricanAmerican, NativeAmerican, Asian American,Pacific Islander), andhistory of gestationaldiabetes. Symptomsusually develop gradually.Cause: Highblood glucosewith ketoneproduction (dueto lack ofinsulin).UnderstandingDiabetes andthe Pancreas'RoleBiguanides(e.g., metformin)decreaseglucoseproduction in theliver.Type 2Diabetes:BloodGlucoseBasics:Should bebelow 140mg/dL after 2hours ofconsumingglucose.Common onsetis around age50 and tends todevelopgradually.Higher riskfor DKA dueto thecomplete lackof insulin.Type 1Diabetes:AutoimmuneCondition: Theimmune systemattacks and destroysbeta cells in thepancreas, whichstops the productionof insulin.UrineScreening:Sick Day Rules:Maintain bloodsugar checks, evenif not eating much,as stress andillness can increaseblood sugar.Reflects averageblood glucoselevels over thepast 3 months. Anormal A1C isbetween 4-6%.

Untitled Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


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  1. Insulin Sensitizers (e.g., pioglitazone) make cells more responsive to insulin.
  2. Diet, exercise, and potentially oral medications that help lower blood glucose.
  3. Types of Insulin:
  4. Glucose Tolerance Test:
  5. Long-Acting Insulin (e.g., Lantus): Works slowly without peaks, providing steady insulin levels over 12-24 hours.
  6. Symptoms: Sudden weight loss, frequent urination (polyuria), extreme thirst (polydipsia), and increased hunger (polyphagia).
  7. Symptoms: Extreme thirst, confusion, dry mouth, no ketones.
  8. Normal Fasting Level: Less than 100 mg/dL.
  9. Checks for glucose and ketones in the urine, as elevated levels may indicate diabetes or uncontrolled blood glucose.
  10. Symptoms: Rapid breathing (Kussmaul), fruity breath, nausea, dehydration.
  11. Understanding Dawn Phenomenon vs. Somogyi Effect
  12. Symptoms: Often similar to Type 1 (polyuria, polydipsia, polyphagia), but less noticeable.
  13. Foot Care: Inspect feet daily for cuts, blisters, or infections; wear proper footwear; avoid barefoot walking.
  14. Pancreas Functions:
  15. Insulin stimulators (e.g., glipizide) help the pancreas release more insulin.
  16. Monitoring blood sugar multiple times daily.
  17. Blood sugar drops during the night (often due to excess insulin before bed), and the body compensates by raising blood glucose, resulting in high morning glucose. Managed by adjusting insulin dosage or adding a bedtime snack.
  18. Treatment: Insulin IV, fluids, and monitoring electrolytes.
  19. Focus on lifestyle changes first:
  20. Rapid-Acting Insulin (e.g., Humalog): Works within 15 minutes and is given before or with meals.
  21. Common in people under 30 and often diagnosed abruptly.
  22. Diabetes Risk Factors
  23. Insulin is essential. Treatment involves:
  24. Complications of Diabetes
  25. Exercise: Can lower blood glucose but requires careful planning to avoid hypoglycemia.
  26. Type 2 Diabetes:
  27. Diabetic Ketoacidosis (DKA):
  28. Higher risk for Diabetic Ketoacidosis (DKA), a life-threatening condition where blood becomes acidic due to a lack of insulin.
  29. Weight gain is common, and HHS is a risk due to high blood glucose without ketones.
  30. Blood sugar rises in the early morning due to hormone release. Managed by adjusting insulin timing.
  31. Blood Glucose Monitoring: Educate on frequency and techniques.
  32. Hemoglobin A1C:
  33. Blood Glucose:
  34. Regular insulin injections or an insulin pump.
  35. Medication Types and Insulin Administration
  36. Diet: Emphasize balanced meals, carb counting, and limiting sugar intake.
  37. Exocrine Function: This involves producing enzymes that help digest food.
  38. Type 1 Diabetes:
  39. Somogyi Effect:
  40. Hyperglycemia (High Blood Sugar): When blood glucose rises, the pancreas releases insulin to help cells absorb glucose and lower blood sugar levels.
  41. Gradual onset, with symptoms often less noticeable.
  42. Oral Hypoglycemics for Type 2:
  43. Type 1: Family history, genetic predisposition, autoimmune conditions, and sometimes following viral infections. It’s more common in those under 30.
  44. Quick onset of symptoms, including weight loss, increased appetite, and the "3 Ps" (polyuria, polydipsia, polyphagia).
  45. Short-Acting Insulin (e.g., Regular insulin): Takes 30-60 minutes to work and is used for meal coverage and DKA emergencies.
  46. Treatment: Insulin IV, fluids, and electrolyte replacement.
  47. Diabetes Mellitus is a condition where the body cannot regulate blood glucose properly, resulting in chronic hyperglycemia (high blood sugar).
  48. Therapeutic Management and Nursing Interventions
  49. Higher risk for Hyperosmolar Hyperglycemic State (HHS), where blood sugar rises dangerously high without the presence of ketones (no acidosis).
  50. Alpha cells: These cells release glucagon when blood glucose is low. Glucagon signals the liver to convert stored glycogen back into glucose and release it into the bloodstream, raising blood sugar.
  51. Types of Diabetes:
  52. Diet control through carbohydrate counting.
  53. Normal Range: Blood glucose should be between 70-110 mg/dL. The brain relies on a steady glucose supply, as it cannot store glucose, making blood glucose regulation crucial for brain function.
  54. Hypoglycemia (Low Blood Sugar): When blood glucose drops, the pancreas releases glucagon to signal the liver to release glucose.
  55. Dawn Phenomenon:
  56. Nursing Interventions:
  57. Signs and Symptoms
  58. Intermediate-Acting Insulin (e.g., NPH): Takes 1-2 hours to start working; used to cover blood sugar between meals.
  59. Cause: Extremely high blood glucose with dehydration, typically in Type 2.
  60. Key Lab Values and Diagnostic Tests
  61. Endocrine Function: The pancreas also regulates blood sugar (glucose) levels. This happens through tiny clusters called the Islets of Langerhans, which contain two critical types of cells:
  62. What is Diabetes Mellitus?
  63. Insulin Resistance: The body produces insulin but doesn't use it effectively, often due to obesity, inactivity, and genetic factors.
  64. Diabetes Mellitus (DM): Types and Differences
  65. Type 2 Diabetes:
  66. Some patients may eventually need insulin.
  67. Type 1 Diabetes:
  68. Hyperosmolar Hyperglycemic State (HHS):
  69. The liver stores glucose in the form of glycogen. When blood glucose levels drop, glucagon from the pancreas tells the liver to release stored glucose, raising blood glucose levels. When blood glucose is high, insulin encourages the liver to stor
  70. Liver's Role:
  71. Beta cells: These cells release insulin and amylin when blood glucose is high. Insulin helps cells absorb glucose for energy, while amylin slows down the absorption of glucose and controls blood sugar spikes.
  72. Type 2: Obesity (high BMI), sedentary lifestyle, family history, certain ethnicities (Hispanic, African American, Native American, Asian American, Pacific Islander), and history of gestational diabetes. Symptoms usually develop gradually.
  73. Cause: High blood glucose with ketone production (due to lack of insulin).
  74. Understanding Diabetes and the Pancreas' Role
  75. Biguanides (e.g., metformin) decrease glucose production in the liver.
  76. Type 2 Diabetes:
  77. Blood Glucose Basics:
  78. Should be below 140 mg/dL after 2 hours of consuming glucose.
  79. Common onset is around age 50 and tends to develop gradually.
  80. Higher risk for DKA due to the complete lack of insulin.
  81. Type 1 Diabetes:
  82. Autoimmune Condition: The immune system attacks and destroys beta cells in the pancreas, which stops the production of insulin.
  83. Urine Screening:
  84. Sick Day Rules: Maintain blood sugar checks, even if not eating much, as stress and illness can increase blood sugar.
  85. Reflects average blood glucose levels over the past 3 months. A normal A1C is between 4-6%.