Signs andSymptomsNormalFastingLevel: Lessthan 100mg/dL.Quick onset ofsymptoms, includingweight loss,increased appetite,and the "3 Ps"(polyuria, polydipsia,polyphagia).Exercise: Canlower bloodglucose butrequires carefulplanning to avoidhypoglycemia.NursingInterventions:Normal Range: Bloodglucose should bebetween 70-110 mg/dL.The brain relies on asteady glucose supply,as it cannot storeglucose, making bloodglucose regulationcrucial for brain function.Biguanides(e.g., metformin)decreaseglucoseproduction in theliver.Somepatients mayeventuallyneed insulin.Endocrine Function:The pancreas alsoregulates blood sugar(glucose) levels. Thishappens through tinyclusters called theIslets of Langerhans,which contain twocritical types of cells:Regularinsulininjections oran insulinpump.Symptoms: Suddenweight loss, frequenturination (polyuria),extreme thirst(polydipsia), andincreased hunger(polyphagia).Monitoringblood sugarmultipletimes daily.Common onsetis around age50 and tends todevelopgradually.HemoglobinA1C:Cause: Highblood glucosewith ketoneproduction (dueto lack ofinsulin).TherapeuticManagementand NursingInterventionsType 1Diabetes:UnderstandingDiabetes andthe Pancreas'RoleGlucoseToleranceTest:ExocrineFunction: Thisinvolvesproducingenzymes thathelp digest food.Intermediate-Acting Insulin(e.g., NPH): Takes1-2 hours to startworking; used tocover blood sugarbetween meals.Short-Acting Insulin(e.g., Regularinsulin): Takes 30-60 minutes to workand is used formeal coverage andDKA emergencies.Higher risk forHyperosmolarHyperglycemic State(HHS), where bloodsugar risesdangerously highwithout the presence ofketones (no acidosis).Symptoms:Often similar toType 1 (polyuria,polydipsia,polyphagia), butless noticeable.DawnPhenomenon:Type 1Diabetes:Symptoms:Extreme thirst,confusion, drymouth, noketones.Hyperglycemia (HighBlood Sugar): Whenblood glucose rises,the pancreasreleases insulin tohelp cells absorbglucose and lowerblood sugar levels.BloodGlucose:UnderstandingDawnPhenomenonvs. SomogyiEffectBlood GlucoseMonitoring:Educate onfrequency andtechniques.Diet, exercise,and potentiallyoralmedicationsthat help lowerblood glucose.SomogyiEffect:Insulin Resistance:The body producesinsulin but doesn'tuse it effectively,often due to obesity,inactivity, andgenetic factors.Hypoglycemia (LowBlood Sugar): Whenblood glucose drops,the pancreasreleases glucagon tosignal the liver torelease glucose.Complicationsof DiabetesHigher risk forDiabetic Ketoacidosis(DKA), a life-threatening conditionwhere bloodbecomes acidic dueto a lack of insulin.Blood sugar drops duringthe night (often due toexcess insulin before bed),and the body compensatesby raising blood glucose,resulting in high morningglucose. Managed byadjusting insulin dosage oradding a bedtime snack.Liver'sRole:UrineScreening:Type 2Diabetes:Type 1Diabetes:TypesofInsulin:PancreasFunctions:Focus onlifestylechangesfirst:DiabetesRiskFactorsWeight gain iscommon, andHHS is a riskdue to highblood glucosewithout ketones.Insulin isessential.Treatmentinvolves:The liver stores glucose inthe form of glycogen. Whenblood glucose levels drop,glucagon from thepancreas tells the liver torelease stored glucose,raising blood glucoselevels. When blood glucoseis high, insulin encouragesthe liver to storDiabetes Mellitus is acondition where thebody cannot regulateblood glucoseproperly, resulting inchronichyperglycemia (highblood sugar).Reflects averageblood glucoselevels over thepast 3 months. Anormal A1C isbetween 4-6%.Types ofDiabetes:Should bebelow 140mg/dL after 2hours ofconsumingglucose.Key LabValues andDiagnosticTestsTreatment:Insulin IV,fluids, andmonitoringelectrolytes.Type 2Diabetes:What isDiabetesMellitus?Treatment:Insulin IV,fluids, andelectrolytereplacement.Beta cells: These cellsrelease insulin and amylinwhen blood glucose ishigh. Insulin helps cellsabsorb glucose for energy,while amylin slows downthe absorption of glucoseand controls blood sugarspikes.BloodGlucoseBasics:Checks for glucoseand ketones in theurine, as elevatedlevels may indicatediabetes oruncontrolled bloodglucose.DiabeticKetoacidosis(DKA):Rapid-ActingInsulin (e.g.,Humalog): Workswithin 15 minutesand is givenbefore or withmeals.HyperosmolarHyperglycemicState (HHS):AutoimmuneCondition: Theimmune systemattacks and destroysbeta cells in thepancreas, whichstops the productionof insulin.Symptoms:Rapid breathing(Kussmaul),fruity breath,nausea,dehydration.MedicationTypes andInsulinAdministrationHigher riskfor DKA dueto thecomplete lackof insulin.OralHypoglycemicsfor Type 2:Sick Day Rules:Maintain bloodsugar checks, evenif not eating much,as stress andillness can increaseblood sugar.Blood sugar risesin the earlymorning due tohormone release.Managed byadjusting insulintiming.Alpha cells: These cellsrelease glucagon whenblood glucose is low.Glucagon signals theliver to convert storedglycogen back intoglucose and release itinto the bloodstream,raising blood sugar.Cause: Extremelyhigh bloodglucose withdehydration,typically in Type2.DiabetesMellitus(DM): TypesandDifferencesCommon inpeople under30 and oftendiagnosedabruptly.Foot Care: Inspectfeet daily for cuts,blisters, orinfections; wearproper footwear;avoid barefootwalking.Insulinstimulators (e.g.,glipizide) helpthe pancreasrelease moreinsulin.InsulinSensitizers (e.g.,pioglitazone)make cells moreresponsive toinsulin.Long-Acting Insulin(e.g., Lantus):Works slowlywithout peaks,providing steadyinsulin levels over12-24 hours.Type 2: Obesity (high BMI),sedentary lifestyle, familyhistory, certain ethnicities(Hispanic, AfricanAmerican, NativeAmerican, Asian American,Pacific Islander), andhistory of gestationaldiabetes. Symptomsusually develop gradually.Diet controlthroughcarbohydratecounting.Type 1: Family history,genetic predisposition,autoimmuneconditions, andsometimes followingviral infections. It’smore common in thoseunder 30.Gradualonset, withsymptomsoften lessnoticeable.Diet: Emphasizebalanced meals,carb counting,and limitingsugar intake.Type 2Diabetes:Signs andSymptomsNormalFastingLevel: Lessthan 100mg/dL.Quick onset ofsymptoms, includingweight loss,increased appetite,and the "3 Ps"(polyuria, polydipsia,polyphagia).Exercise: Canlower bloodglucose butrequires carefulplanning to avoidhypoglycemia.NursingInterventions:Normal Range: Bloodglucose should bebetween 70-110 mg/dL.The brain relies on asteady glucose supply,as it cannot storeglucose, making bloodglucose regulationcrucial for brain function.Biguanides(e.g., metformin)decreaseglucoseproduction in theliver.Somepatients mayeventuallyneed insulin.Endocrine Function:The pancreas alsoregulates blood sugar(glucose) levels. Thishappens through tinyclusters called theIslets of Langerhans,which contain twocritical types of cells:Regularinsulininjections oran insulinpump.Symptoms: Suddenweight loss, frequenturination (polyuria),extreme thirst(polydipsia), andincreased hunger(polyphagia).Monitoringblood sugarmultipletimes daily.Common onsetis around age50 and tends todevelopgradually.HemoglobinA1C:Cause: Highblood glucosewith ketoneproduction (dueto lack ofinsulin).TherapeuticManagementand NursingInterventionsType 1Diabetes:UnderstandingDiabetes andthe Pancreas'RoleGlucoseToleranceTest:ExocrineFunction: Thisinvolvesproducingenzymes thathelp digest food.Intermediate-Acting Insulin(e.g., NPH): Takes1-2 hours to startworking; used tocover blood sugarbetween meals.Short-Acting Insulin(e.g., Regularinsulin): Takes 30-60 minutes to workand is used formeal coverage andDKA emergencies.Higher risk forHyperosmolarHyperglycemic State(HHS), where bloodsugar risesdangerously highwithout the presence ofketones (no acidosis).Symptoms:Often similar toType 1 (polyuria,polydipsia,polyphagia), butless noticeable.DawnPhenomenon:Type 1Diabetes:Symptoms:Extreme thirst,confusion, drymouth, noketones.Hyperglycemia (HighBlood Sugar): Whenblood glucose rises,the pancreasreleases insulin tohelp cells absorbglucose and lowerblood sugar levels.BloodGlucose:UnderstandingDawnPhenomenonvs. SomogyiEffectBlood GlucoseMonitoring:Educate onfrequency andtechniques.Diet, exercise,and potentiallyoralmedicationsthat help lowerblood glucose.SomogyiEffect:Insulin Resistance:The body producesinsulin but doesn'tuse it effectively,often due to obesity,inactivity, andgenetic factors.Hypoglycemia (LowBlood Sugar): Whenblood glucose drops,the pancreasreleases glucagon tosignal the liver torelease glucose.Complicationsof DiabetesHigher risk forDiabetic Ketoacidosis(DKA), a life-threatening conditionwhere bloodbecomes acidic dueto a lack of insulin.Blood sugar drops duringthe night (often due toexcess insulin before bed),and the body compensatesby raising blood glucose,resulting in high morningglucose. Managed byadjusting insulin dosage oradding a bedtime snack.Liver'sRole:UrineScreening:Type 2Diabetes:Type 1Diabetes:TypesofInsulin:PancreasFunctions:Focus onlifestylechangesfirst:DiabetesRiskFactorsWeight gain iscommon, andHHS is a riskdue to highblood glucosewithout ketones.Insulin isessential.Treatmentinvolves:The liver stores glucose inthe form of glycogen. Whenblood glucose levels drop,glucagon from thepancreas tells the liver torelease stored glucose,raising blood glucoselevels. When blood glucoseis high, insulin encouragesthe liver to storDiabetes Mellitus is acondition where thebody cannot regulateblood glucoseproperly, resulting inchronichyperglycemia (highblood sugar).Reflects averageblood glucoselevels over thepast 3 months. Anormal A1C isbetween 4-6%.Types ofDiabetes:Should bebelow 140mg/dL after 2hours ofconsumingglucose.Key LabValues andDiagnosticTestsTreatment:Insulin IV,fluids, andmonitoringelectrolytes.Type 2Diabetes:What isDiabetesMellitus?Treatment:Insulin IV,fluids, andelectrolytereplacement.Beta cells: These cellsrelease insulin and amylinwhen blood glucose ishigh. Insulin helps cellsabsorb glucose for energy,while amylin slows downthe absorption of glucoseand controls blood sugarspikes.BloodGlucoseBasics:Checks for glucoseand ketones in theurine, as elevatedlevels may indicatediabetes oruncontrolled bloodglucose.DiabeticKetoacidosis(DKA):Rapid-ActingInsulin (e.g.,Humalog): Workswithin 15 minutesand is givenbefore or withmeals.HyperosmolarHyperglycemicState (HHS):AutoimmuneCondition: Theimmune systemattacks and destroysbeta cells in thepancreas, whichstops the productionof insulin.Symptoms:Rapid breathing(Kussmaul),fruity breath,nausea,dehydration.MedicationTypes andInsulinAdministrationHigher riskfor DKA dueto thecomplete lackof insulin.OralHypoglycemicsfor Type 2:Sick Day Rules:Maintain bloodsugar checks, evenif not eating much,as stress andillness can increaseblood sugar.Blood sugar risesin the earlymorning due tohormone release.Managed byadjusting insulintiming.Alpha cells: These cellsrelease glucagon whenblood glucose is low.Glucagon signals theliver to convert storedglycogen back intoglucose and release itinto the bloodstream,raising blood sugar.Cause: Extremelyhigh bloodglucose withdehydration,typically in Type2.DiabetesMellitus(DM): TypesandDifferencesCommon inpeople under30 and oftendiagnosedabruptly.Foot Care: Inspectfeet daily for cuts,blisters, orinfections; wearproper footwear;avoid barefootwalking.Insulinstimulators (e.g.,glipizide) helpthe pancreasrelease moreinsulin.InsulinSensitizers (e.g.,pioglitazone)make cells moreresponsive toinsulin.Long-Acting Insulin(e.g., Lantus):Works slowlywithout peaks,providing steadyinsulin levels over12-24 hours.Type 2: Obesity (high BMI),sedentary lifestyle, familyhistory, certain ethnicities(Hispanic, AfricanAmerican, NativeAmerican, Asian American,Pacific Islander), andhistory of gestationaldiabetes. Symptomsusually develop gradually.Diet controlthroughcarbohydratecounting.Type 1: Family history,genetic predisposition,autoimmuneconditions, andsometimes followingviral infections. It’smore common in thoseunder 30.Gradualonset, withsymptomsoften lessnoticeable.Diet: Emphasizebalanced meals,carb counting,and limitingsugar intake.Type 2Diabetes:

Untitled Bingo - Call List

(Print) Use this randomly generated list as your call list when playing the game. There is no need to say the BINGO column name. Place some kind of mark (like an X, a checkmark, a dot, tally mark, etc) on each cell as you announce it, to keep track. You can also cut out each item, place them in a bag and pull words from the bag.


1
2
3
4
5
6
7
8
9
10
11
12
13
14
15
16
17
18
19
20
21
22
23
24
25
26
27
28
29
30
31
32
33
34
35
36
37
38
39
40
41
42
43
44
45
46
47
48
49
50
51
52
53
54
55
56
57
58
59
60
61
62
63
64
65
66
67
68
69
70
71
72
73
74
75
76
77
78
79
80
81
82
83
84
85
  1. Signs and Symptoms
  2. Normal Fasting Level: Less than 100 mg/dL.
  3. Quick onset of symptoms, including weight loss, increased appetite, and the "3 Ps" (polyuria, polydipsia, polyphagia).
  4. Exercise: Can lower blood glucose but requires careful planning to avoid hypoglycemia.
  5. Nursing Interventions:
  6. Normal Range: Blood glucose should be between 70-110 mg/dL. The brain relies on a steady glucose supply, as it cannot store glucose, making blood glucose regulation crucial for brain function.
  7. Biguanides (e.g., metformin) decrease glucose production in the liver.
  8. Some patients may eventually need insulin.
  9. Endocrine Function: The pancreas also regulates blood sugar (glucose) levels. This happens through tiny clusters called the Islets of Langerhans, which contain two critical types of cells:
  10. Regular insulin injections or an insulin pump.
  11. Symptoms: Sudden weight loss, frequent urination (polyuria), extreme thirst (polydipsia), and increased hunger (polyphagia).
  12. Monitoring blood sugar multiple times daily.
  13. Common onset is around age 50 and tends to develop gradually.
  14. Hemoglobin A1C:
  15. Cause: High blood glucose with ketone production (due to lack of insulin).
  16. Therapeutic Management and Nursing Interventions
  17. Type 1 Diabetes:
  18. Understanding Diabetes and the Pancreas' Role
  19. Glucose Tolerance Test:
  20. Exocrine Function: This involves producing enzymes that help digest food.
  21. Intermediate-Acting Insulin (e.g., NPH): Takes 1-2 hours to start working; used to cover blood sugar between meals.
  22. Short-Acting Insulin (e.g., Regular insulin): Takes 30-60 minutes to work and is used for meal coverage and DKA emergencies.
  23. Higher risk for Hyperosmolar Hyperglycemic State (HHS), where blood sugar rises dangerously high without the presence of ketones (no acidosis).
  24. Symptoms: Often similar to Type 1 (polyuria, polydipsia, polyphagia), but less noticeable.
  25. Dawn Phenomenon:
  26. Type 1 Diabetes:
  27. Symptoms: Extreme thirst, confusion, dry mouth, no ketones.
  28. Hyperglycemia (High Blood Sugar): When blood glucose rises, the pancreas releases insulin to help cells absorb glucose and lower blood sugar levels.
  29. Blood Glucose:
  30. Understanding Dawn Phenomenon vs. Somogyi Effect
  31. Blood Glucose Monitoring: Educate on frequency and techniques.
  32. Diet, exercise, and potentially oral medications that help lower blood glucose.
  33. Somogyi Effect:
  34. Insulin Resistance: The body produces insulin but doesn't use it effectively, often due to obesity, inactivity, and genetic factors.
  35. Hypoglycemia (Low Blood Sugar): When blood glucose drops, the pancreas releases glucagon to signal the liver to release glucose.
  36. Complications of Diabetes
  37. Higher risk for Diabetic Ketoacidosis (DKA), a life-threatening condition where blood becomes acidic due to a lack of insulin.
  38. Blood sugar drops during the night (often due to excess insulin before bed), and the body compensates by raising blood glucose, resulting in high morning glucose. Managed by adjusting insulin dosage or adding a bedtime snack.
  39. Liver's Role:
  40. Urine Screening:
  41. Type 2 Diabetes:
  42. Type 1 Diabetes:
  43. Types of Insulin:
  44. Pancreas Functions:
  45. Focus on lifestyle changes first:
  46. Diabetes Risk Factors
  47. Weight gain is common, and HHS is a risk due to high blood glucose without ketones.
  48. Insulin is essential. Treatment involves:
  49. The liver stores glucose in the form of glycogen. When blood glucose levels drop, glucagon from the pancreas tells the liver to release stored glucose, raising blood glucose levels. When blood glucose is high, insulin encourages the liver to stor
  50. Diabetes Mellitus is a condition where the body cannot regulate blood glucose properly, resulting in chronic hyperglycemia (high blood sugar).
  51. Reflects average blood glucose levels over the past 3 months. A normal A1C is between 4-6%.
  52. Types of Diabetes:
  53. Should be below 140 mg/dL after 2 hours of consuming glucose.
  54. Key Lab Values and Diagnostic Tests
  55. Treatment: Insulin IV, fluids, and monitoring electrolytes.
  56. Type 2 Diabetes:
  57. What is Diabetes Mellitus?
  58. Treatment: Insulin IV, fluids, and electrolyte replacement.
  59. Beta cells: These cells release insulin and amylin when blood glucose is high. Insulin helps cells absorb glucose for energy, while amylin slows down the absorption of glucose and controls blood sugar spikes.
  60. Blood Glucose Basics:
  61. Checks for glucose and ketones in the urine, as elevated levels may indicate diabetes or uncontrolled blood glucose.
  62. Diabetic Ketoacidosis (DKA):
  63. Rapid-Acting Insulin (e.g., Humalog): Works within 15 minutes and is given before or with meals.
  64. Hyperosmolar Hyperglycemic State (HHS):
  65. Autoimmune Condition: The immune system attacks and destroys beta cells in the pancreas, which stops the production of insulin.
  66. Symptoms: Rapid breathing (Kussmaul), fruity breath, nausea, dehydration.
  67. Medication Types and Insulin Administration
  68. Higher risk for DKA due to the complete lack of insulin.
  69. Oral Hypoglycemics for Type 2:
  70. Sick Day Rules: Maintain blood sugar checks, even if not eating much, as stress and illness can increase blood sugar.
  71. Blood sugar rises in the early morning due to hormone release. Managed by adjusting insulin timing.
  72. Alpha cells: These cells release glucagon when blood glucose is low. Glucagon signals the liver to convert stored glycogen back into glucose and release it into the bloodstream, raising blood sugar.
  73. Cause: Extremely high blood glucose with dehydration, typically in Type 2.
  74. Diabetes Mellitus (DM): Types and Differences
  75. Common in people under 30 and often diagnosed abruptly.
  76. Foot Care: Inspect feet daily for cuts, blisters, or infections; wear proper footwear; avoid barefoot walking.
  77. Insulin stimulators (e.g., glipizide) help the pancreas release more insulin.
  78. Insulin Sensitizers (e.g., pioglitazone) make cells more responsive to insulin.
  79. Long-Acting Insulin (e.g., Lantus): Works slowly without peaks, providing steady insulin levels over 12-24 hours.
  80. Type 2: Obesity (high BMI), sedentary lifestyle, family history, certain ethnicities (Hispanic, African American, Native American, Asian American, Pacific Islander), and history of gestational diabetes. Symptoms usually develop gradually.
  81. Diet control through carbohydrate counting.
  82. Type 1: Family history, genetic predisposition, autoimmune conditions, and sometimes following viral infections. It’s more common in those under 30.
  83. Gradual onset, with symptoms often less noticeable.
  84. Diet: Emphasize balanced meals, carb counting, and limiting sugar intake.
  85. Type 2 Diabetes: